نتایج جستجو برای: lncap cell line

تعداد نتایج: 1969807  

Journal: :Cancer research 2007
Hector F Valenzuela Karen E Pace Paula V Cabrera Rachel White Katja Porvari Helena Kaija Pirkko Vihko Linda G Baum

Resistance to apoptosis is a critical feature of neoplastic cells. Galectin-1 is an endogenous carbohydrate-binding protein that induces death of leukemia and lymphoma cells, breast cancer cells, and the LNCaP prostate cancer cell line, but not other prostate cancer cell lines. To understand the mechanism of galectin-1 sensitivity of LNCaP cells compared with other prostate cancer cells, we cha...

2014
Xiao-Feng Gao Tie Zhou Guang-Hua Chen Chuan-Liang Xu Ye-Lei Ding Ying-Hao Sun

Radioiodine therapy, the most effective form of systemic radiotherapy available, is currently useful only for thyroid cancer because of the thyroid-specific expression of the human sodium iodide symporter (hNIS). Here, we explore the efficacy of a novel form of gene therapy using prostate-specific membrane antigen (PSMA) promoter-mediated hNIS gene transfer followed by radioiodine administratio...

Journal: :Molecular endocrinology 1997
K B Cleutjens H A van der Korput C C van Eekelen H C van Rooij P W Faber J Trapman

Prostate-specific antigen (PSA) is expressed at a high level in the luminal epithelial cells of the prostate and is absent or expressed at very low levels in other tissues. PSA expression can be regulated by androgens. Previously, two functional androgen-response elements were identified in the proximal promoter of the PSA gene. To detect additional, more distal control elements, DNasel-hyperse...

Journal: :Cancer research 2001
C W Gregory R T Johnson J L Mohler F S French E M Wilson

The androgen receptor (AR) is highly expressed in androgen-dependent and recurrent prostate cancer (CaP) suggesting it has a role in the growth and progression of CaP. Previously proposed mechanisms for AR reactivation in recurrent CaP include altered growth factor signaling leading to protein phosphorylation and AR mutations that broaden ligand specificity. To further establish a role for AR i...

2015
Ryuhei Kanaoka Akifumi Kushiyama Yasuyuki Seno Yusuke Nakatsu Yasuka Matsunaga Toshiaki Fukushima Yoshihiro Tsuchiya Hideyuki Sakoda Midori Fujishiro Takeshi Yamamotoya Hideaki Kamata Akio Matsubara Tomoichiro Asano Natasha Kyprianou

BACKGROUND Prostate cancer initially develops in an androgen-dependent manner but, during its progression, transitions to being androgen-independent in the advanced stage. Pin1, one of the peptidyl-prolyl cis/trans isomerases, is reportedly overexpressed in prostate cancers and is considered to contribute to accelerated cell growth, which may be one of the major factors contributing to their an...

Journal: :Cancer research 2003
Takahito Hara Jun-ichi Miyazaki Hideo Araki Masuo Yamaoka Naoyuki Kanzaki Masami Kusaka Masaomi Miyamoto

Most prostate cancers (PCs) become resistant to combined androgen blockade therapy with surgical or medical castration and antiandrogens after several years. Some of these refractory PCs regress after discontinuation of antiandrogen administration [antiandrogen withdrawal syndrome (AWS)]. Although the molecular mechanisms of the AWS are not fully understood because of the lack of suitable exper...

2013
Ediz Sariisik Denitsa Docheva Daniela Padula Cvetan Popov Jan Opfer Matthias Schieker Hauke Clausen-Schaumann Martin Benoit

Adhesion of metastasizing prostate carcinoma cells was quantified for two carcinoma model cell lines LNCaP (lymph node-specific) and PC3 (bone marrow-specific). By time-lapse microscopy and force spectroscopy we found PC3 cells to preferentially adhere to bone marrow-derived mesenchymal stem cells (SCP1 cell line). Using atomic force microscopy (AFM) based force spectroscopy, the mechanical pat...

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