نتایج جستجو برای: bullosa

تعداد نتایج: 3547  

Journal: :Acta dermato-venereologica 2010
Rieko Kabashima Ryosuke Hino Toshinori Bito Kenji Kabashima Motonobu Nakamura Oyama Bungo Takashi Hashimoto Yoshiki Tokura

Journal: :Archives of dermatology 1999
P D Witt D T Cohen S B Mallory

Peter D. Witt, MD; Daniel T. Cohen, BA; Susan B. Mallory, MD; from the Departments of Surgery, Plastic and Reconstructive (Drs Witt and Mr Cohen), Internal Medicine (Dermatology) (Dr Mallory), and Pediatrics (Dr Mallory), Washington University School of Medicine, St Louis Children’s Hospital, and the Department of Plastic and Reconstructive Surgery, Shriners Hospital for Children (Dr Witt), St ...

2015
Rohan Arya Ziad Boujaoude Irwin Gratz Stephen Akers Wissam Abouzgheib

Epidermolysis bullosa rarely affects lower airways. We present a case of lower airway involvement and stenosis successfully managed with flexible bronchoscopy and balloon dilation.

Journal: :Dermatology 1995
E Masgrau-Peya M Lacour D Salomon

Journal: :The Kurume medical journal 1995
H Hachisuka N Yamamoto H Sakihama Y Sasai

Epidermolysis bullosa is a group of inherited blistering diseases classified into three main sub-groups on the basis of the level of cleavage within the skin. In dominant dystrophic epidermolysis bullosa, characterized by cleavage below the basal lamina, two variants can be distinguished by the presence (Pasini form) or absence (Cockayne-Touraine form) of albo-papuloid lesions. The present stud...

2012
Catherine S Yang Yin Lu Anita Farhi Carol Nelson-Williams Michael Kashgarian Earl J Glusac Richard P Lifton Richard J Antaya Keith A Choate

Epidermolysis bullosa pruriginosa (EBP) is a rare subtype of dystrophic epidermolysis bullosa (DEB) characterized by intense pruritus, nodular or lichenoid lesions, and violaceous linear scarring, most prominently on the extensor extremities. Remarkably, identical mutations in COL7A1, which encodes an anchoring fibril protein present at the dermal-epidermal junction, can cause both DEB and EBP ...

Journal: :The New England journal of medicine 2010
John E Wagner Akemi Ishida-Yamamoto John A McGrath Maria Hordinsky Douglas R Keene David T Woodley Mei Chen Megan J Riddle Mark J Osborn Troy Lund Michelle Dolan Bruce R Blazar Jakub Tolar

BACKGROUND Recessive dystrophic epidermolysis bullosa is an incurable, often fatal mucocutaneous blistering disease caused by mutations in COL7A1, the gene encoding type VII collagen (C7). On the basis of preclinical data showing biochemical correction and prolonged survival in col7 −/− mice, we hypothesized that allogeneic marrow contains stem cells capable of ameliorating the manifestations o...

Journal: :Journal of cell science 1995
S M Morley S R Dundas J L James T Gupta R A Brown C J Sexton H A Navsaria I M Leigh E B Lane

Point mutations in the keratin intermediate filament genes for keratin 5 or keratin 14 are known to cause hereditary skin blistering disorders such as epidermolysis bullosa simplex, in which epidermal keratinocytes are extremely fragile and the skin blisters on mild trauma. We show that in 2 phenotypically diverse cases of epidermolysis bullosa simplex, the keratin mutations result in a thermoi...

2016
Valeria Carolina Alvarez Sini Tellervo Penttilä Valeria Luján Salutto Bjarne Udd Claudio Gabriel Mazia

Epidermolysis bullosa simplex with muscular dystrophy (EBS-MD; OMIM #226670) is an autosomal recessive disorder characterized by neonatal blistering and later-onset muscle weakness.

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