نتایج جستجو برای: myotonia

تعداد نتایج: 1446  

Journal: :Medical History 1968

Journal: :Proceedings of the Royal Society of Medicine 1923

Journal: :Journal of General Physiology 1996

Journal: :Journal of Neurology, Neurosurgery & Psychiatry 1978

Journal: :Journal of neurology, neurosurgery, and psychiatry 1959
J WILSON J N WALTON

Although it has been recognized for many years that symptoms and signs indicating disordered function of the voluntary musculature may be prominent in patients with myxoedema, the muscular manifestations of hypothyroidism have received comparatively little attention in the British literature. Recent reviews have, however, been given by Thomasen (1948), Piaggio Blancho and Malosetti (1950), Mill...

Journal: :Arquivos de neuro-psiquiatria 2003
Rosana Herminia Scola Fabio Massaiti Iwamoto Carlos Henrique Camargo Walter Oleschko Arruda Lineu Cesar Werneck

Approximately 1-2% of the population has a deficiency of the enzyme myoadenylate deaminase. Early reports suggested that patients with myoadenylate deaminase deficiency had various forms of myalgia, and exercise intolerance. However, a deficiency of the enzyme has been described in many conditions, including myopathies, neuropathies, and motor neuron disease. We report a patient with clinical d...

Journal: :Journal of medical genetics 1972
P S Harper D M Johnston

2015
Jongkyu Choi Kirkwood E. Personius Marino DiFranco Warunee Dansithong Carl Yu Saumya Srivastava Donald M. Dixon Darshan B. Bhatt Lucio Comai Julio L. Vergara Sita Reddy

UNLABELLED Loss of Muscleblind-like 1 (Mbnl1) is known to alter Clc-1 splicing to result in myotonia. Mbnl1(ΔE3/ΔE3)/Mbnl3(ΔE2) mice, depleted of Mbnl1 and Mbnl3, demonstrate a profound enhancement of myotonia and an increase in the number of muscle fibers with very low Clc-1 currents, where gClmax values approach ~ 1 mS/cm(2), with the absence of a further enhancement in Clc-1 splice errors, a...

Journal: :Journal of neurology, neurosurgery, and psychiatry 1987
R Grant D L Sutton P O Behan J P Ballantyne

Abnormal calcium transport may be implicated in the membrane defect in myotonic dystrophy. A single blind crossover trial of placebo (t.i.d.), nifedipine 10 mg (t.i.d.) and nifedipine 20 mg (t.i.d.), was performed in 10 patients with myotonic dystrophy. The severity of myotonia was assessed by measuring finger extension time after maximum voluntary finger flexion. A significant improvement in m...

2009
Cristina Bulli Pier Antonio Battistella Marta Bordignon Placido Bramanti Giuseppe Novelli Federica Sangiuolo

Autosomal dominant (Thomsen) and recessive (Becker) congenital myotonia are two different non dystrophic disorders, due to allelic mutations of the muscle chloride channel gene, located on chromosome 7q35. More than two thirds of the muscle chloride channel gene mutations occur independently in unique families and cause the recessive form of the disease. Becker disease is more common and severe...

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