نتایج جستجو برای: cmt1a

تعداد نتایج: 183  

Journal: :Neuromuscular disorders : NMD 2004
Ian J Sutton A Paul Mocroft Victoria H Lindley Richard M Barber R Jane Bryon John B Winer Fiona MacDonald

Hereditary neuropathy with liability to pressure palsies arises as a result of defects at the chromosome 17p11.2-12 locus and in 84% of cases a 1.5 Mb deletion containing the PMP22 gene is detected by analysis that utilises polymorphic (CA)n repeat markers which flank this gene. We report the clinical and electrophysiological findings observed in a kindred with three members affected by HNPP du...

Journal: :The Cochrane database of systematic reviews 2014
Claire M White Pieter A van Doorn Marcel P J Garssen Rachel C Stockley

BACKGROUND Persistent feelings of fatigue (or subjective fatigue), which may be experienced in the absence of physiological factors, affect many people with peripheral neuropathy. A variety of interventions for subjective fatigue are available, but little is known about their efficacy or the likelihood of any adverse effects for people with peripheral neuropathy. OBJECTIVES To assess the effe...

Journal: :Neuromuscular disorders : NMD 2011
M Russo M Laurá J M Polke M B Davis J Blake S Brandner R A C Hughes H Houlden D L H Bennett M P T Lunn M M Reilly

Charcot-Marie-Tooth disease (CMT) is the commonest hereditary neuropathy encompassing a large group of clinically and genetically heterogeneous disorders. The commonest form of CMT, CMT1A, is usually caused by a 1.4 megabase duplication of chromosome 17 containing the PMP22 gene. Mutations of PMP22 are a less common cause of CMT. We describe clinical, electrophysiological and molecular findings...

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