The new strain of coronavirus SARS CoV-2 can affect any organ and system the body. pathogenesis these lesions is due to both direct damage body cells by virus development immunopathological reactions that lead demyelinating diseases nervous system. article presents a clinical case Guillain-Barré syndrome associated with disease in 71-year-old man who developed after infection virus. was hospita...

To cite: Wakerley BR, Yuki N. J Neurol Neurosurg Psychiatry 2014;85: 339–344. ABSTRACT The pharyngeal-cervical-brachial (PCB) variant of Guillain–Barré syndrome is defined by rapidly progressive oropharyngeal and cervicobrachial weakness associated with areflexia in the upper limbs. Serial nerve conduction studies suggest that PCB represents a localised subtype of Guillain–Barré syndrome charac...

A 52-year-old man experienced fever, sore throat, and rhinorrhea for several days. Some days after resolution he developed dizziness, paresthesia on the hands, and unsteady gait. Ocular movement was not limited. His gait was ataxic and no limb weakness on Romberg sign was evident. Muscle stretch reflexes were absent. Acute cerebellar ataxia was suspected. Anti-GQ1b antibody, however, was detect...

The author reports a case of recurrent Guillain-Barre syndrome with a period of nearly complete remission during 2 years between acute episodes. The case is reported because of its rarity.

Guillain-Barré syndrome (GBS) is an autoimmune and post-infectious immune disease. The syndrome includes several pathological subtypes, the most common of which is a multifocal demyelinating disorder of the peripheral nerves. In the present review, the main clinical aspects and the basic features of GBS are discussed along with approaches to diagnosis and treatment. Furthermore, the pathophysio...

Guillain-Barré syndrome (GBS) is viewed as a reactive, self-limited, autoimmune disease triggered by a preceding bacterial or viral infection. Campylobacter jejuni, a major cause of bacterial gastroenteritis worldwide, is the most frequent antecedent pathogen. It is likely that immune responses directed towards the infecting organisms are involved in the pathogenesis of GBS by cross-reaction wi...

within 4 weeks [1] ; in another, the mean time from onset of symptoms to nadir was 9.7 ( 8 6.9) days (median time, 7 days) [2] . In an additional study from Japan [3] , the median time to nadir was 18.0 8 10.4 days for AIDP and 11.5 days 8 7.8 days for the acute motor axonal neuropathy (AMAN) variant. The occurrence of GBS, in which maximal incapacity is reached within a day or Dear Sir, The cr...