نتایج جستجو برای: kit

تعداد نتایج: 29127  

2014
Janet Lau Qiang Zhou Susan E. Sutton Ann E. Herman Christian Schmedt Richard Glynne

AIM/HYPOTHESIS Recent studies indicate that tyrosine kinase inhibitors, including imatinib, can reverse hyperglycemia in non-obese diabetic (NOD) mice, a model of type 1 diabetes (T1D). Imatinib inhibits c-Abl, c-Kit, and PDGFRs. Next-generation tyrosine kinase inhibitors for T1D treatment should maintain activities required for efficacy while sparing inhibition of targets that might otherwise ...

Journal: :jundishapur journal of microbiology 0
seyed fazlolah mousavi department of bacteriology, pasteur institute of iran, tehran, ir iran sara fatemi department of biology, science and research branch, islamic azad university, tehran, ir iran seyed davar siadat department of biology, science and research branch, islamic azad university, tehran, ir iran seyed mohsen zahraei center for communicable disease control, tehran, ir iran elnaz nikanpour department of bacteriology, pasteur institute of iran, tehran, ir iran mohamad ali malekan department of bacteriology, pasteur institute of iran, tehran, ir iran

results the optimal conditions were considered to perform elisa. comparison between results obtained from optimized elisa kit and commercial elisa kit showed a good agreement. background haemophilus influenzae type b (hib) infection has high morbidity and mortality rate, especially in children under 5 years of age. enzyme-linked immunosorbent assay (elisa) technique is the most used method to d...

Journal: :Anticancer research 2005
Yoshinori Nio Hiroshi Omori Koji Hashimoto Masayuki Itakura Makoto Koike Seiji Yano Tomoko Toga Tetsuya Higami Riruke Maruyama

BACKGROUND The receptor tyrosine kinase c-kit is known to play an important role in the progression of gastrointestinal stromal tumors, but its biological significance in other solid malignancies is unclear. Recent publications have suggested a regulatory role for TGF-beta1 in c-kit-mediated cell growth. The present study assessed the clinicopathological significance of c-kit protein (KIT) and ...

2017
Carlos Eduardo Fonseca-Alves Priscilla Emiko Kobayashi Chiara Palmieri Renée Laufer-Amorim

BACKGROUND c-KIT expression has been related to bone metastasis in human prostate cancer, but whether c-KIT expression can be similarly classified in canine prostatic tissue is unknown. This study assessed c-KIT and Ki67 expression in canine prostate cancer (PC). c-KIT gene and protein expression and Ki67 expression were evaluated in forty-four canine prostatic tissues by immunohistochemistry, ...

2014
Yuuki Obata Shota Toyoshima Ei Wakamatsu Shunichi Suzuki Shuhei Ogawa Hiroyasu Esumi Ryo Abe

Kit is a receptor-type tyrosine kinase found on the plasma membrane. It can transform mast cells through activating mutations. Here, we show that a mutant Kit from neoplastic mast cells from mice, Kit(D814Y), is permanently active and allows cells to proliferate autonomously. It does so by activating two signalling pathways from different intracellular compartments. Mutant Kit from the cell sur...

Journal: :Journal of virology 2001
Q Zhong P Oliver W Huang D Good V La Russa Z Zhang J R Cork R W Veith C Theodossiou J K Kolls P Schwarzenberger

We have previously reported effective gene transfer with a targeted molecular conjugate adenovirus vector through the c-kit receptor in hematopoietic progenitor cell lines. However, a c-kit-targeted recombinant retroviral vector failed to transduce cells, indicating the existence of significant differences for c-kit target gene transfer between these two viruses. Here we demonstrate that conjug...

Journal: :Cancer cell 2010
Shujun Liu Lai-Chu Wu Jiuxia Pang Ramasamy Santhanam Sebastian Schwind Yue-Zhong Wu Christopher J Hickey Jianhua Yu Heiko Becker Kati Maharry Michael D Radmacher Chenglong Li Susan P Whitman Anjali Mishra Nicole Stauffer Anna M Eiring Roger Briesewitz Robert A Baiocchi Kenneth K Chan Peter Paschka Michael A Caligiuri John C Byrd Carlo M Croce Clara D Bloomfield Danilo Perrotti Ramiro Garzon Guido Marcucci

The biologic and clinical significance of KIT overexpression that associates with KIT gain-of-function mutations occurring in subsets of acute myeloid leukemia (AML) (i.e., core binding factor AML) is unknown. Here, we show that KIT mutations lead to MYC-dependent miR-29b repression and increased levels of the miR-29b target Sp1 in KIT-driven leukemia. Sp1 enhances its own expression by partici...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2007
Teresa Guida Suresh Anaganti Livia Provitera Richard Gedrich Elizabeth Sullivan Scott M Wilhelm Massimo Santoro Francesca Carlomagno

PURPOSE Targeting of KIT and platelet-derived growth factor receptor (PDGFR) tyrosine kinases by imatinib is an effective anticancer strategy. However, mutations of the gatekeeper residue (T670 in KIT and T681 in PDGFRbeta) render the two kinases resistant to imatinib. The aim of this study was to evaluate whether sorafenib (BAY 43-9006), a multitargeted ATP-competitive inhibitor of KIT and PDG...

Journal: :Transfusion 2010
Laura Cooling Sandra Hoffmann Michelle Herrst Charles Muck Heidi Armelagos Robertson Davenport

BACKGROUND The COBE Spectra AutoPBSC collection set (AUTO-kit; CaridianBCT) is a popular dual-stage collection set for peripheral blood progenitor (PBPC) collection. Although the AUTO-kit is purportedly equivalent to the white blood cell (WBC) collection set (WBC-kit) for PBPC collection, improved CD34 yields after switching from the AUTO-kit to the WBC-kit were anecdotally observed, particular...

2013
Yuan-Shuo Hsueh Chueh-Chuan Yen Neng-Yao Shih Nai-Jung Chiang Chien-Feng Li Li-Tzong Chen

Gastrointestinal stromal tumor (GIST) is a prototype of mutant KIT oncogene-driven tumor. Prolonged tyrosine kinase inhibitor (TKI) treatment may result in a resistant phenotype through acquired secondary KIT mutation. Heat shock protein 90 (HSP90AA1) is a chaperone protein responsible for protein maturation and stability, and KIT is a known client protein of HSP90AA1. Inhibition of HSP90AA1 ha...

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