نتایج جستجو برای: link damage

تعداد نتایج: 412435  

2008
Nurit Mirkin Danae Fonseca Samia Mohammed Murat A. Cevher James L. Manley Frida E. Kleiman

Following DNA damage, mRNA levels decrease, reflecting a coordinated interaction of the DNA repair, transcription and RNA processing machineries. In this study, we provide evidence that transcription and polyadenylation of mRNA precursors are both affected in vivo by UV treatment. We next show that the polyadenylation factor CstF, plays a direct role in the DNA damage response. Cells with reduc...

Journal: :Cancer research 2007
Victoria Valinluck Lawrence C Sowers

Aberrant methylation patterns have long been known to exist in the promoter regions of key regulatory genes in the DNA of tumor cells. However, the mechanisms by which these methylation patterns become altered during the transformation of normal cells to tumor cells have remained elusive. We have recently shown in in vitro studies that inflammation-mediated halogenated cytosine damage products ...

Journal: :MATHEMATICA SCANDINAVICA 1979

2016
Yasuhiro Nakajima Masahito Hara Daisuke Umebayashi Shoichi Haimoto Yu Yamamoto Yusuke Nishimura Toshihiko Wakabayashi

STUDY DESIGN The strength effects of a pedicle screw-rod system supplemented with a novel cross-link configuration were biomechanically evaluated in porcine spines. PURPOSE To assess the biomechanical differences between a conventional cross-link pedicle screw-rod system versus a novel cross-link instrumentation, and to determine the effect of the cross-links. OVERVIEW OF LITERATURE Transve...

2011
Aidan G O'Keeffe Brian D M Tom Vernon T Farewell

In psoriatic arthritis, permanent joint damage characterizes disease progression and represents a major debilitating aspect of the disease. Understanding the process of joint damage will assist in the treatment and disease management of patients. Multistate models provide a means to examine patterns of disease, such as symmetric joint damage. Additionally, the link between damage and the dynami...

2017
Yannick Boege Mohsen Malehmir Marc E. Healy Kira Bettermann Anna Lorentzen Mihael Vucur Akshay K. Ahuja Friederike Böhm Joachim C. Mertens Yutaka Shimizu Lukas Frick Caroline Remouchamps Karun Mutreja Thilo Kähne Devakumar Sundaravinayagam Monika J. Wolf Hubert Rehrauer Christiane Koppe Tobias Speicher Susagna Padrissa-Altés Renaud Maire Jörn M. Schattenberg Ju-Seong Jeong Lei Liu Stefan Zwirner Regina Boger Norbert Hüser Roger J. Davis Beat Müllhaupt Holger Moch Henning Schulze-Bergkamen Pierre-Alain Clavien Sabine Werner Lubor Borsig Sanjiv A. Luther Philipp J. Jost Ricardo Weinlich Kristian Unger Axel Behrens Laura Hillert Christopher Dillon Michela Di Virgilio David Wallach Emmanuel Dejardin Lars Zender Michael Naumann Henning Walczak Douglas R. Green Massimo Lopes Inna Lavrik Tom Luedde Mathias Heikenwalder Achim Weber

Concomitant hepatocyte apoptosis and regeneration is a hallmark of chronic liver diseases (CLDs) predisposing to hepatocellular carcinoma (HCC). Here, we mechanistically link caspase-8-dependent apoptosis to HCC development via proliferation- and replication-associated DNA damage. Proliferation-associated replication stress, DNA damage, and genetic instability are detectable in CLDs before any ...

Journal: :Cell 2004
Ashby J. Morrison Jessica Highland Nevan J. Krogan Ayelet Arbel-Eden Jack F. Greenblatt James E. Haber Xuetong Shen

(Shen et al., 2000). However, it is not known whether the INO80 complex functions in DNA damage repair Waltham, Massachusetts 02454 directly or indirectly through other mechanisms. Here, we show biochemical and genetic evidence that the INO80 complex is directly involved in DNA repair. The Summary key mechanism is through a specific interaction between the INO80 complex and the DNA damage-While...

Journal: :Annual review of biochemistry 2004
Aziz Sancar Laura A Lindsey-Boltz Keziban Unsal-Kaçmaz Stuart Linn

DNA damage is a relatively common event in the life of a cell and may lead to mutation, cancer, and cellular or organismic death. Damage to DNA induces several cellular responses that enable the cell either to eliminate or cope with the damage or to activate a programmed cell death process, presumably to eliminate cells with potentially catastrophic mutations. These DNA damage response reaction...

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