نتایج جستجو برای: myofibroblast

تعداد نتایج: 2555  

Journal: :Kidney International 1992

Journal: :The American Journal of Pathology 2004

2012
Daniel Reis Waisberg Edwin Roger Parra João Valente Barbas-Filho Sandra Fernezlian Vera Luiza Capelozzi

OBJECTIVE This study sought to identify the relationship between fibroblast telomerase expression, myofibroblasts, and telomerase-mediated regulatory signals in idiopathic pulmonary fibrosis. METHODS Thirty-four surgical lung biopsies, which had been obtained from patients with idiopathic pulmonary fibrosis and histologically classified as usual interstitial pneumonia, were examined. Immunohi...

Journal: :Circulation 2017
Jeffery D Molkentin Darrian Bugg Natasha Ghearing Lisa E Dorn Peter Kim Michelle A Sargent Jagadambika Gunaje Kinya Otsu Jennifer Davis

BACKGROUND In the heart, acute injury induces a fibrotic healing response that generates collagen-rich scarring that is at first protective but if inappropriately sustained can worsen heart disease. The fibrotic process is initiated by cytokines, neuroendocrine effectors, and mechanical strain that promote resident fibroblast differentiation into contractile and extracellular matrix-producing m...

Journal: :Journal of Crohn's & colitis 2009
Ulrika Christerson Asa V Keita Johan D Söderholm Christina Gustafson-Svärd

BACKGROUND AND AIMS Myofibroblast hyperplasia contributes to muscularis mucosae thickening and stricture formation in Crohn's disease (CD). Protease-activated receptor-2 (PAR-2) and cytosolic phospholipase A(2) (cPLA(2)) are known regulators of cell growth, but their significance in intestinal myofibroblast proliferation remain to be elucidated. The principle aims of the present study were to i...

Journal: :American journal of physiology. Heart and circulatory physiology 2008
M Pho W Lee D R Watt C Laschinger C A Simmons C A McCulloch

The formation of myofibroblasts in valve interstitial cell (VIC) populations contributes to fibrotic valvular disease. We examined myofibroblast differentiation in VICs from porcine aortic valves. In normal valves, cells immunostained for alpha-smooth muscle actin (alpha-SMA, a myofibroblast marker) were rare (0.69 +/- 0.48%), but in sclerotic valves of animals fed an atherogenic diet, myofibro...

2017
Yusuke Kurita Jun Araya Shunsuke Minagawa Hiromichi Hara Akihiro Ichikawa Nayuta Saito Tsukasa Kadota Kazuya Tsubouchi Nahoko Sato Masahiro Yoshida Kenji Kobayashi Saburo Ito Yu Fujita Hirofumi Utsumi Haruhiko Yanagisawa Mitsuo Hashimoto Hiroshi Wakui Yutaka Yoshii Takeo Ishikawa Takanori Numata Yumi Kaneko Hisatoshi Asano Makoto Yamashita Makoto Odaka Toshiaki Morikawa Katsutoshi Nakayama Kazuyoshi Kuwano

BACKGROUND Pirfenidone (PFD) is an anti-fibrotic agent used to treat idiopathic pulmonary fibrosis (IPF), but its precise mechanism of action remains elusive. Accumulation of profibrotic myofibroblasts is a crucial process for fibrotic remodeling in IPF. Recent findings show participation of autophagy/mitophagy, part of the lysosomal degradation machinery, in IPF pathogenesis. Mitophagy has bee...

Journal: :American journal of physiology. Lung cellular and molecular physiology 2015
J L Judge K M Owens S J Pollock C F Woeller T H Thatcher J P Williams R P Phipps P J Sime R M Kottmann

Pulmonary fibrosis is a common and dose-limiting side-effect of ionizing radiation used to treat cancers of the thoracic region. Few effective therapies are available for this disease. Pulmonary fibrosis is characterized by an accumulation of myofibroblasts and excess deposition of extracellular matrix proteins. Although prior studies have reported that ionizing radiation induces fibroblast to ...

Journal: :American journal of physiology. Heart and circulatory physiology 2006
Jennifer E Naugle Erik R Olson Xiaojin Zhang Sharon E Mase Charles F Pilati Michael B Maron Hans G Folkesson Walter I Horne Kathleen J Doane J Gary Meszaros

Cardiac fibroblast (CF) proliferation and differentiation into hypersecretory myofibroblasts can lead to excessive extracellular matrix (ECM) production and cardiac fibrosis. In turn, the ECM produced can potentially activate CFs via distinct feedback mechanisms. To assess how specific ECM components influence CF activation, isolated CFs were plated on specific collagen substrates (type I, III,...

2015
Mickaël Tobalem Dominik Lévigne Ali Modarressi Fatemeh Atashi Frederic Villard Boris Hinz Brigitte Pittet-Cuénod

BACKGROUND Hyperglycemia is known to adversely affect the outcome of ischemic insults, but its interaction with ischemia has not been investigated in wound repair yet. In this study, we develop a new animal model allowing to investigate the interaction between hyperglycemia and ischemia during the wound repair process. We focus on myofibroblast differentiation, a key element of wound repair. ...

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