نتایج جستجو برای: زمان تا باردار شدن ttp

تعداد نتایج: 311144  

Journal: :Stroke 2004
J Sobesky O Zaro Weber F-G Lehnhardt V Hesselmann A Thiel C Dohmen A Jacobs M Neveling W-D Heiss

BACKGROUND AND PURPOSE In acute ischemic stroke, the hypoperfused but viable tissue is the main therapeutic target. In clinical routine, time-to-peak (TTP) maps are frequently used to estimate the hemodynamic compromise and to calculate the mismatch volume. We evaluated the accuracy of TTP maps to identify penumbral flow by comparison with positron emission tomography (PET). METHODS Magnetic ...

Journal: : 2022

نقش صلاحیت فردی و رضایت شغلی ممیزان مالیاتی در ارتقاء سطح قدرت "مالیات‌ستانی" توسط سازمان امور کشور: بررسی محدودیت (فشار) زمان

Journal: :Molecular and cellular biology 2006
Edward Hitti Tatiana Iakovleva Matthew Brook Stefanie Deppenmeier Achim D Gruber Danuta Radzioch Andrew R Clark Perry J Blackshear Alexey Kotlyarov Matthias Gaestel

The mitogen-activated protein kinase (MAPK) p38/MAPK-activated protein kinase 2 (MK2) signaling pathway plays an important role in the posttranscriptional regulation of tumor necrosis factor (TNF), which is dependent on the adenine/uridine-rich element (ARE) in the 3' untranslated region of TNF mRNA. After lipopolysaccharide (LPS) stimulation, MK2-deficient macrophages show a 90% reduction in T...

2017
E A Ross A J Naylor J D O'Neil T Crowley M L Ridley J Crowe T Smallie T J Tang J D Turner L V Norling S Dominguez H Perlman N M Verrills G Kollias M P Vitek A Filer C D Buckley J L Dean A R Clark

OBJECTIVES Tristetraprolin (TTP), a negative regulator of many pro-inflammatory genes, is strongly expressed in rheumatoid synovial cells. The mitogen-activated protein kinase (MAPK) p38 pathway mediates the inactivation of TTP via phosphorylation of two serine residues. We wished to test the hypothesis that these phosphorylations contribute to the development of inflammatory arthritis, and tha...

2014
Boris Reznik Sandra L. Clement Jens Lykke-Andersen

The tristetraprolin (TTP) family of zinc-finger proteins, TTP, BRF1 and BRF2, regulate the stability of a subset of mRNAs containing 3'UTR AU-rich elements (AREs), including mRNAs coding for cytokines, transcription factors, and proto-oncogenes. To better understand the mechanism by which TTP-family proteins control mRNA stability in mammalian cells, we aimed to identify TTP- and BRF1-interacti...

2014
Yang Jiang Jennifer J. McIntosh Jessica A. Reese Cassandra C. Deford Johanna A. Kremer Hovinga Bernhard Lämmle Deirdra R. Terrell Sara K. Vesely Eric J. Knudtson James N. George

Pregnancy is a recognized risk for precipitating acute episodes of thrombotic thrombocytopenic purpura (TTP), creating concern for the risk of recurrent TTP associated with pregnancies following recovery. Among women with hereditary ADAMTS13 deficiency, the occurrence of an acute episode of TTP during pregnancy with fetal loss is commonly reported and may be inevitable without plasma prophylaxi...

Journal: :Journal of immunology 2004
Seth A Brooks John E Connolly William F C Rigby

Tristetraprolin (TTP) is a regulator of TNF-alpha mRNA stability and is the only trans-acting factor shown to be capable of regulating AU-rich element-dependent mRNA turnover at the level of the intact animal. Using the THP-1 myelomonocytic cell line, we demonstrated for the first time that TTP is encoded by an mRNA with a short half-life under resting conditions. Using pharmacologic inhibitors...

Journal: :The Journal of biological chemistry 2002
Barbra A Johnson Justine R Stehn Michael B Yaffe T Keith Blackwell

The immediate early gene tristetraprolin (TTP) is induced transiently in many cell types by numerous extracellular stimuli. TTP encodes a zinc finger protein that can bind and destabilize mRNAs that encode tumor necrosis factor-alpha (TNFalpha) and other cytokines. We hypothesize that TTP also has a broader role in growth factor-responsive pathways. In support of this model, we have previously ...

Journal: :Mediators of Inflammation 2006
Ulla Jalonen Riina Nieminen Katriina Vuolteenaho Hannu Kankaanranta Eeva Moilanen

In inflammation, the post-transcriptional regulation of transiently expressed genes provides a potential therapeutic target. Tristetraprolin (TTP) is of the factors regulating decay of cytokine mRNAs. The aim of the present study was to identify cytokines whose expression is regulated by TTP. We established a TTP knock-down cell line by expressing shRNA against TTP (shTTP cell line). A cytokine...

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