نتایج جستجو برای: egfr genes

تعداد نتایج: 454104  

Journal: :Molecular cancer therapeutics 2005
Craig D Woodworth Evan Michael Dan Marker Sarah Allen Laura Smith Matthias Nees

The epidermal growth factor receptor (EGFR) is overexpressed in several types of human cancer, and inhibition of EGFR function is a promising strategy for cancer therapy. We used cDNA microarrays to examine alterations in gene expression after treatment of carcinoma cells with PD153035, a specific and reversible inhibitor of EGFR function. When human cervical carcinoma cells were grown on a col...

2016
Kumari Sonal Choudhary Neha Rohatgi Skarphedinn Halldorsson Eirikur Briem Thorarinn Gudjonsson Steinn Gudmundsson Ottar Rolfsson

Epithelial to mesenchymal transition (EMT) is an important event during development and cancer metastasis. There is limited understanding of the metabolic alterations that give rise to and take place during EMT. Dysregulation of signalling pathways that impact metabolism, including epidermal growth factor receptor (EGFR), are however a hallmark of EMT and metastasis. In this study, we report th...

2010
Yu Zhang Qing Huang Jung-Chien Cheng Yoshihiro Nishi Toshihiko Yanase He-Feng Huang Peter CK Leung

BACKGROUND Homeobox (HOX) genes encode transcription factors, which regulate cell proliferation, differentiation, adhesion, and migration. The deregulation of HOX genes is frequently associated with human reproductive system disorders. However, knowledge regarding the role of HOX genes in human granulosa cells is limited. METHODS To determine the role of HOXA7 in the regulation and associated...

2017
In-Su Kim Sushruta Koppula Shin-Young Park Dong-Kug Choi

We employed transcriptome analysis of epidermal growth factor receptor related gene expression changes in cellular and animal models of Parkinson's disease (PD). We used a well-known Parkinsonian toxin 1-methyl-4-phenylpyridine (MPP⁺) to induce neuronal apoptosis in the human neuroblastoma SH-SY5Y cell line. The MPP⁺-treatment of SH-SY5Y cells was capable of inducing neuro-apoptosis, but it rem...

2013
Wen-Qing Li Nan Hu Zhaoming Wang Kai Yu Hua Su Lemin Wang Chaoyu Wang Stephen J. Chanock Laurie Burdett Ti Ding You-Lin Qiao Jin-Hu Fan Yuan Wang Yi Xu Carol Giffen Xiaoqin Xiong Gwen Murphy Margaret A. Tucker Sanford M. Dawsey Neal D. Freedman Christian C. Abnet Alisa M. Goldstein Philip R. Taylor

The epidermal growth factor receptor (EGFR) signaling pathway regulates cell proliferation, differentiation, and survival, and is frequently dysregulated in esophageal and gastric cancers. Few studies have comprehensively examined the association between germline genetic variants in the EGFR pathway and risk of esophageal and gastric cancers. Based on a genome-wide association study in a Han Ch...

2015
Yi-Nan Liu Tzu-Hua Chang Meng-Feng Tsai Shang-Gin Wu Tzu-Hsiu Tsai Hsuan-Yu Chen Sung-Liang Yu James Chih-Hsin Yang Jin-Yuan Shih

Epidermal growth factor receptor (EGFR)-targeted strategy is limited by resistance. We identify the potential genes involved in EGFR TKI (tyrosine kinase inhibitor) resistance and study the therapeutic mechanism in the non-small cell lung cancers. Potential genes involved in resistance were examined by analyzing datasets from a pair of EGFR TKI-sensitive (PC9) and TKI-resistant cells (PC9/gef)....

