نتایج جستجو برای: dna repair enzymes

تعداد نتایج: 713751  

2013
Katarzyna D. Arczewska Gisele G. Tomazella Jessica M. Lindvall Henok Kassahun Silvia Maglioni Alessandro Torgovnick Johan Henriksson Olli Matilainen Bryce J. Marquis Bryant C. Nelson Pawel Jaruga Eshrat Babaie Carina I. Holmberg Thomas R. Bürglin Natascia Ventura Bernd Thiede Hilde Nilsen

Oxidative stress promotes human aging and contributes to common neurodegenerative diseases. Endogenous DNA damage induced by oxidative stress is believed to be an important promoter of neurodegenerative diseases. Although a large amount of evidence correlates a reduced DNA repair capacity with aging and neurodegenerative disease, there is little direct evidence of causality. Moreover, the contr...

Journal: :Molecular cell 2015
Paul Wijnhoven Rebecca Konietzny Andrew N Blackford Jonathan Travers Benedikt M Kessler Ryotaro Nishi Stephen P Jackson

Repair of DNA double-strand breaks is crucial for maintaining genome integrity and is governed by post-translational modifications such as protein ubiquitylation. Here, we establish that the deubiquitylating enzyme USP4 promotes DNA-end resection and DNA repair by homologous recombination. We also report that USP4 interacts with CtIP and the MRE11-RAD50-NBS1 (MRN) complex and is required for Ct...

Journal: :ACS chemical biology 2015
Nathan Donley Pawel Jaruga Erdem Coskun Miral Dizdaroglu Amanda K McCullough R Stephen Lloyd

The DNA base excision repair (BER) pathway, which utilizes DNA glycosylases to initiate repair of specific DNA lesions, is the major pathway for the repair of DNA damage induced by oxidation, alkylation, and deamination. Early results from clinical trials suggest that inhibiting certain enzymes in the BER pathway can be a useful anticancer strategy when combined with certain DNA-damaging agents...

Journal: :E3S web of conferences 2021

Compost is a promising source of thermotolerant enzymes for their application in biotechnology. Homologues bacteriophage T4 DNA glycosylase can find pharmaceuticals and perfumery. Five homologues pyrimidine dimers T4, product the denV gene, were found by comparing using DELTA-BLAST algorithm with compost metagenome proteins. Phylogenetic analysis sequences enzyme was carried out Maximum Likelih...

Journal: :Acta Crystallographica Section A Foundations of Crystallography 2005

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1972
P K Cooper P C Hanawalt

The DNA polymerase I-deficient mutant polA1 is shown to perform an increased amount of UV-stimulated repair synthesis relative to its pol(+) parent. In contrast, a recA recB double mutant is found to perform little detectable repair synthesis. Analysis of the density distribution of sheared DNA of the recA recB mutant reveals that none of the repair synthesis in this strain is in the large repa...

Journal: :Life sciences 2003
Zsolt Radák Peter Apor Jozsef Pucsok Istvan Berkes Helga Ogonovszky Gabor Pavlik Hideko Nakamoto Sataro Goto

Reactive oxygen and nitrogen species generated either as products of aerobic metabolism or as a consequence of environmental mutagens, oxidatively modify DNA. Formamidopyrimidine-DNA glycosylase (Fpg) and endonuclease III (endo III) or their functional mammalian homologues repair 7,8-dihydro-8-oxoguanine (8-oxoG) and damaged pyrimidines, respectively, to curb the deleterious effects of oxidativ...

Base excision repair (BER) pathway is one of the repair systems that have an impact on the radiotherapy and chemotherapy for the cancer patients. The molecular pathogenesis of gallbladder cancer is not known extensively. In the present study we investigated whether the expression of AP endonuclease 1 (APE1) and DNA polymerase β (DNA pol β), key enzymes of BER pathway has any clinical ...

Journal: :Genetics 1975
S Wolff J Bodycote G H Thomas J E Cleaver

The formation of sister chromatid exchanges has been postulated to depend upon the action of DNA repair enzymes. Our experiments with various human cell lines show that the yield of sister chromatid exchanges is within normal limits in both excision-repair-defective and post-replication-repair-defective cells from the autosomal recessive disease, xeroderma pigmentosum. These results indicate th...

Journal: :Journal of virology 1973
K Carlson Z K Lorkiewicz A W Kozinski

Discontinuities of T4 DNA which are caused by excision of UV-damaged areas, by decay of (32)P atoms, or which are present in DNA from rII(-)lig(am) (-) phage produced in a host nonpermissive for amber mutants are all repaired by bacterial enzymes after infection in the presence of chloramphenicol. Escherichia coli DNA polymerase I participates in the host-mediated repair, but an approximately 2...

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