نتایج جستجو برای: hdl

تعداد نتایج: 25774  

Journal: :Circulation 1978

Journal: :Arteriosclerosis, Thrombosis, and Vascular Biology 2008

Journal: :Clinical chemistry 1998
C K Chuang S P Lin Y T Lin F Y Huang

in a single run for HDL-C using these three homogeneous HDL-C reagents. Mean concentrations of HDL-C in group 1 were 508 mg/L for the direct HDL-C assay, 517 mg/L for the N-geneous TM HDL-C assay, and 531 mg/L for the EZ-HDL TM-C assay. However, mean concentrations of HDL-C in group 2 were 253 mg/L for the direct HDL-C assay, 352 mg/L for the N-geneous TM HDL-C assay, and 253 mg/L for the EZ-HD...

Journal: :Circulation 2010
Sajoscha A Sorrentino Christian Besler Lucia Rohrer Martin Meyer Kathrin Heinrich Ferdinand H Bahlmann Maja Mueller Tibor Horváth Carola Doerries Mariko Heinemann Stella Flemmer Andrea Markowski Costantina Manes Matthias J Bahr Hermann Haller Arnold von Eckardstein Helmut Drexler Ulf Landmesser

BACKGROUND High-density lipoprotein (HDL)-raising therapies are currently under intense evaluation, but the effects of HDL may be highly heterogeneous. We therefore compared the endothelial effects of HDL from healthy subjects and from patients with type 2 diabetes mellitus and low HDL (meeting the criteria for metabolic syndrome), who are frequently considered for HDL-raising therapies. Moreov...

2014
Bishnu H Subedi Parag H Joshi Steven R Jones Seth S Martin Michael J Blaha Erin D Michos

Many studies have suggested that a significant risk factor for atherosclerotic cardiovascular disease (ASCVD) is low high-density lipoprotein cholesterol (HDL-C). Therefore, increasing HDL-C with therapeutic agents has been considered an attractive strategy. In the prestatin era, fibrates and niacin monotherapy, which cause modest increases in HDL-C, reduced ASCVD events. Since their introducti...

Journal: :Journal of lipid research 2009
Frank M Sacks Lawrence L Rudel Adam Conner Hassibullah Akeefe Gerhard Kostner Talal Baki George Rothblat Margarita de la Llera-Moya Bela Asztalos Timothy Perlman Chunyu Zheng Petar Alaupovic Jo-Ann B Maltais H Bryan Brewer

Uptake of cholesterol from peripheral cells by nascent small HDL circulating in plasma is necessary to prevent atherosclerosis. This process, termed reverse cholesterol transport, produces larger cholesterol-rich HDL that transfers its cholesterol to the liver facilitating excretion. Most HDL in plasma is cholesterol-rich. We demonstrate that treating plasma with a novel selective delipidation ...

2015
Yan Zhang Sha Li Rui-Xia Xu Yuan-Lin Guo Na-Qiong Wu Cheng-Gang Zhu Ying Gao Qian Dong Geng Liu Jing Sun Jian-Jun Li

The exact mechanisms of hypertension contributing to atherosclerosis have not been fully elucidated. Although multiple studies have clarified the association with low-density lipoprotein (LDL) subfractions, uncertainty remains about its relationship with high-density lipoprotein (HDL) subfractions. Therefore, we aimed to comprehensively determine the relationship between distribution of HDL sub...

Journal: :The Journal of clinical investigation 1998
M Aviram M Rosenblat C L Bisgaier R S Newton S L Primo-Parmo B N La Du

HDL levels are inversely related to the risk of developing atherosclerosis. In serum, paraoxonase (PON) is associated with HDL, and was shown to inhibit LDL oxidation. Whether PON also protects HDL from oxidation is unknown, and was determined in the present study. In humans, we found serum HDL PON activity and HDL susceptibility to oxidation to be inversely correlated (r2 = 0.77, n = 15). Supp...

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