نتایج جستجو برای: pick type c1 npc1

تعداد نتایج: 1363808  

Journal: :The Journal of clinical investigation 2014
Danielle te Vruchte Anneliese O Speak Kerri L Wallom Nada Al Eisa David A Smith Christian J Hendriksz Louise Simmons Robin H Lachmann Alison Cousins Ralf Hartung Eugen Mengel Heiko Runz Michael Beck Yasmina Amraoui Jackie Imrie Elizabeth Jacklin Kate Riddick Nicole M Yanjanin Christopher A Wassif Arndt Rolfs Florian Rimmele Naomi Wright Clare Taylor Uma Ramaswami Timothy M Cox Caroline Hastings Xuntian Jiang Rohini Sidhu Daniel S Ory Begona Arias Mylvaganam Jeyakumar Daniel J Sillence James E Wraith Forbes D Porter Mario Cortina-Borja Frances M Platt

Lysosomal storage disorders (LSDs) occur at a frequency of 1 in every 5,000 live births and are a common cause of pediatric neurodegenerative disease. The relatively small number of patients with LSDs and lack of validated biomarkers are substantial challenges for clinical trial design. Here, we evaluated the use of a commercially available fluorescent probe, Lysotracker, that can be used to me...

2016
Victoria Schlegel Markus Thieme Carsten Holzmann Martin Witt Ulrike Grittner Arndt Rolfs Andreas Wree

Niemann-Pick Type C1 (NPC1) is an autosomal recessive inherited disorder characterized by accumulation of cholesterol and glycosphingolipids. Previously, we demonstrated that BALB/c-npc1nihNpc1-/- mice treated with miglustat, cyclodextrin and allopregnanolone generally performed better than untreated Npc1-/- animals. Unexpectedly, they also seemed to accomplish motor tests better than their sha...

2014
Yuta Tanaka Yoichi Ishitsuka Yusei Yamada Yuki Kondo Toru Takeo Naomi Nakagata Taishi Higashi Keiichi Motoyama Hidetoshi Arima Muneaki Matsuo Katsumi Higaki Kousaku Ohno Tetsumi Irie

Hydroxypropyl-β-cyclodextrin (HPBCD) is an attractive drug candidate against Niemann-Pick Type C (NPC) disease. However, the safety of HPBCD treatment for NPC patients remains to be elucidated. In this study, we examined the acute toxicity of HPBCD in Npc1-deficient mice. When treated with HPBCD (20,000 mg/kg, subcutaneously), over half of the wild-type (Npc1+/+) or Npc1+/- mice died by 72 h af...

Journal: :Journal of cell science 2006
Yuki Ohsaki Yuko Sugimoto Michitaka Suzuki Hiroshi Hosokawa Tamotsu Yoshimori Joanna P Davies Yiannis A Ioannou Marie T Vanier Kousaku Ohno Haruaki Ninomiya

Niemann-Pick disease type C (NPC) is an inherited lipid storage disorder caused by mutations in NPC1 or NPC2. NPC1 is a polytopic glycoprotein that contains a sterol-sensing domain, whereas NPC2 is a soluble protein that contains an MD-2-like lipid-recognition domain. In the current study, we addressed the hypothesis that ubiquitylation of NPC1 might be regulated by cholesterol. We found that d...

Journal: :Human molecular genetics 2013
Sophie Louwette Luc Régal Christine Wittevrongel Chantal Thys Gwenny Vandeweeghde Elisa Decuyper Peter Leemans Rita De Vos Chris Van Geet Jaak Jaeken Kathleen Freson

Niemann-Pick type C is a lysosomal storage disease associated with mutations in NPC1 or NPC2, resulting in an accumulation of cholesterol in the endosomal-lysosomal system. Niemann-Pick type C has a clinical spectrum that ranges from a neonatal rapidly fatal disorder to an adult-onset chronic neurodegenerative disease combined with remarkably, in some cases, hematological defects such as thromb...

Journal: :The Journal of biological chemistry 2003
Barbara Karten Dennis E Vance Robert B Campenot Jean E Vance

Niemann Pick type C (NPC) disease is a progressive neurodegenerative disorder. In cells lacking functional NPC1 protein, endocytosed cholesterol accumulates in late endosomes/lysosomes. We utilized primary neuronal cultures in which cell bodies and distal axons reside in separate compartments to investigate the requirement of NPC1 protein for transport of cholesterol from cell bodies to distal ...

2014
Dorothea Maetzel Sovan Sarkar Haoyi Wang Lina Abi-Mosleh Ping Xu Albert W. Cheng Qing Gao Maisam Mitalipova Rudolf Jaenisch

Niemann-Pick type C (NPC) disease is a fatal inherited lipid storage disorder causing severe neurodegeneration and liver dysfunction with only limited treatment options for patients. Loss of NPC1 function causes defects in cholesterol metabolism and has recently been implicated in deregulation of autophagy. Here, we report the generation of isogenic pairs of NPC patient-specific induced pluripo...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2011
Amal Aqul Benny Liu Charina M Ramirez Andrew A Pieper Sandi Jo Estill Dennis K Burns Bing Liu Joyce J Repa Stephen D Turley John M Dietschy

While unesterified cholesterol (C) is essential for remodeling neuronal plasma membranes, its role in certain neurodegenerative disorders remains poorly defined. Uptake of sterol from pericellular fluid requires processing that involves two lysosomal proteins, lysosomal acid lipase, which hydrolyzes C esters, and NPC1 (Niemann-Pick type C1). In systemic tissues, inactivation of either protein l...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید