نتایج جستجو برای: founder effect

تعداد نتایج: 1647862  

2014
M. Chiara Manzini Lan Xiong Ranad Shaheen Dimira E. Tambunan Stefania Di Costanzo Vanessa Mitisalis David J. Tischfield Antonella Cinquino Mohammed Ghaziuddin Mehtab Christian Qin Jiang Sandra Laurent Zohair A. Nanjiani Saima Rasheed R. Sean Hill Sofia B. Lizarraga Danielle Gleason Diya Sabbagh Mustafa A. Salih Fowzan S. Alkuraya Christopher A. Walsh

Autism spectrum disorder (ASD) and intellectual disability (ID) are often comorbid, but the extent to which they share common genetic causes remains controversial. Here, we present two autosomal-recessive "founder" mutations in the CC2D1A gene causing fully penetrant cognitive phenotypes, including mild-to-severe ID, ASD, as well as seizures, suggesting shared developmental mechanisms. CC2D1A r...

Journal: :Ecology letters 2007
Patrick C Tobin Stefanie L Whitmire Derek M Johnson Ottar N Bjørnstad Andrew M Liebhold

Allee effects can play a critical role in slowing or preventing the establishment of low density founder populations of non-indigenous species. Similarly, the spread of established invaders into new habitats can be influenced by the degree to which small founder populations ahead of the invasion front are suppressed through Allee effects. We develop an approach to use empirical data on the gyps...

2017
Geoffrey P Martin Luis R Gomez-Mejia

We examine the unique nature of agency problems within publicly traded family firms by investigating the earnings management decision of dominant family owners relative to nonfamily. To do so, we draw upon literature demonstrating that family owners are loss averse with respect to the family’s socioemotional wealth (SEW), or the affective endowment derived from firm ownership and control. Our t...

Journal: :Human molecular genetics 1998
A Bernot C da Silva J L Petit C Cruaud C Caloustian V Castet M Ahmed-Arab C Dross M Dupont D Cattan N Smaoui C Dodé C Pêcheux B Nédelec J Medaxian M Rozenbaum I Rosner M Delpech G Grateau J Demaille J Weissenbach I Touitou

Familial Mediterranean fever (FMF) is an autosomal recessive disorder characterized by recurring attacks of fever and serositis. It affects primarily North African Jews, Armenians, Turks and Arabs, in which a founder effect has been demonstrated. The marenostrin-pyrin-encoding gene has been proposed as a candidate gene for the disease ( MEFV ), on the basis of the identification of putative mut...

2012
Lilia Romdhane Rym Kefi Hela Azaiez Nizar Ben Halim Koussay Dellagi Sonia Abdelhak

BACKGROUND Tunisia is a North African country of 10 million inhabitants. The native background population is Berber. However, throughout its history, Tunisia has been the site of invasions and migratory waves of allogenic populations and ethnic groups such as Phoenicians, Romans, Vandals, Arabs, Ottomans and French. Like neighbouring and Middle Eastern countries, the Tunisian population shows a...

Journal: :Genetics 2006
Ruth G Shaw Shu-Mei Chang

For a newly arising mutation affecting a trait under selection, its degree of dominance relative to the preexisting allele(s) strongly influences its evolutionary impact. We have estimated dominance parameters for spontaneous mutations in a subset of lines derived from a highly inbred founder of Arabidopsis thaliana by at least 17 generations of mutation accumulation (MA). The labor-intensive n...

2014
Ahlem Amouri Faten Talmoudi Olfa Messaoud Catherine D d'Enghien Mariem B Rekaya Ines Allegui Héla Azaiez Rym Kefi Ahlem Abdelhak Sondes H Meseddi Lamia Torjemane Monia Ouederni Fethi Mellouli Héla B Abid Lamia Aissaoui Mohamed Bejaoui Tarek B Othmen Dominique S Lyonnet Jean Soulier Mongia Hachicha Koussay Dellagi Sonia Abdelhak Tunisian Fanconi

Tunisian population is characterized by its heterogeneous ethnic background and high rate of consanguinity. In consequence, there is an increase in the frequency of recessive genetic disorders including Fanconi anemia (FA). The aim of this study was to confirm the existence of a founder haplotype among FA Tunisian patients and to identify the associated mutation in order to develop a simple too...

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