نتایج جستجو برای: جهش های gjb2

تعداد نتایج: 479512  

2012
Jennifer A Easton Steven Donnelly Miriam A F Kamps Peter M Steijlen Patricia E Martin Gianluca Tadini René Janssens Rudolf Happle Michel van Geel Maurice A M van Steensel

Porokeratotic eccrine ostial and dermal duct nevus, or porokeratotic eccrine nevus (PEN), is a hyperkeratotic epidermal nevus. Several cases of widespread involvement have been reported, including one in association with the keratitis-ichthyosis-deafness (KID) syndrome (OMIM #148210), a rare disorder caused by mutations in the GJB2 gene coding for the gap junction protein connexin26 (Cx26). The...

2011
Sandra Iossa Elio Marciano Annamaria Franzé

The GJB2 gene is located on chromosome 13q12 and it encodes the connexin 26, a transmembrane protein involved in cell-cell attachment of almost all tissues. GJB2 mutations cause autosomal recessive (DFNB1) and sometimes dominant (DFNA3) non-syndromic sensorineural hearing loss. Moreover, it has been demonstrated that connexins are involved in regulation of growth and differentiation of epidermi...

Journal: :Human molecular genetics 2003
Takayuki Kudo Shigeo Kure Katsuhisa Ikeda An-Ping Xia Yukio Katori Masaaki Suzuki Kanako Kojima Akiko Ichinohe Yoichi Suzuki Yoko Aoki Toshimitsu Kobayashi Yoichi Matsubara

Hereditary deafness affects about 1 in 2000 children and mutations in the GJB2 gene are the major cause in various ethnic groups. GJB2 encodes connexin26, a putative channel component in cochlear gap junction. However, the pathogenesis of hearing loss caused by the GJB2 mutations remains obscure. The generation of a mouse model to study the function of connexin26 during hearing has been hampere...

2012
Bàrbara Castellana Daniel Escuin Gloria Peiró Bárbara Garcia-Valdecasas Tania Vázquez Cristina Pons Maitane Pérez-Olabarria Agustí Barnadas Enrique Lerma

UNLABELLED The mechanism of progression from ductal carcinoma in situ (DCIS) to invasive ductal carcinoma (IDC) remains largely unknown. We compared gene expression in tumors with simultaneous DCIS and IDC to decipher how diverse proteins participate in the local invasive process.Twenty frozen tumor specimens with concurrent, but separated, DCIS and IDC were microdissected and evaluated. Total ...

ژورنال: :مجله دانشگاه علوم پزشکی اراک 0
مهتاب خسروفر mahtab khosrofar department of cellular & molecular biology, school of basic sciences, east tehran branch (ghiamdasht), islamic azad university, tehran, iranگروه زیست شناسی سلولی و ملکولی، دانشکده علوم پایه، واحد تهران شرق(قیامدشت)، دانشگاه آزاد اسلامی، تهران، ایران محمد رضا پوررضا mohammad reza pourreza department of genetics and molecular biology, school of medicine, isfahan university of medical sciences, isfahan, iranگروه ژنتیک و زیست شناسی مولکولی، دانشکده پزشکی، دانشگاه علوم پزشکی اصفهان، اصفهان، ایرانسازمان اصلی تایید شده: دانشگاه آزاد اسلامی علوم و تحقیقات (islamic azad university science and research branch) سمیرا اصغرزاده samira asgharzadeh school of medicine, shahrekord university of medical sciences, shahrekord, iranدانشکده پزشکی، دانشگاه علوم پزشکی شهرکرد، شهرکرد، ایرانسازمان اصلی تایید شده: دانشگاه علوم پزشکی اصفهان (isfahan university of medical sciences) پریسا طهماسبی parisa tahmasebi department of biology, school of sciences, ilam university, ilam, iranگروه زیست شناسی، دانشکده علوم، دانشگاه ایلام، ایلام، ایرانسازمان اصلی تایید شده: دانشگاه علوم پزشکی شهرکرد (shahr kord university of medical sciences) الهه علی عسگری elahe ali asgari department of cellular & molecular biology, school of basic sciences, east tehran branch (ghiamdasht), islamic azad university, tehran, iranگروه زیست شناسی سلولی و ملکولی، دانشکده علوم پایه، واحد تهران شرق(قیامدشت)،دانشگاه آزاد اسلامی، تهران، ایرانسازمان اصلی تایید شده: دانشگاه ایلام (ilam university) رضا قاسمی خواه reza ghasemikhah department of parasitology and mycology, school of medicine, arak university of medical sciences, arak, iranگروه انگل شناسی و قارچ شناسی، دانشکده پزشکی، دانشگاه علوم پزشکی اراک، اراک، ایرانسازمان اصلی تایید شده: دانشگاه آزاد اسلامی علوم و تحقیقات (islamic azad university science and research branch) نادر صاکی

