نتایج جستجو برای: glucose transporter 2

تعداد نتایج: 2678274  

Journal: :The Biochemical journal 1996
F Maher T M Davies-Hill I A Simpson

This study examines the apparent affinity, catalytic-centre activity ("turnover number') and stereospecificity of the neuronal glucose transporter GLUT3 in primary cultured cerebellar granule neurons. Using a novel variation of the 3-O-[14C]methylglucose transport assay, by measuring zero-trans kinetics at 25 degrees C, GLUT3 was determined to be a high-apparent-affinity, high-activity, glucose...

Journal: :FASEB journal : official publication of the Federation of American Societies for Experimental Biology 1994
A Klip T Tsakiridis A Marette P A Ortiz

Glucose transporters are membrane-embedded proteins that mediate the uptake of glucose from the surrounding medium into the cell. Glucose is the main fuel for most cells, and its uptake is rate-limiting for glucose utilization. For this reason, it is expected that glucose transport is tightly regulated. Whereas rapid regulation of glucose transporters by hormones has been known for some time, t...

2016
Marie Mongin Nicolas Mezouar Pauline Dodet Marie Vidailhet Emmanuel Roze

BACKGROUND Glucose transporter type 1 deficiency syndrome is due to de novo mutations in the SLC2A1 gene encoding the glucose transporter type 1. PHENOMENOLOGY SHOWN Paroxysmal motor manifestations induced by exercise or fasting may be the main manifestations of glucose transporter type 1 deficiency syndrome. EDUCATIONAL VALUE Proper identification of the paroxysmal events and early diagnos...

Journal: :American journal of physiology. Endocrinology and metabolism 2013
David R Powell Christopher M DaCosta Jason Gay Zhi-Ming Ding Melinda Smith Jennifer Greer Deon Doree Sabrina Jeter-Jones Faika Mseeh Lawrence A Rodriguez Angela Harris Lindsey Buhring Kenneth A Platt Peter Vogel Robert Brommage Melanie K Shadoan Arthur T Sands Brian Zambrowicz

Sodium-glucose cotransporter 2 (SGLT2) is the major, and SGLT1 the minor, transporter responsible for renal glucose reabsorption. Increasing urinary glucose excretion (UGE) by selectively inhibiting SGLT2 improves glycemic control in diabetic patients. We generated Sglt1 and Sglt2 knockout (KO) mice, Sglt1/Sglt2 double-KO (DKO) mice, and wild-type (WT) littermates to study their relative glycem...

Journal: :The Biochemical journal 1997
N Ahmed M Kansara M V Berridge

Activation of the respiratory burst imposes acute metabolic demands on phagocytic cells. These are met by mobilizing internal energy stores and by increasing the utilization of exogenous energy, including glucose in the circulation. To determine whether the increased glucose uptake that is known to be associated with the respiratory burst involves the regulation of glucose transporter molecules...

2015
Xinyuan Xu Jianying Li Xiang Sun Yan Guo Dake Chu Li Wei Xia Li Guodong Yang Xinping Liu Libo Yao Jian Zhang Lan Shen

Cancer cells use glucose and glutamine as the major sources of energy and precursor intermediates, and enhanced glycolysis and glutamimolysis are the major hallmarks of metabolic reprogramming in cancer. Oncogene activation and tumor suppressor gene inactivation alter multiple intracellular signaling pathways that affect glycolysis and glutaminolysis. N-Myc downstream regulated gene 2 (NDRG2) i...

2006
Tomoichiro Asano Yoshikazu Shibasaki Jiann-Liang Lin Katsunori Tsukuda Hideki Katagiri I lisamilsu Ishihara Yoshio Yazaki Yoshitomo Oka

An increase in expression of the GLUTI glucose transporter gene has been observed to be associated with an increase in glucose transport activity upon oncogenic transformation of the cells. Increased expression of this glucose transporter isoform has been also observed in fetal tissues. To investigate the consequences of this phenomenon on cellular metabo lism and cell growth, an expression vec...

Journal: :Journal of cellular physiology 2005
Maria L Macheda Suzanne Rogers James D Best

Malignant cells are known to have accelerated metabolism, high glucose requirements, and increased glucose uptake. Transport of glucose across the plasma membrane of mammalian cells is the first rate-limiting step for glucose metabolism and is mediated by facilitative glucose transporter (GLUT) proteins. Increased glucose transport in malignant cells has been associated with increased and dereg...

Journal: :Brain : a journal of neurology 2010
Wilhelmina G Leen Joerg Klepper Marcel M Verbeek Maike Leferink Tom Hofste Baziel G van Engelen Ron A Wevers Todd Arthur Nadia Bahi-Buisson Diana Ballhausen Jolita Bekhof Patrick van Bogaert Inês Carrilho Brigitte Chabrol Michael P Champion James Coldwell Peter Clayton Elizabeth Donner Athanasios Evangeliou Friedrich Ebinger Kevin Farrell Rob J Forsyth Christian G E L de Goede Stephanie Gross Stephanie Grunewald Hans Holthausen Sandeep Jayawant Katherine Lachlan Vincent Laugel Kathy Leppig Ming J Lim Grazia Mancini Adela Della Marina Loreto Martorell Joe McMenamin Marije E C Meuwissen Helen Mundy Nils O Nilsson Axel Panzer Bwee T Poll-The Christian Rauscher Christophe M R Rouselle Inger Sandvig Thomas Scheffner Eamonn Sheridan Neil Simpson Parol Sykora Richard Tomlinson John Trounce David Webb Bernhard Weschke Hans Scheffer Michél A Willemsen

Glucose transporter-1 deficiency syndrome is caused by mutations in the SLC2A1 gene in the majority of patients and results in impaired glucose transport into the brain. From 2004-2008, 132 requests for mutational analysis of the SLC2A1 gene were studied by automated Sanger sequencing and multiplex ligation-dependent probe amplification. Mutations in the SLC2A1 gene were detected in 54 patients...

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