نتایج جستجو برای: amyloid beta25 35 folding

تعداد نتایج: 244389  

Journal: :PLoS ONE 2008
Yuji Yoshiike Ryoichi Minai Yo Matsuo Yun-Ru Chen Tetsuya Kimura Akihiko Takashima

Recent in vitro and in vivo studies suggest that destabilized proteins with defective folding induce aggregation and toxicity in protein-misfolding diseases. One such unstable protein state is called amyloid oligomer, a precursor of fully aggregated forms of amyloid. Detection of various amyloid oligomers with A11, an anti-amyloid oligomer conformation-specific antibody, revealed that the amylo...

2013
Xiaowen Feng Nan Liang Dexiao Zhu Qing Gao Lei Peng Haiman Dong Qingwei Yue Haili Liu Lihua Bao Jing Zhang Jing Hao Yingmao Gao Xuejie Yu Jinhao Sun

Alzheimer's disease (AD) is characterized by the accumulation of β-amyloid peptide (Aβ) and loss of neurons. Recently, a growing body of evidences have indicated that as a herbal compound naturally derived from grapes, resveratrol modulates the pathophysiology of AD, however, with a largely unclear mechanism. Therefore, we aimed to investigate the protection of resveratrol against the neurotoxi...

2017
Yu-Sung Cheng Zih-Ten Chen Tai-Yan Liao Chen Lin Howard C-H Shen Ya-Han Wang Chi-Wei Chang Ren-Shyan Liu Rita P-Y Chen Pang-Hsien Tu

Alzheimer's disease (AD) is the most common neurodegenerative disease. Imbalance between the production and clearance of amyloid β (Aβ) peptides is considered to be the primary mechanism of AD pathogenesis. This amyloid hypothesis is supported by the recent success of the human anti-amyloid antibody aducanumab, in clearing plaque and slowing clinical impairment in prodromal or mild patients in ...

Journal: :Alzheimers & Dementia 2021

Abstract Background Here, we investigated the contribution of education and regional amyloid as potential de‐ accelerators in spread tau pathology. Method 85 amyloid‐positive subjects (age=76.80±1.08, education=16.70 ±.50 years, M/F=42/43) were included, for whom [18F]AV‐1451 PET at two timepoints, baseline [18F]AV‐45 PET, ApoE‐status clinical information available ADNI. PETs intensity‐standard...

Journal: :Proceedings of the National Academy of Sciences 2003

Journal: :BioEssays : news and reviews in molecular, cellular and developmental biology 2012
Shu Quan James C A Bardwell

Molecular chaperones assist de novo protein folding and facilitate the refolding of stress-denatured proteins. The molecular chaperone concept was coined nearly 35 years ago, and since then, tremendous strides have been made in understanding how these factors support protein folding. Here, we focus on how various chaperone proteins were first identified to play roles in protein folding. Example...

2015
Melissa E. Murray Val J. Lowe Neill R. Graff-Radford Amanda M. Liesinger Ashley Cannon Scott A. Przybelski Bhupendra Rawal Joseph E. Parisi Ronald C. Petersen Kejal Kantarci Owen A. Ross Ranjan Duara David S. Knopman Clifford R. Jack Dennis W. Dickson

Thal amyloid phase, which describes the pattern of progressive amyloid-β plaque deposition in Alzheimer's disease, was incorporated into the latest National Institute of Ageing - Alzheimer's Association neuropathologic assessment guidelines. Amyloid biomarkers (positron emission tomography and cerebrospinal fluid) were included in clinical diagnostic guidelines for Alzheimer's disease dementia ...

2013
Nicola J. Corbett Paul L. Gabbott Boris Klementiev Heather A. Davies Frances M. Colyer Tatiana Novikova Michael G. Stewart

Increased levels of neurotoxic amyloid-beta in the brain are a prominent feature of Alzheimer's disease. FG-Loop (FGL), a neural cell adhesion molecule-derived peptide that corresponds to its second fibronectin type III module, has been shown to provide neuroprotection against a range of cellular insults. In the present study impairments in social recognition memory were seen 24 days after a 5 ...

2013
Sanjay M Banypersad Steve Bandula Daniel Sado Jennifer H Pinney Simon D Gibbs Viviana Maestrini Marianna Fontana Steven K White Shonit Punwani Stuart Taylor Philip N Hawkins James Moon

Methods Healthy volunteers (n=70; 35 male; 35 female; median age 46 years) and patients with systemic AL Amyloidosis (n=56; 36 male; 20 female; median age 62 years), with clinical indications for CMR scanning, additionally underwent multi-organ ECV measurement. Technical details were: gadoteric acid, 0.1mmol/Kg plus infusion, multibreathhold T1 measurement and equilibrium imaging of heart, live...

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