نتایج جستجو برای: caga gene

تعداد نتایج: 1142254  

Journal: :Memorias do Instituto Oswaldo Cruz 2005
Luisa Caricio Martins Tereza Cristina de Oliveira Corvelo Samia Demachki Marialva T F Araujo Mônica Baraúna Assumpção Simone Cristina Araujo Jucá Vilar Felipe Bonfim Freitas Hivana Patricia Melo Barbosa Amanda Alves Fecury Renata Kelly Costa do Amaral Sidney Emanuel Batista Dos Santos

We have examined the prevalence of gene cagA and vacA alleles in 129 patients, 69 with gastritis and 60 with peptic ulcer diseases from North Brazil and their relation with histopathological data. vacA and cagA genotype were determined by polymerase chain reaction. Hematoxylin-eosin staining was used for histological diagnosis. 96.6% of the patients were colonized by Helicobacter pylori strains...

2016
Rony K. Roy Michal M. Hoppe Supriya Srivastava Animesh Samanta Neel Sharma Kar Tong Tan Henry Yang Dominic C. Voon Brendan Pang Ming Teh Naoko Murata-Kamiya Masanori Hatakeyama Young-Tae Chang Wei Peng Yong Yoshiaki Ito Khek Yu Ho Patrick Tan Richie Soong Phillip H. Koeffler Khay Guan Yeoh Anand D. Jeyasekharan

Early detection of gastric cancers saves lives, but remains a diagnostic challenge. In this study, we aimed to identify cell-surface biomarkers of early gastric cancer. We hypothesized that a subset of plasma membrane proteins induced by the Helicobacter pylori oncoprotein CagA will be retained in early gastric cancers through non-oncogene addiction. An inducible system for expression of CagA w...

Journal: :Indian journal of gastroenterology : official journal of the Indian Society of Gastroenterology 2009
Khalid Mubarak Bindayna Ali Al Mahmeed

BACKGROUND The vacuolating cytotoxin and the cytotoxinassociated protein, encoded by vacA and cagA, respectively, are important virulence determinants of Helicobacter pylori. OBJECTIVE The aim of this study was to perform vacA genotyping and evaluate its association with cagA genotype and clinical outcome. METHODS One hundred and twenty H. pylori strains were isolated from dyspeptic patient...

2010
Song-Ze Ding Wolfgang Fischer Maria Kaparakis-Liaskos George Liechti D. Scott Merrell Patrick A. Grant Richard L. Ferrero Sheila E. Crowe Rainer Haas Masanori Hatakeyama Joanna B. Goldberg

Histone modifications are critical in regulating gene expression, cell cycle, cell proliferation, and development. Relatively few studies have investigated whether Helicobacter pylori, the major cause of human gastric diseases, affects histone modification. We therefore investigated the effects of H. pylori infection on histone modifications in a global and promoter-specific manner in gastric e...

Journal: :Journal of clinical microbiology 2000
J Yahav A Fradkin B Weisselberg A Diver-Haver H Shmuely A Jonas

A potential virulence determinant of Helicobacter pylori is the cagA gene product. To determine the relevance of the expression of CagA to the clinical picture and outcome of H. pylori infection in children, we examined 104 consecutive children diagnosed with H. pylori infection. Serum samples were collected to test for the presence of immunoglobulin G (IgG) anti-CagA antibodies. Forty-five pat...

Journal: :Carcinogenesis 2002
Atsuko Shibata Julie Parsonnet Teri A Longacre Maria Isabel Garcia Balaram Puligandla R Eric Davis Joseph H Vogelman Norman Orentreich Laurel A Habel

Infection with Helicobacter pylori (H. pylori) increases stomach cancer risk. Helicobacter pylori strains with the cag pathogenicity island (PAI) induce more severe inflammation in the gastric epithelium and are more strongly associated with stomach cancer risk than strains lacking the PAI. We examined whether the prevalence of somatic p53 mutation in gastric adenocarcinoma differed between sub...

Journal: :The Journal of Experimental Medicine 2000
Momoyo Asahi Takeshi Azuma Shigeji Ito Yoshiyuki Ito Hiroyuki Suto Yukifumi Nagai Misao Tsubokawa Yumi Tohyama Shin Maeda Masao Omata Toshihiko Suzuki Chihiro Sasakawa

Attachment of Helicobacter pylori to gastric epithelial cells induces various cellular responses, including the tyrosine phosphorylation of an unknown 145-kD protein and interleukin 8 production. Here we show that this 145-kD protein is the cagA product of H. pylori, an immunodominant, cytotoxin-associated antigen. Epithelial cells infected with various H. pylori clinical isolates resulted in g...

Journal: :Gut 2000
P Y Zheng J Hua K G Yeoh B Ho

BACKGROUND Studies in Western populations suggest that cagA, iceA, and vacA gene status in Helicobacter pylori isolates is associated with increased virulence and peptic ulcer disease. AIM To investigate the relationship between peptic ulcer and expression of Lewis (Le) antigens as well as cagA, iceA, and vacA in H pylori isolates in Singapore. METHODS Expression of Le antigens in H pylori ...

2015
Fatemeh Sayehmiri Faezeh Kiani Kourosh Sayehmiri Setareh Soroush Khairollah Asadollahi Mohammad Yousef Alikhani Ali Delpisheh Mohammad Emaneini Lidija Bogdanović Ali Mohammad Varzi Raffaele Zarrilli Morovat Taherikalani

The varieties of infections caused by Helicobacter pylori may be due to differences in bacterial genotypes and virulence factors as well as environmental and host-related factors. This study aimed to investigate the prevalence of cagA and vacA genes among H. pylori-infected patients in Iran and analyze their relevance to the disease status between two clinical groups via a meta-analysis method....

2015
Thuy Trang Nguyen Seong-Jin Kim Jong Min Park Ki Baik Hahm Ho-Jae Lee

Helicobacter pylori (H. pylori) infection causes chronic gastric inflammation, peptic ulceration, and gastric carcinogenesis, in which H. pylori cytotoxin-associated gene A (CagA) plays major pathogenic action. Since transforming growth factor-β (TGF-β) and its signaling also are principally implicated in either modulating gastric mucosal inflammatory responses or causing carcinogenesis and are...

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