نتایج جستجو برای: botulinum neurotoxin type e bonte

تعداد نتایج: 2292107  

Journal: :Clinical infectious diseases : an official publication of the Infectious Diseases Society of America 2013
Patricia C Juliao Susan Maslanka Janet Dykes Linda Gaul Satish Bagdure Lynae Granzow-Kibiger Ellen Salehi Donald Zink Robert P Neligan Casey Barton-Behravesh Carolina Lúquez Matthew Biggerstaff Michael Lynch Christine Olson Ian Williams Ezra J Barzilay

BACKGROUND On 7 and 11 July 2007, health officials in Texas and Indiana, respectively, reported 4 possible cases of type A foodborne botulism to the US Centers for Disease Control and Prevention. Foodborne botulism is a rare and sometimes fatal illness caused by consuming foods containing botulinum neurotoxin. METHODS Investigators reviewed patients' medical charts and food histories. Clinica...

Journal: :Medicina oral, patologia oral y cirugia bucal 2010
Burcu Baş Bora Ozan Mehtap Muğlali Nükhet Celebi

Masseter muscle hypertrophy is a rare condition of unknown cause which is important in the differential diagnosis of head and neck masses, located in the cheek. Several treatment options reported for masseter hypertrophy, which range from simple pharmacotherapy to more invasive surgical reduction. Botulinum toxin type A is a powerful neurotoxin which is produced by the anaerobic organism Clostr...

2008
Rakhi Agarwal Subramanyam Swaminathan

CATALYTICALLY ACTIVE CLOSTRIDIUM BOTULINUM NEUROTOXIN E Rakhi Agarwal and Subramanyam Swaminathan Biology Department, Brookhaven National Laboratory, Upton, New York 11973 USA Running title: Crystal structure of BoNT E -SNAP-25 peptide complex Corresponding author: S. Swaminathan, Biology Department, Brookhaven National Laboratory, Upton, NY 11973, USA. Phone-1(631)344-3187, Fax: 1(631)344-3407...

Journal: :The Journal of pharmacology and experimental therapeutics 2007
Gary Lawrence Jiafu Wang C K N Kwo Chion K Roger Aoki J Oliver Dolly

The unique ability of a family of botulinum neurotoxins to block neuroexocytosis specifically-by selective interaction with peripheral cholinergic nerve endings, endocytotic uptake, translocation to the cytosol, and enzymic cleavage of essential proteins-underlies their increasing therapeutic applications. Although clinical use of type A is most widespread due to its prolonged inactivation of t...

Journal: :International journal of clinical and experimental pathology 2015
C Bandala A L Cortés-Algara C M Mejía-Barradas I Ilizaliturri-Flores R Dominguez-Rubio C I Bazán-Méndez E Floriano-Sánchez J P Luna-Arias M Anaya-Ruiz E Lara-Padilla

AIM It is known that botulinum neurotoxin type A (BoNTA) improves some kinds of cancer (e.g. prostate) and that synaptic vesicle glycoprotein 2 (SV2) is the molecular target of this neurotoxin. Besides having potential therapeutic value, this glycoprotein has recently been proposed as a molecular marker for several types of cancer. Although the mechanisms of cancer development and the improveme...

Journal: :Iranian biomedical journal 2013
Ali Sayadmanesh Firouz Ebrahimi Abbas Hajizade Mosayeb Rostamian Hani Keshavarz

BACKGROUND Botulinum neurotoxin (BoNT) complexes consist of neurotoxin and neurotoxin-associated proteins. Hemagglutinin-33 (HA-33) is a member of BoNT type A (BoNT/A) complex. Considering the protective role of HA-33 in preservation of BoNT/A in gastrointestinal harsh conditions and also its adjuvant role, recombinant production of this protein is favorable. Thus in this study, HA-33 was expre...

2011
Juwan Park Michael S Lee Andrew R Harrison

Even though conventional botulinum neurotoxin (BoNT) products have shown successful treatment results in patients with benign blepharospasm (BEB), the main, potential long-term side effect of BoNT use is the development of immunologic resistance due to the production of neutralizing antibody to the neurotoxin after repeated injections. Xeomin(®) (incobotulinumtoxinA), a unique botulinum neuroto...

Journal: :Microbiological Reviews 1980

Journal: :Neuropsychiatric Disease and Treatment 2007
John P Ney Kevin R Joseph

This article reviews the current and most neurologic uses of botulinum neurotoxin type A (BoNT-A), beginning with relevant historical data, neurochemical mechanism at the neuromuscular junction. Current commercial preparations of BoNT-A are reviewed, as are immunologic issues relating to secondary failure of BoNT-A therapy. Clinical uses are summarized with an emphasis on controlled clinical tr...

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