نتایج جستجو برای: dna methyltransferase mgmt

تعداد نتایج: 522903  

Journal: :Endocrine-related cancer 2016
J Cros O Hentic V Rebours M Zappa N Gille N Theou-Anton D Vernerey F Maire P Lévy P Bedossa V Paradis P Hammel P Ruszniewski A Couvelard

Temozolomide (TEM) showed encouraging results in well-differentiated pancreatic neuroendocrine tumors (WDPNETs). Low O(6)-methylguanine-DNA methyltransferase (MGMT) expression and MGMT promoter methylation within tumors correlate with a better outcome under TEM-based chemotherapy in glioblastoma. We aimed to assess whether MGMT expression and MGMT promoter methylation could help predict the eff...

Journal: :Cancer research 2002
Frank D Gilliland Heidi J Harms Richard E Crowell Yu-Fen Li Randy Willink Steven A Belinsky

Inactivation of the p16(INK4a) tumor suppressor gene and O(6)-methylguanine-DNA methyltransferase (MGMT) DNA repair gene by aberrant promoter methylation appears to be an important step in respiratory carcinogenesis after exposure to tobacco smoke and radon progeny. The determinants of aberrant promoter methylation are not well characterized. Polymorphic variants of genes of which the products ...

Journal: :Asian Pacific journal of cancer prevention : APJCP 2014
Leila Larijani Zahra Madjd Ali Samadikuchaksaraei Shima Younespour Hanieh Zham Azadeh Rakhshan Foruzan Mohammadi Ali Rahbari Afshin Moradi

BACKGROUND Meningiomas are the second most common primary intracranial tumors after gliomas. Epigenetic biomarkers such as DNA methylation, which is found in many tumors and is thus important in tumorigenesis can help diagnose meningiomas and predict response to adjuvant chemotherapy. We investigated aberrant O6- methyl guanine methyltransferase (MGMT) methylation in meningiomas. MATERIALS AN...

2002
Frank D. Gilliland Heidi J. Harms Richard E. Crowell Yu-Fen Li Randy Willink Steven A. Belinsky

Inactivation of the p16 tumor suppressor gene and O-methylguanine-DNA methyltransferase (MGMT) DNA repair gene by aberrant promoter methylation appears to be an important step in respiratory carcinogenesis after exposure to tobacco smoke and radon progeny. The determinants of aberrant promoter methylation are not well characterized. Polymorphic variants of genes of which the products are involv...

Journal: :Neuro-oncology 2010
George C Bobustuc Cheryl H Baker Arati Limaye Wayne D Jenkins Gary Pearl Nicholas G Avgeropoulos Santhi D Konduri

Antiepileptic drugs (AEDs) are frequently used to treat seizures in glioma patients. AEDs may have an unrecognized impact in modulating O(6)-methylguanine-DNA methyltransferase (MGMT), a DNA repair protein that has an important role in tumor cell resistance to alkylating agents. We report that levetiracetam (LEV) is the most potent MGMT inhibitor among several AEDs with diverse MGMT regulatory ...

Journal: :Hormones 2011
Olga Moshkin Luis V Syro Bernd W Scheithauer Leon D Ortiz Camilo E Fadul Humberto Uribe Ricardo Gonzalez Michael Cusimano Eva Horvath Fabio Rotondo Kalman Kovacs

Temozolomide (TMZ) has recently been recommended as a novel approach in the management of aggressive pituitary tumors. Herein, we present the case of a 43-year-old man with a 20-year history of silent subtype 2 pituitary corticotroph adenoma. Nine surgical resections and radiotherapy had failed to provide a cure. Morphological evaluation of the tumor revealed a mildly pleomorphic adenoma, the c...

Journal: :Carcinogenesis 1996
T Grombacher S Mitra B Kaina

Repair of alkylated bases in DNA is performed by O6-methylguanine-DNA methyltransferase (MGMT) and a set of enzymes of the base excision repair pathway involving N-methylpurine-DNA glycosylase (MPG), apurinic endonuclease (APE), DNA polymerase beta (Pol beta) and DNA ligase. The level of expression of these enzymes may exert a profound effect on resistance of cells towards alkylating drugs. We ...

Journal: :Cancer research 2008
Michael D Milsom Moran Jerabek-Willemsen Chad E Harris Axel Schambach Emily Broun Jeff Bailey Michael Jansen David Schleimer Kalpana Nattamai Jamie Wilhelm Amanda Watson Hartmut Geiger Geoffrey P Margison Thomas Moritz Christopher Baum Jürgen Thomale David A Williams

Retroviral-mediated delivery of the P140K mutant O(6)-methylguanine-DNA methyltransferase (MGMT(P140K)) into hematopoietic stem cells (HSC) has been proposed as a means to protect against dose-limiting myelosuppressive toxicity ensuing from chemotherapy combining O(6)-alkylating agents (e.g., temozolomide) with pseudosubstrate inhibitors (such as O(6)-benzylguanine) of endogenous MGMT. Because ...

Journal: :Journal of neurochemistry 2012
Caroline Happold Patrick Roth Wolfgang Wick Natalie Schmidt Ana-Maria Florea Manuela Silginer Guido Reifenberger Michael Weller

Temozolomide (TMZ) is an alkylating chemotherapeutic agent that prolongs the survival of patients with glioblastoma. Clinical benefit is more prominent in patients with methylation of the O(6) -methyl-guanine DNA methyltransferase (MGMT) promoter. However, all patients eventually suffer from tumor progression because their tumors become resistant to TMZ. Here, we modeled acquired TMZ resistance...

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