نتایج جستجو برای: el tor variant

تعداد نتایج: 386589  

2014
Konstantin V. Kuleshov Sergey O. Vodop’ianov Vladimir G. Dedkov Mikhail L. Markelov Anton V. Kermanov Vladimir D. Kruglikov Alexey S. Vodop’ianov Ruslan V. Pisanov Olga S. Chemisova Alexey B. Mazrukho Svetlana V. Titova German A. Shipulin

We report the draft genome sequencing of five Vibrio cholerae O1 El Tor clinical isolates collected in the Russian Federation from imported cholera cases in 2006, 2010, and 2012. In the initial phylogenetic analysis, one isolate clustered with the Haiti/Nepal-4 group.

2017
Nina I. Smirnova Yaroslav M. Krasnov Elena Y. Agafonova Elena Y. Shchelkanova Zhanna V. Alkhova Vladimir V. Kutyrev

Here, we present the draft whole-genome sequence of Vibrio cholerae O1 El Tor strains 76 and M3265/80, isolated in Mariupol, Ukraine, and Moscow, Russia. The presence of various mutations detected in virulence-associated mobile elements indicates high genetic similarity of the strains reported here with new highly virulent variants of the cholera agent V. cholerae.

2017
Ali Al-Fendi Rafidah Hanim Shueb Phiaw Chong Foo Manickam Ravichandran Chan Yean Yean

The complete genome sequence of bacteriophage VPUSM 8 against O1 El Tor Inaba Vibrio cholerae is reported here. The isolated VPUSM 8 has potential use in future phage therapy or as a biocontrol agent for the prevention and treatment of cholera.

2011
Afsar Ali Yuansha Chen Judith A. Johnson Edsel Redden Yfto Mayette Mohammed H. Rashid O. Colin Stine J. Glenn Morris

Altered El Tor Vibrio cholerae O1, with classical cholera toxin B gene, was isolated from 16 patients with severe diarrhea at St. Mark's Hospital, Arbonite, Haiti, <3 weeks after onset of the current cholera epidemic. Variable-number tandem-repeat typing of 187 isolates showed minimal diversity, consistent with a point source for the epidemic.

Journal: :Journal of clinical microbiology 2009
Binh Minh Nguyen Je Hee Lee Ngo Tuan Cuong Seon Young Choi Nguyen Tran Hien Dang Duc Anh Hye Ri Lee M Ansaruzzaman Hubert P Endtz Jongsik Chun Anna Lena Lopez Cecil Czerkinsky John D Clemens Dong Wook Kim

Vibrio cholerae O1 isolates collected during cholera outbreaks occurring from late 2007 to early 2008 in northern Vietnam were revealed to represent an altered strain containing the RS1 element followed by a CTX prophage harboring El Tor type rstR and classical ctxB on the large chromosome.

2005
Anna Salim Ruiting Lan Peter R. Reeves

We resolved the relationships between 2 pandemic clones of Vibrio cholerae. Using 26 housekeeping genes, we showed that the US Gulf clone, the Australian clone, and 3 El Tor strains isolated before the seventh pandemic were related to the seventh pandemic clone. The sixth pandemic clone was well separated from them.

Journal: :Antimicrobial agents and chemotherapy 1985
M J Dupont M Jouvenot G Couetdic Y Michel-Briand

The persistence of Vibrio cholerae, biotype el tor, in a patient treated with trimethoprim-sulfamethoxazole was due to the acquisition of a conjugative resistance plasmid. The plasmid, with a molecular size of 72 megadaltons, belonged to incompatibility group 6-C and conferred resistance to ampicillin, chloramphenicol, sulfonamide, and trimethoprim.

2012
Cindy Shuan Ju Teh Zarizal Suhaili King Ting Lim Muhamad Afif Khamaruddin Fariha Yahya Mohd Hailmi Sajili Chew Chieng Yeo Kwai Lin Thong

A cholera outbreak in Terengganu, Malaysia, in November 2009 was caused by 2 El Tor Vibrio cholerae variants resistant to typical antimicrobial drugs. Evidence of replacement of treatable V. cholerae infection in the region with antimicrobial-resistant strains calls for increased surveillance and prevention measures.

Journal: :Infection and immunity 1972
S C Agarwal N K Ganguly

Oral administration of heat-inactivated lysates of Vibrio cholerae Ogawa L-forms elicited significantly high coproantibody, serum indirect hemagglutinating antibody, and vibriocidal antibody responses in human volunteers, against both homologous and heterologous subtypes of V. cholerae and V. El Tor. Five oral biweekly doses produced an adequate antibody response which persisted for at least 6 ...

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