نتایج جستجو برای: pi3k akt

تعداد نتایج: 47986  

Journal: :Cancer research 2014
Aifu Lin Hai-Long Piao Li Zhuang Dos D Sarbassov Li Ma Boyi Gan

The PI3K-AKT pathway is hyperactivated in many human cancers, and several drugs to inhibit this pathway, including the PI3K/mTOR dual inhibitor NVP-BEZ235, are currently being tested in various preclinical and clinical trials. It has been shown that pharmacologic inhibition of the PI3K-AKT pathway results in feedback activation of other oncogenic signaling pathways, which likely will limit the ...

Journal: :Cancer cell 2009
Krishna M Vasudevan David A Barbie Michael A Davies Rosalia Rabinovsky Chontelle J McNear Jessica J Kim Bryan T Hennessy Hsiuyi Tseng Panisa Pochanard So Young Kim Ian F Dunn Anna C Schinzel Peter Sandy Sebastian Hoersch Qing Sheng Piyush B Gupta Jesse S Boehm Jan H Reiling Serena Silver Yiling Lu Katherine Stemke-Hale Bhaskar Dutta Corwin Joy Aysegul A Sahin Ana Maria Gonzalez-Angulo Ana Lluch Lucia E Rameh Tyler Jacks David E Root Eric S Lander Gordon B Mills William C Hahn William R Sellers Levi A Garraway

Dysregulation of the phosphatidylinositol 3-kinase (PI3K) signaling pathway occurs frequently in human cancer. PTEN tumor suppressor or PIK3CA oncogene mutations both direct PI3K-dependent tumorigenesis largely through activation of the AKT/PKB kinase. However, here we show through phosphoprotein profiling and functional genomic studies that many PIK3CA mutant cancer cell lines and human breast...

2014
SHUQING SHI HAIMEI CAO

The present study aimed to investigate the effect of shikonin on autophagy in BXPC-3 human pancreatic cancer cells and its underlying mechanism. Cell viability was assessed using the Cell Counting Kit-8 assay and the expression of light chain (LC) 3, p62, phosphoinositide 3-kinase (PI3K), Akt, phosphorylated (p)-PI3K and p-Akt was analyzed using western blot analysis. Following treatment with 1...

Journal: :Molecular and cellular biology 2004
Kun Jiang Jiazhi Sun Jin Cheng Julie Y Djeu Sheng Wei Saïd Sebti

Although recent evidence supports a tumor-suppressive role for the GTPase RhoB, little is known about its regulation by signal transduction pathways. Here we demonstrate that Ras downregulates RhoB expression by a phosphatidylinositol 3-kinase (PI3K)- and Akt- but not Mek-dependent mechanism. Furthermore, genetic and pharmacological blockade of PI3K/Akt results in upregulation of RhoB expressio...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2011
Chi Bun Chan Xia Liu Sompol Pradoldej Chunhai Hao Jie An Manuel Yepes Hongbo R Luo Keqiang Ye

Phosphoinositide 3-kinase enhancer (PIKE) binds and enhances phosphatidylinositol 3-kinase (PI3K)/Akt activities. However, its physiological functions in brain have never been explored. Here we show that PIKE is important in regulating the neuronal survival and development of neocortex. During development, enhanced apoptosis is observed in the ventricular zone of PIKE knock-out (PIKE(-/-)) cort...

2014
AI-GUI JIANG HONG YU JIAN-AN HUANG

The overactivation of the phosphatidylinositol 3-kinase (PI3K)/protein kinase B (Akt) signal transduction pathway has been examined in various carcinomas and is reported to be significantly correlated with prognosis. However, little is known with regard to the PI3K/Akt signal transduction pathway in advanced non-small cell lung carcinoma (NSCLC). The present study investigated the expression of...

Journal: :Neuro-oncology 2022

Abstract BACKGROUND Mutations of the isocitrate dehydrogenase (IDH) gene are highly prevalent in WHO II/III glioma. IDH-mutated glioma frequently exhibits strong molecular signatures PI3K/AKT/mTOR signaling, whereas precise roles this oncogenic pathway remain elusive. METHODS In study, we performed unbiased RNA sequencing and set enrichment analysis (GSEA) to investigate relevant pathways gover...

Journal: :Journal of B.U.ON. : official journal of the Balkan Union of Oncology 2015
Chi-zhi Zhang Xiao-dong Wang Hong-wei Wang Yue Cai Li-qin Chao

PURPOSE The purpose of this study was to determine the impact of sorafenib on PI3K/AKT/mTOR signaling pathway and to further define its mechanism for treating hepatocellular carcinoma (HCC). METHODS Human SMMC-7721 hepatic carcinoma cells were treated with or without 4 μmoL/L sorafenib. SMMC- 7721 cells were harvested at various time points (0-48 hrs) and assessed for changes in PI3K, mTOR, a...

Journal: :Journal of virology 2005
Maitreyi S Rajala Raju V S Rajala Roger A Astley Amir L Butt James Chodosh

Adenovirus type 19 is a major cause of epidemic keratoconjunctivitis, the only ocular adenoviral infection associated with prolonged corneal inflammation. In this study, we investigated the role of phosphoinositide 3-kinase (PI3K) and Akt and their downstream targets in adenovirus infection, and here we report the novel finding that adenovirus type 19 utilizes the PI3K/Akt pathway to maintain c...

Journal: :The Journal of biological chemistry 2003
Lin Yang Hui-Kuan Lin Saleh Altuwaijri Shaozhen Xie Liang Wang Chawnshang Chang

APPL may function as an adapter protein to modulate the phosphatidylinositol 3-kinase (PI3K)/Akt pathway. Although we have previously proven that the PI3K/Akt pathway can suppress androgen receptor (AR) transactivation, the potential linkage from APPL to the AR remains unclear. Here we demonstrated that APPL could suppress AR-mediated transactivation in a dose-dependent manner in LNCaP and PC-3...

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