نتایج جستجو برای: kcnq1

تعداد نتایج: 1121  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2007
Lei Chen Michelle L Marquardt David J Tester Kevin J Sampson Michael J Ackerman Robert S Kass

A-kinase anchoring proteins (AKAPs) recruit signaling molecules and present them to downstream targets to achieve efficient spatial and temporal control of their phosphorylation state. In the heart, sympathetic nervous system (SNS) regulation of cardiac action potential duration (APD), mediated by beta-adrenergic receptor (betaAR) activation, requires assembly of AKAP9 (Yotiao) with the I(Ks) p...

2015
Deng Ao Hai-jun Wang Li-fang Wang Jie-yun Song Hui-xia Yang Yan Wang Marta Letizia Hribal

OBJECTIVE Recent genetic studies have shown that potassium voltage-gated channel, KQT-like subfamily, member1 (KCNQ1) gene is related to gestational diabetes mellitus (GDM). However, studies for the rs2237892 polymorphism in KCNQ1 and GDM remain conflicting in Asians. Furthermore, associations of this polymorphism with glucose levels during oral glucose tolerance test (OGTT) have not been descr...

2016
Kaiping Gao Jinjin Wang Linlin Li Yujia Zhai Yongcheng Ren Haifei You Bingyuan Wang Xuli Wu Jianna Li Zichen Liu Xiong Li Yaxin Huang Xin-Ping Luo Dongsheng Hu Kinji Ohno Chongjian Wang

Genetic variants at KCNQ1 rs151290, KLF14 rs972283, GCKR rs780094 and MTNR1B rs10830963 have been associated with type 2 diabetes mellitus (T2DM), but the results are contradictory in Chinese populations. The aim of the present study was to investigate the association of these four SNPs with T2DM in a large population of Han Chinese at Henan province, China. Seven-hundred-thirty-six patients wi...

2007
A. Tinker

LQTS (long QT syndrome) is an important cause of cardiac sudden death. LQTS is characterized by a prolongation of the QT interval on an electrocardiogram. This prolongation predisposes the individual to torsade-de-pointes and subsequent sudden death by ventricular fibrillation. Mutations in a number of genes that encode ion channels have been implicated in LQTS. Hereditary mutations in the αand...

Journal: :Circulation. Cardiovascular genetics 2013
John R Giudicessi Michael J Ackerman

BACKGROUND- Homozygous or compound heterozygous mutations in KCNQ1 cause Jervell and Lange-Nielsen syndrome, a rare, autosomal-recessive form of long-QT syndrome characterized by deafness, marked QT prolongation, and a high risk of sudden death. However, it is not understood why some individuals with mutations on both KCNQ1 alleles present without deafness. In this study, we sought to determine...

2009
Anna Jonsson Bo Isomaa Tiinamaija Tuomi Jalal Taneera Albert Salehi Peter Nilsson Leif Groop Valeriya Lyssenko

OBJECTIVE Two independent genome-wide association studies for type 2 diabetes in Japanese subjects have recently identified common variants in the KCNQ1 gene that are strongly associated with type 2 diabetes. Here we studied whether a common variant in KCNQ1 would influence BMI as well as insulin secretion and action and predict future type 2 diabetes in subjects from Sweden and Finland. RESE...

Journal: :The Journal of General Physiology 2001
J. Kurokawa H.K. Motoike R.S. Kass

I(Ks), a slowly activating delayed rectifier K(+) current through channels formed by the assembly of two subunits KCNQ1 (KvLQT1) and KCNE1 (minK), contributes to the control of the cardiac action potential duration. Coassembly of the two subunits is essential in producing the characteristic and physiologically critical kinetics of assembled channels, but it is not yet clear where or how these s...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2010
Manjot Bal Jie Zhang Ciria C Hernandez Oleg Zaika Mark S Shapiro

M-type channels are localized to neuronal, cardiovascular, and epithelial tissues, where they play critical roles in control of excitability and K(+) transport, and are regulated by numerous receptors via G(q/11)-mediated signals. One pathway shown for KCNQ2 and muscarinic receptors uses PKC, recruited to the channels by A-kinase anchoring protein (AKAP)79/150. As M-type channels can be various...

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