نتایج جستجو برای: chloride channel protein 2 neuroprotection

تعداد نتایج: 3666164  

Journal: :American journal of physiology. Cell physiology 2010
Yongping Chai Yu-Fung Lin

The ATP-sensitive potassium (K(ATP)) channel couples intracellular metabolic state to membrane excitability. Recently, we demonstrated that neuronal K(ATP) channels are functionally enhanced by activation of a nitric oxide (NO)/cGMP/cGMP-dependent protein kinase (PKG) signaling cascade. In this study, we further investigated the intracellular mechanism underlying PKG stimulation of neuronal K(A...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1998
A P Naren M W Quick J F Collawn D J Nelson K L Kirk

Previously we showed that the functional activity of the epithelial chloride channel that is encoded by the cystic fibrosis gene (CFTR) is reciprocally modulated by two components of the vesicle fusion machinery, syntaxin 1A and Munc-18. Here we report that syntaxin 1A inhibits CFTR chloride channels by means of direct and domain-specific protein-protein interactions. Syntaxin 1A stoichiometric...

Journal: :Stroke 2004
Cesar V Borlongan Martin Hadman Cyndy Davis Sanberg Paul R Sanberg

BACKGROUND AND PURPOSE To date, stem cell graft-mediated neuroprotection is equated with graft survival and secretion of neurotrophic factors in the brain. Here, we examined whether neuroprotection by systemically delivered human umbilical cord blood (HUCB) cells was dependent on their entry into the central nervous system in a rodent model of acute stroke. METHODS Adult male Sprague-Dawley r...

Journal: :Molecules 2016
Yisong Qian Xuzhen Tang Teng Guan Yunman Li Hongbin Sun

Glutamate-mediated excitotoxicity is a major cause of ischemic brain damage. MK-801 confers neuroprotection by attenuating the activation of the N-methyl-d-aspartate (NMDA) receptor, but it failed in clinical use due to the short therapeutic window. Here we aim to investigate the effects of maslinic acid, a natural product from Olea europaea, on the therapeutic time window and dose range for th...

2017
Jonas Friard Michel Tauc Marc Cougnon Vincent Compan Christophe Duranton Isabelle Rubera

Chloride channels play an essential role in a variety of physiological functions and in human diseases. Historically, the field of chloride channels has long been neglected owing to the lack of powerful selective pharmacological agents that are needed to overcome the technical challenge of characterizing the molecular identities of these channels. Recently, members of the LRRC8 family have been...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2010
Renée E Haskew-Layton Jimmy B Payappilly Natalya A Smirnova Thong C Ma Kelvin K Chan Timothy H Murphy Hengchang Guo Brett Langley Rukhsana Sultana D Allan Butterfield Sandro Santagata Melissa J Alldred Irina G Gazaryan George W Bell Stephen D Ginsberg Rajiv R Ratan

Neurons rely on their metabolic coupling with astrocytes to combat oxidative stress. The transcription factor nuclear factor erythroid 2-related factor 2 (Nrf2) appears important for astrocyte-dependent neuroprotection from oxidative insults. Indeed, Nrf2 activators are effective in stroke, Parkinson disease, and Huntington disease models. However, key endogenous signals that initiate adaptive ...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2008
Samantha J Pitt Lucia G Sivilotti Marco Beato

The time course of currents mediated by native and recombinant glycine receptors was examined with a combination of rapid agonist applications to outside-out patches and single-channel recording. The deactivation time constant of currents evoked by brief, saturating pulses of glycine is profoundly affected by the chloride concentration on the intracellular side of the cell membrane. Deactivatio...

2012
Paola Russo Antonietta Santoro Lucia Prota Mariateresa Stigliani Rita P. Aquino

Cystic Fibrosis (CF) is the most common lethal monogenic disorder in Caucasians, estimated to affect one per 2500-4000 newborns. CF is caused by mutations in the gene encoding the CF transmembrane conductance regulator (CFTR) [1, 2]. CFTR acts mainly as a chloride channel and has other regulatory roles, including inhibition of sodium transport through the epithelial sodium channel, regulation o...

Journal: :Biochimica et Biophysica Acta (BBA) - Biomembranes 2011

Journal: :Archives of Otolaryngology–Head & Neck Surgery 2008

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