چکیده زمینه و هدف: ناشنوایی یکی از متداول ترین ناهنجاری های مادر زادی است. یک تا دو مورد از هر 1000 تولد دارای ناشنوایی پیش از تکلم می باشند. ناشنوایی غیر نشانگانی مغلوب اتوزومی متداول ترین نوع ناشنوایی ارثی است .رواج ناشنوایی در کشورهای در حال توسعه بیش تر می باشد که عواملی چون ژنتیک و محیط(عوامل فرهنگی- بهداشتی) در بروز آن نقش دارند. ناشنوایی طیف گسترده ای از تظاهرات بالینی مادرزادی یا دیررس،...

Journal: :Archives of otolaryngology--head & neck surgery 2004
Lawrence R Lustig Doris Lin Holly Venick Jan Larky Jennifer Yeagle Jill Chinnici Colleen Polite Anand N Mhatre John K Niparko Anil K Lalwani

OBJECTIVE To determine the prevalence of GJB2 gene mutations in patients undergoing cochlear implantation (CI) and their impact on rehabilitative outcome following implantation. DESIGN Prospective determination of GJB2 mutation by sequence analysis by denaturing high-performance liquid chromatography and its correlation with outcome following CI. SETTINGS Two tertiary academic medical cente...

Journal: :Journal of medical genetics 2004
K Cryns E Orzan A Murgia P L M Huygen F Moreno I del Castillo G Parker Chamberlin H Azaiez S Prasad R A Cucci E Leonardi R L Snoeckx P J Govaerts P H Van de Heyning C M Van de Heyning R J H Smith G Van Camp

INTRODUCTION Mutations in GJB2 are the most common cause of non-syndromic autosomal recessive hearing impairment, ranging from mild to profound. Mutation analysis of this gene is widely available as a genetic diagnostic test. OBJECTIVE To assess a possible genotype-phenotype correlation for GJB2. DESIGN Retrospective analysis of audiometric data from people with hearing impairment, segregat...

2015
So Young Kim Ah Reum Kim Kyu Hee Han Min Young Kim Eun-Hee Jeon Ja-Won Koo Seung Ha Oh Byung Yoon Choi Berta Alsina

INTRODUCTION The contribution of Gap junction beta-2 protein (GJB2) to the genetic load of deafness and its mutation spectra vary among different ethnic groups. OBJECTIVE In this study, the mutation spectrum and audiologic features of patients with GJB2 mutations were evaluated with a specific focus on residual hearing. METHODS An initial cohort of 588 subjects from 304 families with varyin...

2016
Ichiro Fukunaga Ayumi Fujimoto Kaori Hatakeyama Toru Aoki Atena Nishikawa Tetsuo Noda Osamu Minowa Nagomi Kurebayashi Katsuhisa Ikeda Kazusaku Kamiya

Mutation of the Gap Junction Beta 2 gene (GJB2) encoding connexin 26 (CX26) is the most frequent cause of hereditary deafness worldwide and accounts for up to 50% of non-syndromic sensorineural hearing loss cases in some populations. Therefore, cochlear CX26-gap junction plaque (GJP)-forming cells such as cochlear supporting cells are thought to be the most important therapeutic target for the ...

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