نتایج جستجو برای: chronic myeloproliferative disorder

تعداد نتایج: 1043759  

2014
Ruben A. Mesa Jean - Jacques Kiladjian Srdan Verstovsek Haifa Kathrin Al - Ali Jason Gotlib Heinz Gisslinger Richard Levy Andres Siulnik Vikas Gupta Mahmudul Khan John F. DiPersio John V. Catalano Deborah S. Hunter Laurent Knoops Michael Deininger Carole Miller Alessandro M. Vannucchi Richard T. Silver Tiziano Barbui Moshe Talpaz Giovanni Barosi Elliott F. Winton Estella Mendeson Jimmie H. Harvey Murat O. Arcasoy Elizabeth Hexner Roger M. Lyons Ronald Paquette Azra Raza William Sun Victor Sandor Hagop M. Kantarjian Claire Harrison

© F rra ta St ort i F ou nd ati on approved in November 2011 by the US Food and Drug Administration for the treatment of intermediateor highrisk myelofibrosis and more recently by Health Canada and the European Commission for the treatment of myelofibrosis-related splenomegaly or symptoms. Ruxolitinib was approved based on data from two pivotal phase 3 COntrolled MyeloFibrosis study with ORal J...

Journal: :Best practice & research. Clinical haematology 2007
Kenneth Kaushansky

In 1951, William Dameshek speculated on the common origin of the chronic myeloproliferative disorders--polycythemia vera (PV), essential thrombocythemia (ET), chronic idiopathic myelofibrosis (IMF), and chronic myelogenous leukemia (CML). Subsequent work suggested that all arose from the hematopoietic stem cell. About 20 years ago the oncogene responsible for CML, bcr-abl, was identified, and m...

Journal: :Hematology. American Society of Hematology. Education Program 2003
Jerry L Spivak Giovanni Barosi Gianni Tognoni Tiziano Barbui Guido Finazzi Roberto Marchioli Monia Marchetti

The Philadelphia chromosome-negative chronic myeloproliferative disorders (CMPD), polycythemia vera (PV), essential thrombocythemia (ET) and chronic idiopathic myelofibrosis (IMF), have overlapping clinical features but exhibit different natural histories and different therapeutic requirements. Phenotypic mimicry amongst these disorders and between them and nonclonal hematopoietic disorders, la...

Journal: :Cancer research 2004
José L Vizmanos Francisco J Novo José P Román E Joanna Baxter Idoya Lahortiga María J Larráyoz María D Odero Pilar Giraldo María J Calasanz Nicholas C P Cross

We describe a new PDGFRB fusion associated with a t(5;14)(q33;q24) in a patient with a longstanding chronic myeloproliferative disorder with eosinophilia. After confirmation of PDGFRB involvement and definition of the chromosome 14 breakpoint by fluorescence in situ hybridization, candidate partner genes were selected on the basis of the presence of predicted oligomerization domains believed to...

2013
Caroline FA Moreira-Nunes Tereza CB Azevedo Ana CS Beltrão Larissa TVM Francês Rodrigo GMA Sousa Israel T Silva Artur Silva Wilson A Silva José AR Lemos

Background Chronic Myeloid Leukemia (CML) is a clonal myeloproliferative disorder characterized by formation of BCR-ABL fusion that encodes the p210 oncoprotein, which has a tyrosine kinase activity that confers an adaptive advantage to leukemic cells. Imatinib mesylate (IM) acts specifically on p210. Imatinib is able to reduce the differentiated cells (CD66b+) efficiently, but it has not the s...

Journal: :Journal of clinical pathology 1985
L Camba M V Joyner

We report a case of non-sideroblastic refractory anaemia which evolved to a double lymphomyeloproliferative disorder. At presentation, bone marrow appearances and peripheral blood pancytopenia without myelomonocytosis were consistent with a diagnosis of non-sideroblastic refractory anaemia. Subsequently, the patient developed pronounced myelomonocytosis and lymphocytosis with prolymphocytes. Li...

2015
Giovanni Amabile Annalisa Di Ruscio Fabian Müller Robert S Welner Henry Yang Alexander K Ebralidze Hong Zhang Elena Levantini Lihua Qi Giovanni Martinelli Thijn Brummelkamp Michelle M Le Beau Maria E Figueroa Christoph Bock Daniel G Tenen

Chronic myeloid leukaemia (CML) is a myeloproliferative disorder characterized by the genetic translocation t(9;22)(q34;q11.2) encoding for the BCR-ABL fusion oncogene. However, many molecular mechanisms of the disease progression still remain poorly understood. A growing body of evidence suggests that the epigenetic abnormalities are involved in tyrosine kinase resistance in CML, leading to le...

2014
DONG-WEN WANG YONG ZHANG JIAN-MIN YAO ZHI-BIN XIAO

Essential thrombocythemia (ET) is a chronic clonal myeloproliferative disorder, which is often complicated by arterial or venous thrombosis and idiopathic bleeding diathesis. The present study reports a female patient with ET complicated by acute myocardial infarction, leading to ventricular aneurysm following interventional therapy for 3 years and a subsequent in-stent restenosis. Following ca...

Journal: :Leukemia research reports 2016
Rochelle Nagales Nagamos Teresa Gentile Neerja Vajpayee

Chronic myelogenous leukemia (CML) is a myeloproliferative disorder where over a period of time 15-20% of patients show blastic transformation with majority transforming into acute myeloid leukemia, most of which are of granulocytic lineage. Erythroid blast phase of CML is relatively rare with the incidence ranging from 0-10%. Further the incidence of acute erythroid leukemia by itself is fairl...

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