نتایج جستجو برای: polycystic kidneys

تعداد نتایج: 39226  

Journal: :Developmental biology 2000
L Ostrom M J Tang P Gruss G R Dressler

The murine cpk mouse develops a rapid-onset polycystic kidney disease (PKD) with many similarities to human PKD. During kidney development, the transcription factor Pax2 is required for the specification and differentiation of the renal epithelium. In humans, Pax2 is also expressed in juvenile cystic kidneys where it correlates with cell proliferation. In this report, Pax2 expression is demonst...

Journal: :Human molecular genetics 2011
Ayumi Takakura Erik A Nelson Nadeem Haque Benjamin D Humphreys Kambiz Zandi-Nejad David A Frank Jing Zhou

Autosomal dominant polycystic kidney disease (ADPKD) is a commonly inherited disorder mostly caused by mutations in PKD1, encoding polycystin-1 (PC1). The disease is characterized by development and growth of epithelium-lined cyst in both kidneys, often leading to renal failure. There is no specific treatment for this disease. Here, we report a sustained activation of the transcription factor s...

2013
Katherine I. Swenson-Fields Carolyn J. Vivian Sally M. Salah Jacqueline D. Peda Bradley M. Davis Nico van Rooijen Darren P. Wallace Timothy A. Fields

Renal M2-like macrophages have critical roles in tissue repair, stimulating tubule cell proliferation and, if they remain, fibrosis. M2-like macrophages have also been implicated in promoting cyst expansion in mouse models of autosomal dominant polycystic kidney disease (ADPKD). While renal macrophages have been documented in human ADPKD, there are no studies in autosomal recessive polycystic k...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2011
Jeffrey J Talbot Jonathan M Shillingford Shivakumar Vasanth Nicholas Doerr Sambuddho Mukherjee Mike T Kinter Terry Watnick Thomas Weimbs

Mutations in polycystin-1 (PC1) lead to autosomal-dominant polycystic kidney disease (ADPKD), a leading cause of renal failure for which no treatment is available. PC1 is an integral membrane protein, which has been implicated in the regulation of multiple signaling pathways including the JAK/STAT pathway. Here we show that membrane-anchored PC1 activates STAT3 in a JAK2-dependent manner, leadi...

Journal: :Circulation 2014
Wissam A Aboualaiwi Brian S Muntean Shobha Ratnam Bina Joe Lijun Liu Robert L Booth Ingrid Rodriguez Britney S Herbert Robert L Bacallao Marcus Fruttiger Tak W Mak Jing Zhou Surya M Nauli

BACKGROUND Cystic kidneys and vascular aneurysms are clinical manifestations seen in patients with polycystic kidney disease, a cilia-associated pathology (ciliopathy). Survivin overexpression is associated with cancer, but the clinical pathology associated with survivin downregulation or knockout has never been studied before. The present studies aim to examine whether and how cilia function (...

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