نتایج جستجو برای: nf kappa b

تعداد نتایج: 954140  

2010
Zhujun Zhang Rongcun Yang Jingyi Liu Miaomiao Zhang Jiangbo Oyang Xin Yuan Jing Zhang Xin Huang Zhizi Jing Zhiqian Zhang Yuan Zhang

2000
Bryan R. G. Williams Maryam Zamanian-Daryoush Trine H. Mogensen MARYAM ZAMANIAN-DARYOUSH TRINE H. MOGENSEN JOSEPH A. DIDONATO BRYAN R. G. WILLIAMS

Journal: :The Journal of Experimental Medicine 2001
Min You Leah M. Flick Dehua Yu Gen-Sheng Feng

Shp-2, a src homology (SH)2-containing phosphotyrosine phosphatase, appears to be involved in cytoplasmic signaling downstream of a variety of cell surface receptors, although the mechanism is unclear. Here, we have determined a role of Shp-2 in the cytokine circuit for inflammatory and immune responses. Production of interleukin (IL)-6 in response to IL-1 alpha or tumor necrosis factor (TNF)-a...

Journal: :The Journal of clinical investigation 1996
Z Zhu W Tang A Ray Y Wu O Einarsson M L Landry J Gwaltney J A Elias

To further understand the biology of rhinovirus (RV), we determined whether IL-6 was produced during RV infections and characterized the mechanism by which RV stimulates lung cell IL-6 production. In contrast to normals and minimally symptomatic volunteers, IL-6 was detected in the nasal washings from patients who developed colds after RV challenge. RV14 and RV1A, major and minor receptor group...

Journal: :Journal of Korean Medical Science 2002
Sang Hyun Kim Hyung Jun Chu Dae Hwan Kang Geun Am Song Mong Cho Ung Suk Yang Hyon Jeen Kim Hae Young Chung

The involvement of NF-kappa B binding activity is known to be important in the mechanism of acute liver injury and in the induction of cyclooxygenase (COX-2). This study was performed to evaluate NF-kappa B binding activity and the expression of COX-2 in chronic liver injury induced by carbon tetrachloride (CCl(4)). Liver tissues from Sprague-Dawley rats were collected at 1, 3, 5, and 7th week ...

Journal: :The Biochemical journal 2003
YunJin Jung Jennifer S Isaacs Sunmin Lee Jane Trepel Zheng-Gang Liu Len Neckers

Tumour necrosis factor alpha (TNF-alpha) binds to its receptor (TNFR1) and activates both death- and inflammation/survival-related signalling pathways. The inflammation and survival-related signalling cascade results in the activation of the transcription factor, nuclear factor kappa B (NF-kappa B) and requires recruitment of receptor-interacting protein (RIP) to TNFR1. The indispensable role o...

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