نتایج جستجو برای: nhej

تعداد نتایج: 1322  

2008
Li Liang Li Deng Son C. Nguyen Xin Zhao Christopher D. Maulion Changshun Shao Jay A. Tischfield

DNA nonhomologous end-joining (NHEJ) and homologous recombination are two distinct pathways of DNA double-strand break repair in mammalian cells. Biochemical and genetic studies showed that DNA ends can also be joined via microhomology-mediated end joining (MHEJ), especially when proteins responsible for NHEJ, such as Ku, are reduced or absent. While it has been known that Ku-dependent NHEJ req...

2016
Anirban Chakraborty Nisha Tapryal Tatiana Venkova Nobuo Horikoshi Raj K. Pandita Altaf H. Sarker Partha S. Sarkar Tej K. Pandita Tapas K. Hazra

DNA double-strand breaks (DSBs) leading to loss of nucleotides in the transcribed region can be lethal. Classical non-homologous end-joining (C-NHEJ) is the dominant pathway for DSB repair (DSBR) in adult mammalian cells. Here we report that during such DSBR, mammalian C-NHEJ proteins form a multiprotein complex with RNA polymerase II and preferentially associate with the transcribed genes afte...

2016
Howard H. Y. Chang Go Watanabe Christina A. Gerodimos Takashi Ochi Tom L. Blundell Stephen P. Jackson Michael R. Lieber

The nonhomologous DNA end-joining (NHEJ) pathway is a key mechanism for repairing dsDNA breaks that occur often in eukaryotic cells. In the simplest model, these breaks are first recognized by Ku, which then interacts with other NHEJ proteins to improve their affinity at DNA ends. These include DNA-PKcs and Artemis for trimming the DNA ends; DNA polymerase μ and λ to add nucleotides; and the DN...

2018
Alisa Dewan Mengtan Xing Marie Benner Lundbæk Raquel Gago‐Fuentes Carole Beck Per Arne Aas Nina‐Beate Liabakk Siri Sæterstad Khac Thanh Phong Chau Bodil Merete Kavli Valentyn Oksenych

To ensure genome stability, mammalian cells employ several DNA repair pathways. Nonhomologous DNA end joining (NHEJ) is the DNA repair process that fixes double-strand breaks throughout the cell cycle. NHEJ is involved in the development of B and T lymphocytes through its function in V(D)J recombination and class switch recombination (CSR). NHEJ consists of several core and accessory factors, i...

Journal: :American journal of human genetics 2015
Jennie E Murray Mirjam van der Burg Hanna IJspeert Paula Carroll Qian Wu Takashi Ochi Andrea Leitch Edward S Miller Boris Kysela Alireza Jawad Armand Bottani Francesco Brancati Marco Cappa Valerie Cormier-Daire Charu Deshpande Eissa A Faqeih Gail E Graham Emmanuelle Ranza Tom L Blundell Andrew P Jackson Grant S Stewart Louise S Bicknell

Non-homologous end joining (NHEJ) is a key cellular process ensuring genome integrity. Mutations in several components of the NHEJ pathway have been identified, often associated with severe combined immunodeficiency (SCID), consistent with the requirement for NHEJ during V(D)J recombination to ensure diversity of the adaptive immune system. In contrast, we have recently found that biallelic mut...

Journal: :The EMBO journal 1999
Y Yamaguchi-Iwai E Sonoda M S Sasaki C Morrison T Haraguchi Y Hiraoka Y M Yamashita T Yagi M Takata C Price N Kakazu S Takeda

Yeast Mre11 functions with Rad50 and Xrs2 in a complex that has pivotal roles in homologous recombination (HR) and non-homologous end-joining (NHEJ) DNA double-strand break (DSB) repair pathways. Vertebrate Mre11 is essential. Conditionally, MRE11 null chicken DT40 cells accumulate chromosome breaks and die upon Mre11 repression, showing frequent centrosome amplification. Mre11 deficiency also ...

2007
Michael B. Smeaton Paul S. Miller Gary Ketner Les A. Hanakahi

The repair of DNA by nucleotide excision repair (NER) and non-homologous end joining (NHEJ) is essential for maintenance of genomic integrity and cell viability. Examination of NHEJ and NER in vitro using cell-free extracts has led to a deeper understanding of the biochemical mechanisms that underlie these processes. Current methods for production of whole-cell extracts (WCEs) to investigate NE...

Journal: :Science 2012
Agnel Sfeir Titia de Lange

The telomere end-protection problem is defined by the aggregate of DNA damage signaling and repair pathways that require repression at telomeres. To define the end-protection problem, we removed the whole shelterin complex from mouse telomeres through conditional deletion of TRF1 and TRF2 in nonhomologous end-joining (NHEJ) deficient cells. The data reveal two DNA damage response pathways not p...

Journal: :The Journal of Cell Biology 2005
Jong-Soo Kim Tatiana B. Krasieva Hitoshi Kurumizaka David J. Chen A. Malcolm R. Taylor Kyoko Yokomori

Damage recognition by repair/checkpoint factors is the critical first step of the DNA damage response. DNA double strand breaks (DSBs) activate checkpoint signaling and are repaired by nonhomologous end-joining (NHEJ) and homologous recombination (HR) pathways. However, in vivo kinetics of the individual factor responses and the mechanism of pathway choice are not well understood. We report cel...

2013
Edward Bartlett Nigel C. Brissett Aidan J. Doherty

Non homologous end-joining (NHEJ) pathways repair DNA double-strand breaks (DSBs) in eukaryotes and many prokaryotes, although not reported to operate in the third domain of life, archaea. Here, we describe a complete NHEJ complex, consisting of DNA ligase (Lig), polymerase (Pol), phosphoesterase (PE) and Ku from a mesophillic archaeon, Methanocella paludicola (Mpa). Mpa ligase has limited DNA ...

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