نتایج جستجو برای: oxygen deficiency

تعداد نتایج: 342153  

Journal: :Pediatric blood & cancer 2008
Priya Bhat India Sisler Anderson B Collier

To the Editor: A recent letter in this journal described methemoglobinemia and hemolysis due to rasburicase in a patient with glucose-6-phosphate dehydrogenase (G6PD) deficiency [1]. We would like to report on the management of a patient with rasburicase induced methemoglobinemia and G6PD deficiency. DS is a 12-year-old Laotian male who presented with a white blood cell count of 533,900/mm (89%...

2017
Dong‐Min Yu Seung Hee Jung Hyoung‐Tae An Sungsoo Lee Jin Hong Jun Sub Park Hyun Lee Hwayeon Lee Myeong‐Suk Bahn Hyung Chul Lee Na‐Kyung Han Jesang Ko Jae‐Seon Lee Young‐Gyu Ko

Paradoxical observations have been made regarding the role of caveolin-1 (Cav-1) during cellular senescence. For example, caveolin-1 deficiency prevents reactive oxygen species-induced cellular senescence despite mitochondrial dysfunction, which leads to senescence. To resolve this paradox, we re-addressed the role of caveolin-1 in cellular senescence in human diploid fibroblasts, A549, HCT116,...

2014
Stine Normann Hansen Pernille Tveden-Nyborg Jens Lykkesfeldt

Vitamin C is a pivotal antioxidant in the brain and has been reported to have numerous functions, including reactive oxygen species scavenging, neuromodulation, and involvement in angiogenesis. Absence of vitamin C in the brain has been shown to be detrimental to survival in newborn SVCT2(-/-) mice and perinatal deficiency have shown to reduce hippocampal volume and neuron number and cause decr...

Journal: :Circulation 2011
Jan Horstkotte Tamara Perisic Manuela Schneider Philipp Lange Melanie Schroeder Claudia Kiermayer Rabea Hinkel Tilman Ziegler Pankaj K Mandal Robert David Sabine Schulz Sabine Schmitt Julian Widder Fred Sinowatz Bernhard F Becker Johann Bauersachs Michael Naebauer Wolfgang M Franz Irmela Jeremias Markus Brielmeier Hans Zischka Marcus Conrad Christian Kupatt

BACKGROUND Excessive formation of reactive oxygen species contributes to tissue injury and functional deterioration after myocardial ischemia/reperfusion. Especially, mitochondrial reactive oxygen species are capable of opening the mitochondrial permeability transition pore, a harmful event in cardiac ischemia/reperfusion. Thioredoxins are key players in the cardiac defense against oxidative st...

Journal: :American journal of physiology. Renal physiology 2015
Ivonne Loeffler Gunter Wolf

Acute kidney injury is a common complication of critically ill patients and may occur as a result of various factors and coexisting previous illnesses. Some pathophysiological responses seen in critical illness can be similar to the human physiological response to extreme environmental challenges, such as hypoxia from reduced oxygen availability at high altitudes (systemic hypoxia). Due to oxyg...

2016
Chaohua Sun Ting Wu Longmei Zhai Duyue Li Xinzhong Zhang Xuefeng Xu Huiqin Ma Yi Wang Zhenhai Han

Reactive oxygen species (ROS) are important signaling molecules in plants that contribute to stress acclimation. This study demonstrated that ROS play a critical role in Fe deficiency-induced signaling at an early stage in Malus xiaojinensis. Once ROS production has been initiated, prolonged Fe starvation leads to activation of ROS scavenging mechanisms. Further, we demonstrated that ROS scaven...

Journal: :Carbon energy 2023

Oxygen deficiency has crucial effects on the crystal structure and electrochemical performance of spinel oxide lithium electrode materials such as LiNi0.5Mn1.5O4 (LNMO) cathode. In particular, oxygen stoichiometry surface differs from that interior in LNMO. The detection local loss LNMO its correlation with cycling stability remain challenging. this study, effect controlled by sintering tempera...

Journal: :American journal of physiology. Cell physiology 2004
Petr Pecina Erich Gnaiger Jirí Zeman Ewa Pronicka Josef Houstek

Mutations in the gene SURF1 prevent synthesis of cytochrome-c oxidase (COX)-specific assembly protein and result in a fatal neurological disorder, Leigh syndrome. Because this severe COX deficiency presents with barely detectable changes of cellular respiratory rates under normoxic conditions, we analyzed the respiratory response to low oxygen in cultured fibroblasts harboring SURF1 mutations w...

Journal: :TEION KOGAKU (Journal of Cryogenics and Superconductivity Society of Japan) 1990

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