نتایج جستجو برای: c282y

تعداد نتایج: 552  

2015
Libia M Rodriguez Mabel C Giraldo Laura I Velasquez Cristiam M Alvarez Luis F Garcia Marlene Jimenez-Del-Rio Carlos Velez-Pardo

A significant association between HFE gene mutations and the HLA-A*03-B*07 and HLA-A*29-B*44 haplotypes has been reported in the Spanish population. It has been proposed that these mutations are probably connected with Celtic and North African ancestry, respectively. We aimed to find the possible ancestral association between HLA alleles and haplotypes associated with the HFE gene (C282Y and H6...

1999
Joanne E. Levy Lynne K. Montross Dena E. Cohen Mark D. Fleming Nancy C. Andrews

Targeted mutagenesis was used to produce two mutations in the murine hemochromatosis gene (Hfe) locus. The first mutation deletes a large portion of the coding sequence, generating a null allele. The second mutation introduces a missense mutation (C282Y) into the Hfe locus, but otherwise leaves the gene intact. This mutation is identical to the disease-causing mutation in patients with heredita...

Journal: :Haematologica 2011
Patricia Aguilar-Martinez Bernard Grandchamp Séverine Cunat Estelle Cadet François Blanc Marlène Nourrit Kaiss Lassoued Jean-François Schved Jacques Rochette

BACKGROUND Heterozygotes for the p.Cys282Tyr (C282Y) mutation of the HFE gene do not usually express a hemochromatosis phenotype. Apart from the compound heterozygous state for C282Y and the widespread p.His63Asp (H63D) variant allele, other rare HFE mutations can be found in trans on chromosome 6. DESIGN AND METHODS We performed molecular investigation of the genes implicated in hereditary h...

Journal: :Human molecular genetics 2014
Felix Stickel Stephan Buch Heinz Zoller Rolf Hultcrantz Sabina Gallati Christoph Österreicher Armin Finkenstedt Andreas Stadlmayr Elmar Aigner Enijad Sahinbegovic Christoph Sarrazin Clemens Schafmayer Felix Braun Wiebke Erhart Michael Nothnagel Markus M Lerch Julia Mayerle Henry Völzke André Schaller Wolfgang Kratzer Bernhard O Boehm Bence Sipos Mauro D'Amato Leif Torkvist Per Stal Alexander Arlt Andre Franke Thomas Becker Michael Krawczak Jochen Zwerina Thomas Berg Holger Hinrichsen Elisabeth Krones Christian Dejaco Michael Strasser Christian Datz Jochen Hampe

Genome-wide association studies (GWAS) have revealed genetic determinants of iron metabolism, but correlation of these with clinical phenotypes is pending. Homozygosity for HFE C282Y is the predominant genetic risk factor for hereditary hemochromatosis (HH) and may cause liver cirrhosis. However, this genotype has a low penetrance. Thus, detection of yet unknown genetic markers that identify pa...

Journal: :Blood 2002
Chrysoula Belessi Kostas Stamatopolous Christos Kosmas

238 may protect against cirrhosis in homozygotes for hemochromatosis. The population studied by Fargion et al was drawn from patients who had been diagnosed clinically as having hemochromatosis. We have had the opportunity to genotype a large number of patients attending a health appraisal clinic, patients with a median age of 56 who represent the full spectrum of the phenotype associated with ...

Journal: :Polskie Archiwum Medycyny Wewnetrznej 2010
Joanna Raszeja-Wyszomirska Grzegorz Kurzawski Iwona Zawada Janina Suchy Jan Lubinski Piotr Milkiewicz

INTRODUCTION Hereditary hemochromatosis has been linked with C282Y and H63D mutations of the HFE gene encoding human hemochromatosis protein. It is genetic disorder of iron metabolism, leading to iron accumulation and increased liver fibrosis. The association between alcoholic liver disease (ALD) and HFE gene mutations remains unclear and requires clarification. OBJECTIVES The aim of the stud...

2017
Sang Y Lee Junjia Zhu Anna C Salzberg Bo Zhang Dajiang J Liu Joshua E Muscat Sara T Langan James R Connor

Human hemochromatosis protein (HFE) is involved in iron metabolism. Two major HFE polymorphisms, H63D and C282Y, have been associated with an increased risk of cancers. Previously, we reported decreased gender effects in overall survival based on H63D or C282Y HFE polymorphisms patients with glioblastoma multiforme (GBM). However, the effect of other single nucleotide variation (SNV) in the HFE...

Journal: :Heart 2003
S Campbell D K George S D Robb R Spooner T A McDonagh H J Dargie P R Mills

OBJECTIVES Excess iron stores have been postulated to enhance the risk of ischaemic heart disease. This study aims to determine whether the two major mutations of the haemochromatosis (HFE) gene (C282Y and H63D) are associated with ischaemic heart disease (IHD) or myocardial infarction (MI). DESIGN Cross sectional case-control study. SETTING The geographical area studied by the MONICA (moni...

Journal: :Environmental Health Perspectives 2004
Robert O Wright Edwin K Silverman Joel Schwartz Shring-Wern Tsaih Jody Senter David Sparrow Scott T Weiss Antonio Aro Howard Hu

Because body iron burden is inversely associated with lead absorption, genes associated with hemochromatosis may modify body lead burden. Our objective was to determine whether the C282Y and/or H63D hemochromatosis gene (HFE) is associated with body lead burden. Patella and tibia lead levels were measured by K X-ray fluorescence in subjects from the Normative Aging Study. DNA samples were genot...

2016
Yang‐Fan Lv Xian Chang Rui‐Xi Hua Guang‐Ning Yan Gang Meng Xiao‐Yu Liao Xi Zhang Qiao‐Nan Guo

To investigate the association between mutation of HFE (the principal pathogenic gene in hereditary haemochromatosis) and risk of cancer, we conducted a meta-analysis of all available case-control or cohort studies relating to two missense mutations, C282Y and H63D mutations. Eligible studies were identified by searching databases including PubMed, Embase and the ISI Web of Knowledge. Overall a...

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