نتایج جستجو برای: methylmalonic academia

تعداد نتایج: 18694  

Journal: :Journal of Biological Chemistry 2004

2011
Zohre Karamizadeh Setilla Dalili Hamdollah Karamifar Gholam Hossein Amirhakimi

Dear Editor, Methylmalonic acidemia, one of the organic acidemias, is associated with a variety of clinical presentations ranging from very sick newborn infants to asymptomatic adults, regardless of the nature of the enzymatic defect or the biochemical abnormalities. A 6-year-old boy with a past history of methylmalonic acidemia presented to the emergency room with a one-week history of inflamm...

Journal: :Biology of the neonate 2004
Anne-Lise Bjørke Monsen Stein Emil Vollset Helga Refsum Trond Markestad Per Magne Ueland

Hematological parameters, serum cobalamin and folate levels, and the concentrations of the functional markers plasma methylmalonic acid and total homocysteine were determined in 173 newborns and 46 infants at 6 weeks to see whether maternal smoking influences the hematological parameters and the vitamin status of the newborn. At birth, there was a strong inverse correlation between the number o...

2014
Matthias R Baumgartner Friederike Hörster Carlo Dionisi-Vici Goknur Haliloglu Daniela Karall Kimberly A Chapman Martina Huemer Michel Hochuli Murielle Assoun Diana Ballhausen Alberto Burlina Brian Fowler Sarah C Grünert Stephanie Grünewald Tomas Honzik Begoña Merinero Celia Pérez-Cerdá Sabine Scholl-Bürgi Flemming Skovby Frits Wijburg Anita MacDonald Diego Martinelli Jörn Oliver Sass Vassili Valayannopoulos Anupam Chakrapani

Methylmalonic and propionic acidemia (MMA/PA) are inborn errors of metabolism characterized by accumulation of propionic acid and/or methylmalonic acid due to deficiency of methylmalonyl-CoA mutase (MUT) or propionyl-CoA carboxylase (PCC). MMA has an estimated incidence of ~ 1: 50,000 and PA of ~ 1:100'000 -150,000. Patients present either shortly after birth with acute deterioration, metabolic...

2017
Mehmet Gunduz Ozlem Unal Birce Dilge Taskin Zeynep Selen Karalok

Cobalamin C deficiency (CblC) is the most frequent inborn error of cobalamin (Cbl) metabolism, which has a wide clinical spectrum. Cbl C defect causes the accumulation of methylmalonic acid and homocysteine and decreased methionine synthesis. Here we presented two distinct clinical forms of patients with CblC. First patient with early onset form was presented with failure to thrive, mild hypoto...

Journal: :Clinical chemistry 1977
G Morrow B Revsin J Lebowitz W Britt H Giles

We report a method for rapid prenatal detection of methylmalonic acidemia, consisting of measuring methylmalonly-CoA mutase (EC 5.4.99.2) activity in non-cultured amniotic cells and measuring the concentration of methylmalonate in the amniotic fluid. Immediate stabilization of the mutase activity in the non-cultured amniotic cell by its coenzyme adenosycobalamin, and use of methylmalonyl-CoA wi...

2002
Joshua W. Miller Marisa I. Ramos Marjorie G. Garrod Margaret A. Flynn

A common polymorphism (775G>C) in the vitamin B12 transport protein, transcobalamin II (TCII), has been identified in which proline replaces arginine at codon 259. We determined the influence of TCII genotype on indices of B12 status, including total serum B12, the amount of B12 bound to TCII (holoTCII), methylmalonic acid, and homocysteine, in 128 healthy older adults (ages 40-88 years). Mean ...

Journal: :World journal of pediatrics : WJP 2014
Raymond Y Wang Richard C Chang Mary E Sowa Anthony C Chang Jose E Abdenur

BACKGROUND Effects of circulatory arrest upon an inborn error of metabolism patient are unknown. METHODS A retrospective chart review was performed of outcome and biochemical parameters obtained during palliative cardiac surgery for a mutase-deficient methylmalonic aciduria patient with Ebstein's cardiac anomaly was performed. RESULTS The levels of ammonia, methylmalonic acid, free carnitin...

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