نتایج جستجو برای: myopathy

تعداد نتایج: 12241  

Journal: :Journal of Korean Medical Science 2001
M. J. Lee J. S. Lee M. C. Lee

Apoptotic cell death of differentiated skeletal muscle has been reported in an experimental steroid-induced myopathy of rats. To investigate the underlying molecular changes in the apoptosis of skeletal muscle, in situ end labeling (ISEL), Fas expression, and Western blot analysis for apoptosis-related proteins in the soleus muscle of triamcinolone acetonide (TA)-induced myopathy of rats were s...

Journal: :Journal of neurology, neurosurgery, and psychiatry 1999
S Kiechl U Kohlendorfer C Thaler D Skladal M Jaksch B Obermaier-Kusser J Willeit

OBJECTIVE To characterise the main clinical phenotypes of debrancher deficiency myopathy and to increase awareness for this probably underdiagnosed disorder. METHODS The diagnosis of debrancher deficiency was established by laboratory tests, EMG, and muscle and liver biopsy. RESULTS Four patients with debrancher deficiency myopathy were identified in the Tyrol, a federal state of Austria wi...

Journal: :Neurology 2011
V Guergueltcheva K Peeters J Baets C Ceuterick-de Groote J J Martin A Suls E De Vriendt V Mihaylova T Chamova L Almeida-Souza E Ydens C Tzekov G Hadjidekov M Gospodinova K Storm E Reyniers S Bichev P F M van der Ven D O Fürst V Mitev H Lochmüller V Timmerman I Tournev P De Jonghe A Jordanova

OBJECTIVE In this study, we investigated the detailed clinical findings and underlying genetic defect in 3 presumably related Bulgarian families displaying dominantly transmitted adult onset distal myopathy with upper limb predominance. METHODS We performed neurologic, electrophysiologic, radiologic, and histopathologic analyses of 13 patients and 13 at-risk but asymptomatic individuals from ...

2015
Lucia Marseglia Gabriella D’Angelo Sara Manti Vincenzo Salpietro Teresa Arrigo Vittorio Cavallari Eloisa Gitto

BACKGROUND Nemaline myopathy is a rare, non progressive congenital skeletal muscle disorder defined by the presence of inclusions known as nemaline rods in muscle fibers. Several clinical subtypes have been described, according to degree of muscle weakness, severity and age at onset. The course of nemaline myopathy is very slowly progressive, and death is usually due to respiratory failure. Car...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2002
Anna Wredenberg Rolf Wibom Hans Wilhelmsson Caroline Graff Heidi H Wiener Steven J Burden Anders Oldfors Håkan Westerblad Nils-Göran Larsson

We have generated an animal model for mitochondrial myopathy by disrupting the gene for mitochondrial transcription factor A (Tfam) in skeletal muscle of the mouse. The knockout animals developed a myopathy with ragged-red muscle fibers, accumulation of abnormally appearing mitochondria, and progressively deteriorating respiratory chain function in skeletal muscle. Enzyme histochemistry, electr...

2016
Metha Apiwattanakul Margherita Milone Sean J. Pittock Thomas J. Kryzer James P. Fryer Orna O'toole Andrew Mckeon Vanda A. Lennon

INTRODUCTION Paraneoplastic autoantibody screening of 150,000 patient sera by tissue-based immunofluorescence incidentally revealed 170 with unsuspected signal recognition particle (SRP) immunoglobulin G (IgG), which is a recognized biomarker of autoimmune myopathy. Of the 77 patients with available information, 54 had myopathy. We describe the clinical/laboratory associations. METHODS Distin...

2016
Josef Finsterer Claudia Stöllberger

OBJECTIVES Arrhythmogenic right ventricular dysplasia (ARVD) is a rare, genetic disorder predominantly affecting the right ventricle. There is increasing evidence that in some cases, ARVD is due to mutations in genes, which have also been implicated in primary myopathies. This review gives an overview about myopathy-associated ARVD and how these patients can be managed. METHODS A literature r...

Journal: :Annals of child neurology 2022

This corrects the article "Clinical Characteristics and Neurologic Outcomes of X-Linked Myotubular Myopathy" on page 127.

Journal: :Postgraduate medical journal 1987
R A Sheehan-Dare A V Simmons

A 57 year old female presented with an amyloid myopathy in association with lambda light chain myeloma. Treatment with melphalan and prednisolone resulted in remission of both myeloma and myopathy.

Journal: :Neurology 2004
Nortina Shahrizaila James Lowe Adrian Wills

The authors describe familial tubular aggregate myopathy associated with abnormal pupils. Four family members from two generations had myopathy and pupillary abnormalities. The myopathologic findings consisted of tubular aggregates in many fibers but predominantly type I fibers.

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