Journal: :Human molecular genetics 2012
Daniel I Chasman Christian Fuchsberger Cristian Pattaro Alexander Teumer Carsten A Böger Karlhans Endlich Matthias Olden Ming-Huei Chen Adrienne Tin Daniel Taliun Man Li Xiaoyi Gao Mathias Gorski Qiong Yang Claudia Hundertmark Meredith C Foster Conall M O'Seaghdha Nicole Glazer Aaron Isaacs Ching-Ti Liu Albert V Smith Jeffrey R O'Connell Maksim Struchalin Toshiko Tanaka Guo Li Andrew D Johnson Hinco J Gierman Mary F Feitosa Shih-Jen Hwang Elizabeth J Atkinson Kurt Lohman Marilyn C Cornelis Asa Johansson Anke Tönjes Abbas Dehghan Jean-Charles Lambert Elizabeth G Holliday Rossella Sorice Zoltan Kutalik Terho Lehtimäki Tõnu Esko Harshal Deshmukh Sheila Ulivi Audrey Y Chu Federico Murgia Stella Trompet Medea Imboden Stefan Coassin Giorgio Pistis Tamara B Harris Lenore J Launer Thor Aspelund Gudny Eiriksdottir Braxton D Mitchell Eric Boerwinkle Helena Schmidt Margherita Cavalieri Madhumathi Rao Frank Hu Ayse Demirkan Ben A Oostra Mariza de Andrade Stephen T Turner Jingzhong Ding Jeanette S Andrews Barry I Freedman Franco Giulianini Wolfgang Koenig Thomas Illig Christa Meisinger Christian Gieger Lina Zgaga Tatijana Zemunik Mladen Boban Cosetta Minelli Heather E Wheeler Wilmar Igl Ghazal Zaboli Sarah H Wild Alan F Wright Harry Campbell David Ellinghaus Ute Nöthlings Gunnar Jacobs Reiner Biffar Florian Ernst Georg Homuth Heyo K Kroemer Matthias Nauck Sylvia Stracke Uwe Völker Henry Völzke Peter Kovacs Michael Stumvoll Reedik Mägi Albert Hofman Andre G Uitterlinden Fernando Rivadeneira Yurii S Aulchenko Ozren Polasek Nick Hastie Veronique Vitart Catherine Helmer Jie Jin Wang Bénédicte Stengel Daniela Ruggiero Sven Bergmann Mika Kähönen Jorma Viikari Tiit Nikopensius Michael Province Shamika Ketkar Helen Colhoun Alex Doney Antonietta Robino Bernhard K Krämer Laura Portas Ian Ford Brendan M Buckley Martin Adam Gian-Andri Thun Bernhard Paulweber Margot Haun Cinzia Sala Paul Mitchell Marina Ciullo Stuart K Kim Peter Vollenweider Olli Raitakari Andres Metspalu Colin Palmer Paolo Gasparini Mario Pirastu J Wouter Jukema Nicole M Probst-Hensch Florian Kronenberg Daniela Toniolo Vilmundur Gudnason Alan R Shuldiner Josef Coresh Reinhold Schmidt Luigi Ferrucci David S Siscovick Cornelia M van Duijn Ingrid B Borecki Sharon L R Kardia Yongmei Liu Gary C Curhan Igor Rudan Ulf Gyllensten James F Wilson Andre Franke Peter P Pramstaller Rainer Rettig Inga Prokopenko Jacqueline Witteman Caroline Hayward Paul M Ridker Afshin Parsa Murielle Bochud Iris M Heid W H Linda Kao Caroline S Fox Anna Köttgen

In conducting genome-wide association studies (GWAS), analytical approaches leveraging biological information may further understanding of the pathophysiology of clinical traits. To discover novel associations with estimated glomerular filtration rate (eGFR), a measure of kidney function, we developed a strategy for integrating prior biological knowledge into the existing GWAS data for eGFR fro...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2011
Shi-Yi Yang Tsung-Ying Yang Kun-Chieh Chen Yao-Jen Li Kuo-Hsuan Hsu Chi-Rne Tsai Chih-Yi Chen Chung-Ping Hsu Jiun-Yi Hsia Cheng-Yen Chuang Ying-Huang Tsai Kuan-Yu Chen Ming-Shyan Huang Wu-Chou Su Yuh-Min Chen Chao A Hsiung Chen-Yang Shen Gee-Chen Chang Pan-Chyr Yang Chien-Jen Chen

PURPOSE To assess whether polymorphisms of genes related to estrogen biosynthesis and metabolism are associated with EGFR mutations. EXPERIMENTAL DESIGN We studied 617 patients with lung adenocarcinoma, including 302 never-smoking women. On the basis of multiple candidate genes approach, the effects of polymorphisms of CYP17, CYP19A1, ERα, and COMT in association with the occurrence of EGFR m...

2012
KOJIRO YANO

Inhibitors of lipid metabolic pathways, particularly drugs targeting the mevalonate pathway, have been suggested to be valuable in enhancing the effectiveness of epidermal growth factor receptor-tyrosine kinase inhibitors (EGFR-TKIs) and these compounds may also be effective in patients with inherent or acquired resistance to EGFR-TKIs. The present study examined gene expression profiles in lun...

Journal: :The Journal of clinical investigation 2017
Loren J Martin Shad B Smith Arkady Khoutorsky Claire A Magnussen Alexander Samoshkin Robert E Sorge Chulmin Cho Noosha Yosefpour Sivaani Sivaselvachandran Sarasa Tohyama Tiffany Cole Thang M Khuong Ellen Mir Dustin G Gibson Jeffrey S Wieskopf Susana G Sotocinal Jean Sebastien Austin Carolina B Meloto Joseph H Gitt Christos Gkogkas Nahum Sonenberg Joel D Greenspan Roger B Fillingim Richard Ohrbach Gary D Slade Charles Knott Ronald Dubner Andrea G Nackley Alfredo Ribeiro-da-Silva G Gregory Neely William Maixner Dmitri V Zaykin Jeffrey S Mogil Luda Diatchenko

The EGFR belongs to the well-studied ErbB family of receptor tyrosine kinases. EGFR is activated by numerous endogenous ligands that promote cellular growth, proliferation, and tissue regeneration. In the present study, we have demonstrated a role for EGFR and its natural ligand, epiregulin (EREG), in pain processing. We show that inhibition of EGFR with clinically available compounds strongly ...

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