نتایج جستجو برای: collagen type vi

تعداد نتایج: 1418081  

2014
Andrezza C. Chagas Peter McPhie Hong San David Narum Karine Reiter Fuyuki Tokomasu Fabio A. Brayner Luiz C. Alves José M. C. Ribeiro Eric Calvo

BACKGROUND Among the several challenges faced by bloodsucking arthropods, the vertebrate hemostatic response against blood loss represents an important barrier to efficient blood feeding. Here we report the first inhibitor of collagen-induced platelet aggregation derived from the salivary glands of a black fly (Simulium nigrimanum), named Simplagrin. METHODS AND FINDINGS Simplagrin was expres...

Journal: :Human molecular genetics 1996
M C Speer R Tandan P N Rao T Fries J M Stajich P A Bolhuis G J Jöbsis J M Vance K D Viles K Sheffield C James S G Kahler M Pettenati J R Gilbert P H Denton L H Yamaoka M A Pericak-Vance

The Bethlem myopathy, a childhood onset autosomal dominant myopathy with joint contractures, has recently been localized to 21q in a series of Dutch families and the alpha 1 and alpha 2 subunits of type VI collagen (COL6A1 and COL6A2) have been postulated as candidate genes. We investigate a large family of French Canadian descent (family 1489) in which the Bethlem myopathy is segregating. Fami...

2014
Véronique Bolduc Yaqun Zou Dayoung Ko Carsten G Bönnemann

Congenital muscular dystrophy type Ullrich (UCMD) is a severe disorder of early childhood onset for which currently there is no effective treatment. UCMD commonly is caused by dominant-negative mutations in the genes coding for collagen type VI, a major microfibrillar component of the extracellular matrix surrounding the muscle fibers. To explore RNA interference (RNAi) as a potential therapy f...

Journal: :iranian journal of basic medical sciences 0
elham mohammadzadeh 1department of anatomy and cell biology, school of medicine, mashhad university of medical sciences, mashhad, iran mohammad reza nikravesh 1department of anatomy and cell biology, school of medicine, mashhad university of medical sciences, mashhad, iran mehdi jalali 1department of anatomy and cell biology, school of medicine, mashhad university of medical sciences, mashhad, iran alireza fazel 1department of anatomy and cell biology, school of medicine, mashhad university of medical sciences, mashhad, iran vahid ebrahimi 1department of anatomy and cell biology, school of medicine, mashhad university of medical sciences, mashhad, iran ali reza ebrahimzadeh-bideskan 1department of anatomy and cell biology, school of medicine, mashhad university of medical sciences, mashhad, iran

objective(s):skin extracellular matrix, which contains type i and type iii collagens, is involved in skin development. the aim of this study was to investigate type iii collagen distribution pattern as well as its changes during pre and post-natal skin morphogenesis in rats. materials and methods: ventral skins of wistar rat embryos at different stages from 10 to 20 gestational day (e10-e20) an...

2014
LARRY VOILES DAVID E. LEWIS LING HAN IVAN P. LUPOV TSANG-LONG LIN MICHAEL J. ROBERTSON IRINA PETRACHE HUA-CHEN CHANG

Type VI collagen (COL6), an extracellular matrix protein, is important in maintaining the integrity of lung tissue. An increase in COL6 mRNA and protein deposition was found in the lungs of patients with pulmonary fibrosis, a chronic inflammatory condition with a strong association with lung cancer. In the present study, we demonstrated overexpression of COL6 in the lungs of non-small cell lung...

Journal: :Journal of orthopaedic research : official publication of the Orthopaedic Research Society 2009
Roman M Natoli Donald J Responte Benjamin Y Lu Kyriacos A Athanasiou

Increasing tensile properties and collagen content is a recognized need in articular cartilage tissue engineering. This study tested the hypothesis that multiple applications of chondroitinase ABC (C-ABC), a glycosaminoglycan (GAG) degrading enzyme, could increase construct tensile properties in a scaffold-less approach for articular cartilage tissue engineering. Developing constructs were trea...

Journal: :Human molecular genetics 2005
Naomi L Baker Matthias Mörgelin Rachel Peat Nathalie Goemans Kathryn N North John F Bateman Shireen R Lamandé

Mutations in the three collagen VI genes COL6A1, COL6A2 and COL6A3 cause Bethlem myopathy and Ullrich congenital muscular dystrophy (UCMD). UCMD, a severe disorder characterized by congenital muscle weakness, proximal joint contractures and marked distal joint hyperextensibility, has been considered a recessive condition, and homozygous or compound heterozygous mutations have been defined in CO...

Journal: :Investigative ophthalmology & visual science 2002
Jun Ueda Kelly Wentz-Hunter Beatrice Y J T Yue

PURPOSE To examine ultrastructurally the composition of extracellular matrix (ECM) materials, the distribution of myocilin, and the colocalization of myocilin with ECM components in the juxtacanalicular tissue (JCT) of normal human eyes. METHODS Postembedding immunoelectron microscopic studies were performed with antibodies specific for major ECM components, including fibronectin, laminin, vi...

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