نتایج جستجو برای: kcnj11

تعداد نتایج: 484  

2011
Chloe Y. Y. Cheung Annette W. K. Tso Bernard M. Y. Cheung Aimin Xu Carol H. Y. Fong K. L. Ong Lawrence S. C. Law Nelson M. S. Wat Edward D. Janus Pak C. Sham Karen S. L. Lam

CONTEXT The KCNJ11 E23K variant is associated with type 2 diabetes mellitus (T2DM) in cross-sectional studies, but conflicting findings have been reported from prospective studies. OBJECTIVE This study aimed to evaluate whether the E23K variant could predict glycaemic progression in a Southern Chinese population. METHODS/PRINCIPAL FINDINGS We performed a long-term prospective study on 1912 ...

Journal: :Philosophical transactions of the Royal Society of London. Series B, Biological sciences 2009
Timothy J Nelson Almudena Martinez-Fernandez Andre Terzic

KCNJ11-encoded Kir6.2 assembles with ATP-binding cassette sulphonylurea receptors to generate ATP-sensitive K+ (KATP) channel complexes. Expressed in tissues with dynamic metabolic flux, these evolutionarily conserved yet structurally and functionally unique heteromultimers serve as high-fidelity rheostats that adjust membrane potential-dependent cell functions to match energetic demand. Geneti...

2016
Sevim Ünal Deniz Gönülal Ahmet Uçaktürk Betül Siyah Bilgin Sarah E. Flanagan Fatih Gürbüz Meltem Tayfun Selin Elmaoğulları Aslıhan Araslı Fatma Demirel Sian Ellard Khalid Hussain

Congenital hyperinsulinism (CHI) is the most common cause of neonatal persistent hypoglycemia caused by mutations in nine known genes. Early diagnosis and treatment are important to prevent brain injury. The clinical presentation and response to pharmacological therapy may vary depending on the underlying pathology. Genetic analysis is important in the diagnosis, treatment, patient follow-up, a...

2017
Yen-Yu Yang Roger K Long Christine T Ferrara Stephen E Gitelman Michael S German Shi-Bing Yang

The ATP-sensitive potassium channel (KATP) functions as a metabo-electric transducer in regulating insulin secretion from pancreatic β-cells. The pancreatic KATP channel is composed of a pore-forming inwardly-rectifying potassium channel, Kir6.2, and a regulatory subunit, sulphonylurea receptor 1 (SUR1). Loss-of-function mutations in either subunit often lead to the development of persistent hy...

2012
Anders H. Rosengren Matthias Braun Taman Mahdi Sofia A. Andersson Mary E. Travers Makoto Shigeto Enming Zhang Peter Almgren Claes Ladenvall Annika S. Axelsson Anna Edlund Morten Gram Pedersen Anna Jonsson Reshma Ramracheya Yunzhao Tang Jonathan N. Walker Amy Barrett Paul R.V. Johnson Valeriya Lyssenko Mark I. McCarthy Leif Groop Albert Salehi Anna L. Gloyn Erik Renström Patrik Rorsman Lena Eliasson

The majority of genetic risk variants for type 2 diabetes (T2D) affect insulin secretion, but the mechanisms through which they influence pancreatic islet function remain largely unknown. We functionally characterized human islets to determine secretory, biophysical, and ultrastructural features in relation to genetic risk profiles in diabetic and nondiabetic donors. Islets from donors with T2D...

Journal: :Acta pharmaceutica 2010
Una Glamočlija Adlija Jevrić-Čaušević

Due to new genetic insights, etiologic classification of diabetes is under constant scrutiny. Hundreds, or even thousands, of genes are linked with type 2 diabetes. Three common variants (Lys23 of KCNJ11, Pro12 of PPARG, and the T allele at rs7903146 of TCF7L2) have been shown to be predisposed to type 2 diabetes mellitus across many large studies. Individually, each of these polymorphisms is o...

2016
Saliha Rizvi Syed Tasleem Raza Qamar Rahman Farzana Mahdi

Type 2 diabetes (T2DM) is a polygenic metabolic disorder characterized by hyperglycemia occurring as a result of impaired insulin secretion or insulin resistance. Various environmental and genetic factors interact and increase the risk of T2DM and its complications. Among the various genetic factors associated with T2DM, single nucleotide polymorphism in different candidate genes have been stud...

2012
Mohammad Fatehi Mobeen Raja Christian Carter Daniel Soliman Andrew Holt Peter E. Light

Pancreatic β-cell ATP-sensitive K(+) (K(ATP)) channels are composed of Kir6.2 and SUR1 subunits encoded by the KCNJ11 and ABCC8 genes, respectively. Although rare monogenic activating mutations in these genes cause overt neonatal diabetes, the common variants E23K (KCNJ11) and S1369A (ABCC8) form a tightly heritable haplotype that is associated with an increased susceptibility to type 2 diabete...

Journal: :Orphanet Journal of Rare Diseases 2007
Michel Polak Hélène Cavé

Transient (TNDM) and Permanent (PNDM) Neonatal Diabetes Mellitus are rare conditions occurring in 1:300,000-400,000 live births. TNDM infants develop diabetes in the first few weeks of life but go into remission in a few months, with possible relapse to a permanent diabetes state usually around adolescence or as adults. The pancreatic dysfunction in this condition may be maintained throughout l...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید