نتایج جستجو برای: internal tandem duplication

تعداد نتایج: 302669  

Journal: :Haematologica 2003
Isabel Moreno Guillermo Martín Pascual Bolufer Eva Barragán Eva Rueda José Román Pascual Fernández Pilar León Armando Mena José Cervera Antonio Torres Miguel A Sanz

BACKGROUND AND OBJECTIVES Cytogenetics is the most important prognostic factor in acute myeloid leukemia (AML). However, a high proportion of patients show normal or intermediate-risk karyotypes. In these patients, other determinants could help to identify those with a higher risk of relapse. Recently, internal tandem duplications (ITD) and D835 mutations in FLT3 tyrosine kinase receptor have b...

2017
Vanessa Y.N. de Arruda Lisa N. Matsuzaki Maria de Lourdes Chauffaille

cute myeloid leukemia (AML) is the most frequent acute eukemia of adult patients with an estimated 10,070 new cases n Brazil in 2016. Most of the cases are de novo, without a efined etiology, and around two thirds of the patients will die f the disease, according to the Instituto Nacional de Câncer INCA).1 Genetic aberrations may help to differentiate patients ho have a good from those with a d...

Journal: :Journal of the National Comprehensive Cancer Network : JNCCN 2015
Smith Giri Shadi Hamdeh Vijaya Raj Bhatt James K Schwarz

Old age (≤65 years), relapsed or refractory disease, and the presence of FMS-like receptor tyrosine kinase-3 (FLT3) internal tandem duplication (ITD) mutation are poor prognostic factors in acute myeloid leukemia (AML). FLT3 inhibitors such as sorafenib have been shown to have a potential role in treating relapsed or refractory AML with FLT3 mutations. In the present report, the use of sorafeni...

Journal: :Blood 2006
Jessica A Pollard Todd A Alonzo Robert B Gerbing William G Woods Beverly J Lange David A Sweetser Jerald P Radich Irwin D Bernstein Soheil Meshinchi

Acute myeloid leukemia (AML) is a clonal disease characterized by heterogeneous involvement of hematopoietic stem cell/progenitor cell populations. Using FLT3 internal tandem duplication (FLT3/ITD) as a molecular marker, we tested the hypothesis that clinical outcome in AML correlates with disease involvement of CD34(+)/CD33(-) precursors. Diagnostic specimens from 24 children with FLT3/ITD-pos...

Journal: :Oman medical journal 2013
Adhra Al-Mawali David Gillis Ian Lewis

OBJECTIVES Constitutive activation of the fms-like tyrosine kinase 3 (FLT3) receptor by internal tandem duplication (ITD) of the juxtamembrane region has been described in patients with acute myeloid leukemia. FLT3/ITDs are present in about 20-30% of all acute myeloid leukemia cases. It has been shown that the mutation is correlated with worse prognosis. However, none of the previous studies in...

Journal: :Blood 2001
S Meshinchi W G Woods D L Stirewalt D A Sweetser J D Buckley T K Tjoa I D Bernstein J P Radich

The Flt3 gene encodes a tyrosine kinase receptor that regulates proliferation and differentiation of hematopoietic stem cells. An internal tandem duplication of the Flt3 gene (Flt3/ITD) has been reported in acute myelogenous leukemia (AML) and may be associated with poor prognosis. We analyzed diagnostic bone marrow specimens from 91 pediatric patients with AML treated on Children's Cancer Grou...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2006
Christophe Marzac I Teyssandier Ors'Anton Calendini Jean-Yves Perrot Anne-Marie Faussat Ruoping Tang Nicole Casadevall Jean-Pierre Marie Ollivier Legrand

PURPOSE Patients with adult acute myeloid leukemia (AML) with intermediate cytogenetics remain a heterogeneous group with highly variable individual prognoses. New molecular markers could help to refine cytogenetic stratification. EXPERIMENTAL DESIGN We assessed P-glycoprotein (Pgp) activity and Flt3 internal tandem duplication (ITD+) because of their known prognostic value and because they m...

Journal: :Genetics and molecular research : GMR 2008
C O Possamai F M Carvalho M F C Silva E V Wolfgramm M P N Sartori F S V Malta V P Ribeiro V P Spina K B Gomes A C S Ferreira I D Louro

Charcot-Marie-Tooth type 1A disease (CMT1A) is most frequently caused by a tandem DNA duplication of a 1.4-Mb genomic fragment in the 17p11.2-12 chromosomal region. The disease is probably the product of a dosage effect of the peripheral myelin protein 22 gene located within the duplicated segment. We sought to study the largest reported Brazilian family with suspected diagnosis of CMT1A using ...

Journal: :The Journal of Experimental Medicine 2007
Jennifer O'Neil Joelle Tchinda Alejandro Gutierrez Lisa Moreau Richard S. Maser Kwok-Kin Wong Wei Li Keith McKenna X. Shirley Liu Bin Feng Donna Neuberg Lewis Silverman Daniel J. DeAngelo Jeffery L. Kutok Rodney Rothstein Ronald A. DePinho Lynda Chin Charles Lee A. Thomas Look

Recent studies have demonstrated that the MYB oncogene is frequently duplicated in human T cell acute lymphoblastic leukemia (T-ALL). We find that the human MYB locus is flanked by 257-bp Alu repeats and that the duplication is mediated somatically by homologous recombination between the flanking Alu elements on sister chromatids. Nested long-range PCR analysis indicated a low frequency of homo...

2016
Ohad Elishco Farzad Farnoud Moshe Schwartz Jehoshua Bruck

To be considered for the 2016 IEEE Jack Keil Wolf ISIT Student Paper Award. We study random stringduplication systems, called Pólya string models, motivated by certain random mutation processes in the genome of living organisms. Unlike previous works that study the combinatorial capacity of string-duplication systems, or peripheral properties such as symbol frequency, this work provides exact c...

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