نتایج جستجو برای: ox40

تعداد نتایج: 901  

2013
Minji Byun Cindy S. Ma Arzu Akçay Vincent Pedergnana Umaimainthan Palendira Jinjong Myoung Danielle T. Avery Yifang Liu Avinash Abhyankar Lazaro Lorenzo Monika Schmidt Hye Kyung Lim Olivier Cassar Melanie Migaud Flore Rozenberg Nur Canpolat Gönül Aydoğan Bernhard Fleckenstein Jacinta Bustamante Capucine Picard Antoine Gessain Emmanuelle Jouanguy Ethel Cesarman Martin Olivier Philippe Gros Laurent Abel Michael Croft Stuart G. Tangye Jean-Laurent Casanova

Kaposi sarcoma (KS), a human herpes virus 8 (HHV-8; also called KSHV)-induced endothelial tumor, develops only in a small fraction of individuals infected with HHV-8. We hypothesized that inborn errors of immunity to HHV-8 might underlie the exceedingly rare development of classic KS in childhood. We report here autosomal recessive OX40 deficiency in an otherwise healthy adult with childhood-on...

Journal: :Journal of immunology 2012
Dalya R Soond Fabien Garçon Daniel T Patton Julia Rolf Martin Turner Cheryl Scudamore Oliver A Garden Klaus Okkenhaug

PTEN, one of the most commonly mutated or lost tumor suppressors in human cancers, antagonizes signaling by the PI3K pathway. Mice with thymocyte-specific deletion of Pten rapidly develop peripheral lymphomas and autoimmunity, which may be caused by failed negative selection of thymocytes or from dysregulation of postthymic T cells. We induced conditional deletion of Pten from CD4 Th cells usin...

Journal: :Journal of immunology 2007
Ian R Humphreys Andrea Loewendorf Carl de Trez Kirsten Schneider Chris A Benedict Michael W Munks Carl F Ware Michael Croft

The mechanisms that regulate CMV-specific T cell responses in vivo are poorly understood. During murine CMV infection of B6 mice, primary responses in the spleen are dominated by CD8 T cells reactive with antigenic epitopes in M45, M57, and m139 murine CMV gene products. However, during the later persistent phase of infection, CD8 T cell responses to epitopes in m139 and M38 viral gene products...

2014
Julie K Laustsen Tue K Rasmussen Kristian Stengaard-Pedersen Kim Hørslev-Petersen Merete L Hetland Mikkel Østergaard Peter Junker Malene Hvid Bent Deleuran

INTRODUCTION OX40 and its ligand OX40L are key components in the generation of adaptive memory response and provide necessary co-stimulatory signals for activated effector T cells. Here we investigate the dual roles of the membrane and soluble (s) forms of OX40 and OX40L in plasma and synovial fluid and their association with autoantibodies and disease activity in rheumatoid arthritis (RA). M...

ژورنال: :دانشور پزشکی 0
فاطمه رحیمی جمنانی fatemeh rahimi jamnani department of medical biotechnology, biotechnology research center, pasteur institute of iranگروه بیوتکنولوژی پزشکی، مرکز تحقیقات بیوتکنولوژی، انستیتو پاستور ایران فاطمه رهبری زاده fatemeh rahbarizadeh department of medical biotechnology, faculty of medical sciences, tarbiat modares universityگروه بیوتکنولوژی پزشکی، دانشکده علوم پزشکی، دانشگاه تربیت مدرس فریدون مهبودی fereidoun mahboudi department of medical biotechnology, biotechnology research center, pasteur institute of iranگروه بیوتکنولوژی پزشکی، مرکز تحقیقات بیوتکنولوژی، انستیتو پاستور ایران محمدعلی شکرگزار mohammad ali shokrgozar national cell bank of iran, pasteur institute of iranبانک سلولی ایران، انستیتو پاستور ایران

مقدمه و هدف: به­کارگیری سیستم ایمنی فرد بیمار و سلول های t (به­عنوان بازوی قدرتمند سیستم ایمنی) در ازبین­بردن سلول های سرطانی، ازجمله روش های درمانی امیدبخش محسوب­می شود؛ در این راستا، سلول هایt  مهندسی­شده بیان­کننده رسپتورهای کایمریک دارای نانوبادی (آنتی­بادی تک دومینی شتری) که قادر به شناسایی آنتی ژن سرطانی her2 هستند، راهکاری مؤثر در درمان هدفمند سرطان سینه است.   مواد و روش ها: از نانوبادی...

2014
Silvia Piconese Eleonora Timperi Vincenzo Barnaba

Human regulatory T cells (Tregs) comprise an array of distinct subsets displaying diverse functions in response to microenvironmental signals. Here, we review our recent findings demonstrating the preferential accumulation of uncommitted, Th1-like and OX40- Tregs in non-cirrhotic tissues in contrast to the presence of committed, Th1-suppressing and OX40+ Tregs in cirrhotic and tumor contexts in...

2014
Ryan Montler Richard B Bell Rom Leidner Marka Crittenden Tuan Bui Allen Cheng Ashish Patel Christopher Tucker Colin Thalhofer Andrew Weinberg

OX40 is a potent co-stimulatory receptor on the surface of T lymphocytes. An OX40 agonist was recently tested in a Phase I clinical trial and was found to be well tolerated and enhanced both humoral and cellular immunity in patients with metastatic cancer. Both PD-1 and CTLA-4 blockade are showing therapeutic clinical activity in late stage cancer patients. Hence, we hypothesize that the immune...

Journal: :The Journal of clinical investigation 2007
Yui-Hsi Wang Yong-Jun Liu

Recent advances in understanding the cellular and molecular mechanisms of atopy have shed light on potential targets for the development of new therapies for allergic diseases. In this issue of the JCI, Seshasayee et al. provide direct in vivo evidence that OX40 has critical roles in allergic inflammation mediated by thymic stromal lymphopoietin (TSLP) (see the related article beginning on page...

Journal: :The Journal of clinical investigation 2004
Reiko Horai Akiko Nakajima Katsuyoshi Habiro Motoko Kotani Susumu Nakae Taizo Matsuki Aya Nambu Shinobu Saijo Hayato Kotaki Katsuko Sudo Akihiko Okahara Hidetoshi Tanioka Toshimi Ikuse Naoto Ishii Pamela L Schwartzberg Ryo Abe Yoichiro Iwakura

IL-1 receptor antagonist-deficient (IL-1Ra(-/-)) mice spontaneously develop autoimmune arthritis. We demonstrate here that T cells are required for the induction of arthritis; T cell-deficient IL-1Ra(-/-) mice did not develop arthritis, and transfer of IL-1Ra(-/-) T cells induced arthritis in nu/nu mice. Development of arthritis was also markedly suppressed by TNF-alpha deficiency. We found tha...

2016
Robert Janowski Gitta A. Heinz Andreas Schlundt Nina Wommelsdorf Sven Brenner Andreas R. Gruber Michael Blank Thorsten Buch Raymund Buhmann Mihaela Zavolan Dierk Niessing Vigo Heissmeyer Michael Sattler

The RNA-binding protein Roquin is required to prevent autoimmunity. Roquin controls T-helper cell activation and differentiation by limiting the induced expression of costimulatory receptors such as tumor necrosis factor receptor superfamily 4 (Tnfrs4 or Ox40). A constitutive decay element (CDE) with a characteristic triloop hairpin was previously shown to be recognized by Roquin. Here we use S...

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