نتایج جستجو برای: factor viia

تعداد نتایج: 844421  

Journal: :Thrombosis and haemostasis 1998
J Y Chang D M Monroe J A Oliver D K Liles H R Roberts

Tissue factor pathway inhibitor (TFPI) acts to regulate the initiation of coagulation by first inhibiting factor Xa. The complex of factor Xa/TFPI then inhibits the factor VIIa/tissue factor complex. The cDNA sequences of TFPI from several different species have been previously reported. A high level of similarity is present among TFPIs at the molecular level (DNA and protein sequences) as well...

Journal: :Journal of Postgraduate Medicine 2007

Journal: :Japanese Journal of Thrombosis and Hemostasis 2014

Journal: :Journal of Thrombosis and Haemostasis 2003

Journal: :The Israel Medical Association journal : IMAJ 2009
Tzipora Strauss Gili Kenet Irit Schushan-Eisen Ram Mazkereth Jacob Kuint

Journal: :Blood 1996
J Jesty A Lorenz J Rodriguez T C Wun

Activation of factor X by both the unactivated tissue factor:factor VII complex (TF:VII) and the activated tissue factor:factor VIIa complex (TF:VIIa) has been studied in the presence of tissue factor pathway inhibitor (TFPI), antithrombin III (ATIII), and heparin. At near-plasma concentrations of TFPI, ATIII, and factor X, factor X activation that occurs in response to TF:VII is essentially ab...

2011
Emily K. Waters Ryan M. Genga Michael C. Schwartz Jennifer A. Nelson Robert G. Schaub Karen A. Olson Jeffrey C. Kurz Kathleen E. McGinness

Hemophilia A and B are caused by deficiencies in coagulation factor VIII (FVIII) and factor IX, respectively, resulting in deficient blood coagulation via the intrinsic pathway. The extrinsic coagulation pathway, mediated by factor VIIa and tissue factor (TF), remains intact but is negatively regulated by tissue factor pathway inhibitor (TFPI), which inhibits both factor VIIa and its product, f...

Journal: :Arteriosclerosis, thrombosis, and vascular biology 2004
Simone J H Wielders Suzette Béguin H Coenraad Hemker Theo Lindhout

OBJECTIVE Feedback activation of factor XI by thrombin is a likely alternative for tissue factor-dependent propagation of thrombus formation. However, the hypothesis that thrombin can initiate and propagate its formation in a factor XI-dependent and platelet-dependent manner has not been tested in a plasma milieu. METHODS AND RESULTS We investigated thrombin generation in recalcified platelet...

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