نتایج جستجو برای: mody

تعداد نتایج: 686  

Journal: : 2022

ZarzÄ dzanie Mediami » 2022 Tom 10, Numer 2 Recenzja monografii Michała Wójciaka pt. Metafory mody. markÄ autorskÄ w kontekście przemian rynkowych – perspektywa projektantów A

Journal: :Yearbook of pediatric endocrinology 2021

It is often difficult to identify maturity-onset diabetes of the young (MODY) in pediatric patients close onset. Hence, misdiagnosis and unnecessary insulin treatment are still common.

2014
Květoslava Křížková Václav Veverka Lenka Maletínská Rozálie Hexnerová Andrzej M. Brzozowski Jiří Jiráček Lenka Žáková

The insulin gene mutation c.137G>A (R46Q), which changes an arginine at the B22 position of the mature hormone to glutamine, causes the monogenic diabetes variant maturity-onset diabetes of the young (MODY). In MODY patients, this mutation is heterozygous, and both mutant and wild-type (WT) human insulin are produced simultaneously. However, the patients often depend on administration of exogen...

Journal: :American journal of physiology. Renal physiology 2014
James P Calvet

IT IS SUPPOSED that every gene in the body has a constellation of controlling elements serving as binding sites for transcription factors working coordinately in a tissue-specific, temporal, and/or developmental fashion. These DNA elements are bound by a community of protein factors organized around transcription start sites to regulate the relative activities and levels of expression of genes ...

Journal: :Current Diabetes Reports 2015

2010
Caroline Bonner Siobhán Bacon Caoimhín G. Concannon Syed R. Rizvi Mathurin Baquié Angela M. Farrelly Seán M. Kilbride Heiko Dussmann Manus W. Ward Chantal M. Boulanger Claes B. Wollheim Rolf Graf Maria M. Byrne Jochen H.M. Prehn

OBJECTIVE In diabetes, β-cell mass is not static but in a constant process of cell death and renewal. Inactivating mutations in transcription factor 1 (tcf-1)/hepatocyte nuclear factor1a (hnf1a) result in decreased β-cell mass and HNF1A-maturity onset diabetes of the young (HNF1A-MODY). Here, we investigated the effect of a dominant-negative HNF1A mutant (DN-HNF1A) induced apoptosis on the rege...

Journal: :Bioscience reports 2011
Geun Bae Rha Guangteng Wu Young-In Chi

HNF4α (hepatocyte nuclear factor 4α) is a culprit gene product for a monogenic and dominantly inherited form of diabetes, referred to as MODY (maturity onset diabetes of the young). As a member of the NR (nuclear receptor) superfamily, HNF4α recruits transcriptional co-activators such as SRC-1α (steroid receptor co-activator-1α) and PGC-1α (peroxisome-proliferator-activated receptor γ co-activa...

Journal: : 2022

AMAÇ: Glukokinaz (GCK) gen mutasyonu pozitif olan Genç Erişkin Dönemde Başlayan Diyabet (Maturity Onset Diabetes of Young - MODY) tanısı alan olgularla sağlıklı bireylerin koroid tabakası kalınlıklarının karşılaştırılması amaçlandı.GEREÇ VE YÖNTEM: MODY GCK bakılarak doğrulanmış ama diyabetik retinopatisi olmayan 30 hasta (MODY grubu) ve sağlıklı kişi (kontrol çalışmaya dahil edildi. Kontrol gr...

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