نتایج جستجو برای: ii bcl2

تعداد نتایج: 584067  

Journal: :Molecular cancer therapeutics 2016
Ryan S Soderquist Alan Eastman

Antiapoptotic BCL2 proteins play a major role in tumor cell survival. Hence, BCL2 inhibitors have been developed as direct inducers of apoptosis. ABT-199 (venetoclax) received breakthrough therapy designation from the FDA due to its apparent efficacy in CLL and AML. However, resistance to ABT-199 is mediated by other BCL2 proteins including BCLXL and MCL1. Considerable effort has been expended ...

2014
A Barreca C Martinengo L Annaratone L Righi A Chiappella M Ladetto A Demurtas L Chiusa A Stacchini N Crosetto A van Oudenaarden R Chiarle

Most follicular lymphomas (FLs) are genetically defined by the t(14;18)(q32;q21) translocation that juxtaposes the BCL2 gene to the immunoglobulin heavy chain (IgH) 3' regulatory regions (IgH-3'RRs). Despite this recurrent translocation, FL cases are heterogeneous in terms of intratumoral clonal diversity for acquired mutations and variations in the tumor microenvironment. Here we describe an a...

2015
Ting-Xun Lu Lei Fan Li Wang Jia-Zhu Wu Kou-Rong Miao Jin-Hua Liang Qi-Xing Gong Zhen Wang Ken H. Young Wei Xu Zhi-Hong Zhang Jian-Yong Li

Diffuse large B-cell lymphoma (DLBCL) is the most common non-Hodgkin lymphoma (NHL). Patients with DLBCL harboring MYC aberrations concurrent with BCL2 or/and BCL6 aberrations constitute a specific group with extremely poor outcome. In this study, we retrospectively investigated the incidence and prognosis of MYC, BCL2, and BCL6 aberrations with DLBCL patients in Chinese population. We applied ...

2017
Charlotte Viant Sophie Guia Robert J. Hennessy Jai Rautela Kim Pham Claire Bernat Wilford Goh Yuhao Jiao Rebecca Delconte Michael Roger Vanina Simon Fernando Souza-Fonseca-Guimaraes Stephanie Grabow Gabrielle T. Belz Benjamin T. Kile Andreas Strasser Daniel Gray Phillip D. Hodgkin Bruce Beutler Eric Vivier Sophie Ugolini Nicholas D. Huntington

Natural killer (NK) cells are innate lymphoid cells with antitumor functions. Using an N-ethyl-N-nitrosourea (ENU)-induced mutagenesis screen in mice, we identified a strain with an NK cell deficiency caused by a hypomorphic mutation in the Bcl2 (B cell lymphoma 2) gene. Analysis of these mice and the conditional deletion of Bcl2 in NK cells revealed a nonredundant intrinsic requirement for BCL...

Journal: :Blood 2012
Abner Louissaint Adam M Ackerman Dora Dias-Santagata Judith A Ferry Ephraim P Hochberg Mary S Huang A John Iafrate Daniel O Lara Geraldine S Pinkus Itziar Salaverria Zakir Siddiquee Reiner Siebert Howard J Weinstein Lawrence R Zukerberg Nancy Lee Harris Robert P Hasserjian

Pediatric follicular lymphoma (PFL) is a variant of follicular lymphoma (FL) presenting as localized lymphadenopathy in children. Unlike conventional adult FL, PFL typically does not recur or progress. Clear diagnostic criteria for PFL are lacking, and it is uncertain whether this indolent lymphoma is defined by age or may occur in adults. We analyzed 27 FL in patients < 40 years of age and fou...

Journal: :Cancer research 2004
Gerhard Regl Maria Kasper Harald Schnidar Thomas Eichberger Graham W Neill Michael P Philpott Harald Esterbauer Cornelia Hauser-Kronberger Anna-Maria Frischauf Fritz Aberger

Aberrant activation of the Hedgehog (HH)/GLI signaling pathway has been implicated in the development of basal cell carcinoma (BCC). The zinc finger transcription factors GLI1 and GLI2 are considered mediators of the HH signal in epidermal cells, although their tumorigenic nature and their relative contribution to tumorigenesis are only poorly understood. To shed light on the respective role of...

Journal: :Cancer research 1998
S Haldar A Basu C M Croce

Taxoids and other microtubule-damaging drugs are known to induce Bcl2 phosphorylation at the G2-M phase of the cell cycle, with concomitant apoptosis in malignant cells derived from a variety of human malignancies, including leukemia, lymphoma, and breast and prostate cancer. We have investigated the ability of another antineoplastic drug, dolastatin 10, in inducing Bcl2 phosphorylation and apo...

Journal: :Blood 1997
O Monni H Joensuu K Franssila J Klefstrom K Alitalo S Knuutila

Gene activation by translocation between an oncogene and an immunoglobulin heavy-chain gene, which leads to increased expression of the oncoprotein, is a well-known mechanism in the genesis of B-cell lymphomas. In contrast, the role of gene amplification in activation of oncogenes in non-Hodgkin's lymphomas is poorly characterized. To study the BCL2 amplification we performed comparative genomi...

2016
Stephanie Berger Erik Procko Daciana Margineantu Erinna F Lee Betty W Shen Alex Zelter Daniel-Adriano Silva Kusum Chawla Marco J Herold Jean-Marc Garnier Richard Johnson Michael J MacCoss Guillaume Lessene Trisha N Davis Patrick S Stayton Barry L Stoddard W Douglas Fairlie David M Hockenbery David Baker

Many cancers overexpress one or more of the six human pro-survival BCL2 family proteins to evade apoptosis. To determine which BCL2 protein or proteins block apoptosis in different cancers, we computationally designed three-helix bundle protein inhibitors specific for each BCL2 pro-survival protein. Following in vitro optimization, each inhibitor binds its target with high picomolar to low nano...

2015
Jose Manuel Bravo-San Pedro Yongjie Wei Valentina Sica Maria Chiara Maiuri Zhongju Zou Guido Kroemer Beth Levine

Disruption of the complex of BECN1 with BCL2 or BCL2L1/BCL-XL is an essential switch that turns on cellular autophagy in response to environmental stress or treatment with BH3 peptidomimetics. Recently, it has been proposed that BCL2 and BCL2L1/BCL-XL may inhibit autophagy indirectly through a mechanism dependent on the proapoptotic BCL2 family members, BAX and BAK1. Here we report that the BH3...

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