نتایج جستجو برای: gp91

تعداد نتایج: 611  

Journal: :Environmental Health Perspectives 1998
J R Hoidal P Xu T Huecksteadt K A Sanders K Pfeffer A B Sturrock

Acute lung injury represents a wide spectrum of pathologic processes, the most severe end of the spectrum being the acute respiratory distress syndrome. Reactive oxygen intermediates have been implicated as important in the pathobiochemistry of acute lung injury. The endogenous sources that contribute to the generation of reactive oxygen intermediates in acute lung injury are poorly defined but...

Journal: :Circulation research 2002
Hanke Mollnau Maria Wendt Katalin Szöcs Bernard Lassègue Eberhard Schulz Mathias Oelze Huige Li Martin Bodenschatz Michael August Andrei L Kleschyov Nikolaus Tsilimingas Ulrich Walter Ulrich Förstermann Thomas Meinertz Kathy Griendling Thomas Münzel

Angiotensin II infusion causes endothelial dysfunction by increasing NAD(P)H oxidase-mediated vascular superoxide production. However, it remains to be elucidated how in vivo angiotensin II treatment may alter the expression of the gp91(phox) isoforms and the endothelial nitric oxide synthase (NOS III) and subsequent signaling events and whether, in addition to the NAD(P)H oxidase, NOS III cont...

Journal: :The Journal of biological chemistry 2005
Noriko Ueno Ryu Takeya Kei Miyano Hideaki Kikuchi Hideki Sumimoto

Nox3, a member of the superoxide-producing NADPH oxidase (Nox) family, participates in otoconia formation in mouse inner ears, which is required for perception of balance and gravity. The activity of other Nox enzymes such as gp91(phox)/Nox2 and Nox1 is known to absolutely require both an organizer protein (p47(phox) or Noxo1) andanactivatorprotein (p67(phox) or Noxa1); for the p47(phox)-depend...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1999
S L Archer H L Reeve E Michelakis L Puttagunta R Waite D P Nelson M C Dinauer E K Weir

The rapid response to hypoxia in the pulmonary artery (PA), carotid body, and ductus arteriosus is partially mediated by O2-responsive K+ channels. K+ channels in PA smooth muscle cells (SMCs) are inhibited by hypoxia, causing membrane depolarization, increased cytosolic calcium, and hypoxic pulmonary vasoconstriction. We hypothesize that the K+ channels are not themselves "O2 sensors" but rath...

2005
Paul Barrow Neil Foster Scott Hulme Margaret Lovell Katharine Reed

10.1128/IAI.73.8.4539-4547.2005. 2005, 73(8):4539. DOI: Infect. Immun. and Paul Barrow Neil Foster, Scott Hulme, Margaret Lovell, Katharine Reed without Inducing Intestinal Pathology with Serovar Typhimurium Strain F98 Gnotobiotic Piglets from Lethal Challenge Serovar Infantis Strain Protects enterica Salmonella Neutrophils by an Avirulent and Reactive Oxygen Species in Stimulation of gp91 Phag...

2002
Thomas Münzel Ulrich Walter Ulrich Förstermann Thomas Meinertz Kathy Griendling Hanke Mollnau Maria Wendt Katalin Szöcs Bernard Lassègue Eberhard Schulz Mathias Oelze Huige Li Martin Bodenschatz Michael August Andrei L. Kleschyov Nikolaus Tsilimingas

Angiotensin II infusion causes endothelial dysfunction by increasing NAD(P)H oxidase–mediated vascular superoxide production. However, it remains to be elucidated how in vivo angiotensin II treatment may alter the expression of the gp91 isoforms and the endothelial nitric oxide synthase (NOS III) and subsequent signaling events and whether, in addition to the NAD(P)H oxidase, NOS III contribute...

Journal: :The Journal of General Physiology 2002
Thomas E. DeCoursey Deri Morgan Vladimir V. Cherny

During the “respiratory burst” in phagocytes, NADPH oxidase helps kill microbes by producing superoxide anion, O 2 . As illustrated in the cartoon in Fig. 1, the NADPH oxidase complex has several components. In unstimulated cells, four components (p67 phox , p40 phox , p47 phox , and a G protein, Rac) are located in the cytosol, and gp91 phox and p22 phox are membrane bound. Upon stimulation by...

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