نتایج جستجو برای: bcl11a

تعداد نتایج: 301  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2015
Mary-Lee Dequéant Delphine Fagegaltier Yanhui Hu Kerstin Spirohn Amanda Simcox Gregory J Hannon Norbert Perrimon

The use of time series profiling to identify groups of functionally related genes (synexpression groups) is a powerful approach for the discovery of gene function. Here we apply this strategy during Ras(V12) immortalization of Drosophila embryonic cells, a phenomenon not well characterized. Using high-resolution transcriptional time-series datasets, we generated a gene network based on temporal...

Journal: :Haematologica 2011
Catherine Badens Philippe Joly Imane Agouti Isabelle Thuret Katia Gonnet Synda Fattoum Alain Francina Marie-Claude Simeoni Anderson Loundou Serge Pissard

A cohort of 106 patients included in the French National Registry for Thalassemia were genotyped for 5 genetic modifiers of severity: i) β-thalassemia mutations; (ii) the XmnI SNP; (iii) the -3.7 kb α-thal deletion; (iv) the tag-SNP rs 11886868 in BCL11A exon 2; and (v) the tag-SNP rs9399137 in the HBSB1L-cMYB inter-region. Multivariate analysis was performed to study the risk of thalassemia In...

Journal: :Blood 2011
Julie Makani Stephan Menzel Siana Nkya Sharon E Cox Emma Drasar Deogratius Soka Albert N Komba Josephine Mgaya Helen Rooks Nisha Vasavda Gregory Fegan Charles R Newton Martin Farrall Swee Lay Thein

Fetal hemoglobin (HbF, α(2)γ(2)) is a major contributor to the remarkable phenotypic heterogeneity of sickle cell anemia (SCA). Genetic variation at 3 principal loci (HBB cluster on chromosome 11p, HBS1L-MYB region on chromosome 6q, and BCL11A on chromosome 2p) have been shown to influence HbF levels and disease severity in β-thalassemia and SCA. Previous studies in SCA, however, have been rest...

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