نتایج جستجو برای: glucosephosphate dehydrogenase deficiency

تعداد نتایج: 199743  

2014
Natsuda Jamornthanyawat Ghulam R. Awab Naowarat Tanomsing Sasithon Pukrittayakamee Fazel Yamin Arjen M. Dondorp Nicholas P. J. Day Nicholas J. White Charles J. Woodrow Mallika Imwong

Glucose-6-phosphate dehydrogenase (G6PD) deficiency is a common inherited enzyme defect and an important problem in areas with Plasmodium vivax infection because of the risk of haemolysis following administration of primaquine to treat the liver forms of the parasite. We undertook a genotypic survey of 713 male individuals across nine provinces of Afghanistan in which malaria is found, four in ...

Journal: :medical journal of islamic republic of iran 0
z karamizadeh from the department of pediatrics, shiraz university of medical sciences, shiraz, islamic republic of iran. gh amirhakimi

in this study the data on 115 cases of congenital adrenal hyperplasia (cah) who were followed in the pediatric endocrine clinic at nemazee hospital, shiraz will be reported. among these cases 51 were male and 64 female. the most common type of cah in these patients was the salt-losing type of 21 -hydroxylase deficiency (85 .2%). ll-hydroxylase deficiency was present in 13.04% of patients. there...

Journal: :Canadian Anaesthetists’ Society Journal 1983

Journal: :Journal of Nutritional Science and Vitaminology 1992

Journal: :Postgraduate Medical Journal 1994

2000
B. Stankoff A. Tourbah S. Suarez E. Turell J. L. Stievenart C. Payan A. Coutellier S. Herson L. Baril F. Bricaire V. Calvez E. A. Cabanis L. Lacomblez C. Lubetzki

inflammatory leukoencephalopathy and dihydropyrimidine dehydrogenase deficiency Article abstract—Multifocal inflammatory leukoencephalopathy (MIL) is a cerebral demyelinating syndrome that develops after chemotherapy with 5-fluorouracil (5-FU) and levamisole. The authors report a patient who developed MIL after 5-FU administration not in association with levamisole. She was subsequently diagnos...

Journal: :Annals of oncology : official journal of the European Society for Medical Oncology 2001
J Volk F Reinke A B van Kuilenburg A H van Gennip C Schlichting A Ganser P Schöffski

Dihydropyrimidine dehydrogenase deficiency is diagnosed more frequently and is now generally accepted as a potentially life-threatening condition. It predisposes patients receiving treatment with fluoropyrimidines such as 5-fluorouracil (5-FU) to severe and, in case of complete dihydropyrimidine dehydrogenase deficiency, often fatal toxicity. A patient who had severe side effects following stan...

2014
Shane C. Quinonez Andrea H. Seeley Mary Seeterlin Eleanor Stanley Ayesha Ahmad

Dihydrolipoamide dehydrogenase deficiency, also known as maple syrup urine disease (MSUD) type III, is caused by the deficiency of the E3 subunit of branched chain alpha-ketoacid dehydrogenase (BCKDH), α-ketoglutarate dehydrogenase (αKGDH), and pyruvate dehydrogenase (PDH). DLD deficiency variably presents with either a severe neonatal encephalopathic phenotype or a primarily hepatic phenotype....

Journal: :Archives of disease in childhood. Fetal and neonatal edition 1995
C Largillière C Vianey-Saban M Fontaine C Bertrand N Kacet J P Farriaux

Very long chain acyl-CoA dehydrogenase is a newly characterised enzyme in mitochondrial fatty acid oxidation. A girl who presented on the second day of life with a sudden and severe illness due to deficiency of this enzyme is reported. There is evidence that some children (and perhaps all) originally diagnosed with a deficiency of long-chain acyl-CoA dehydrogenase, in fact, have a defect involv...

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