Cholestatic liver injury was experimentally induced in rats by administration of alpha-naphthylisothiocyanate (ANIT) and the peak activity of mitochondrial L-aspartate: 2-oxoglutarate aminotransferase (m-GOT) released in the serum was found to precede the peak of total GOT activity. To investigate the permeability of the mitochondrial inner membrane to m-GOT, liver mitochondria obtained from ra...

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