نتایج جستجو برای: گیرنده ccr5

تعداد نتایج: 10087  

2016
Assunta Venuti Claudia Pastori Rosamaria Pennisi Agostino Riva Maria Teresa Sciortino Lucia Lopalco

CCR5 stimulation with natural ligands, such as RANTES, classically induces short-term internalization with transient activation of β-arrestins and rapidly recycling on the cell surface. Here we discovered that, in T cells, natural CCR5 antibodies induce a CCR5-negative phenotype with the involvement of β-arrestin2, which leads to the formation of a stable CCR5 signalosome with both β-arrestin2 ...

Journal: :The Journal of Experimental Medicine 2006
Heather L. Van Epps

A genetic mutation that protects against HIV infection increases the risk of developing clinical West Nile virus (WNV) infection, according to Glass and colleagues on page 35. The mutation in question is a 32-bp deletion in a gene that encodes the chemokine receptor CCR5, which was identified in 1996 as a cellular coreceptor for HIV. Individuals homozygous for this mutation (CCR5∆32) are highly...

2016
Reza Abdolmohammadi Saleh Shahbazi Azar Ayyoob Khosravi Majid Shahbazi

The CC chemokine receptor 5 (CCR5) delta 32 allele results in a nonfunctional form of the chemokine receptor and has been implicated in a variety of immune-mediated diseases. CCR5Δ32 may also predispose one to chronic liver disease or be linked with resistance to HBV infection.This study was undertaken to investigate any association between CCR5 polymorphism with resistance to hepatitis B or su...

Journal: :Oncology reports 2014
Zhiyuan Shen Tianyi Li Da Chen Sen Jia Xiangming Yang Liang Liang Juan Chai Xiaobing Cheng Xinjie Yang Moyi Sun

Salivary adenoid cystic carcinoma (SACC) has a unique tendency for perineural invasion (PNI), which results in tumor recurrence and poor prognosis. Recent studies have shown that the chemokine CCL5 and its receptor CCR5 play important roles in tumor invasion and metastasis. However, the role of the CCL5/CCR5 axis in the PNI of SACC has not been studied to date. In the present study, we evaluate...

Journal: :Journal of immunology 2006
Bing Ma Wei Liu Robert J Homer Patty J Lee Anthony J Coyle Jose M Lora Chun Geun Lee Jack A Elias

IL-13 is a major effector at sites of Th2 inflammation and tissue remodeling. In these locations, it frequently coexists with the CCR5 chemokine receptor and its ligands MIP-1alpha/CCL3 and MIP-1beta/CCL4. We hypothesized that CCR5 induction and activation play important roles in the pathogenesis of IL-13-induced tissue responses. To test this hypothesis, we evaluated the effects of IL-13 on th...

2009
Reem Berro Rogier W. Sanders Min Lu Per J. Klasse John P. Moore

HIV-1 variants resistant to small molecule CCR5 inhibitors recognize the inhibitor-CCR5 complex, while also interacting with free CCR5. The most common genetic route to resistance involves sequence changes in the gp120 V3 region, a pathway followed when the primary isolate CC1/85 was cultured with the AD101 inhibitor in vitro, creating the CC101.19 resistant variant. However, the D1/86.16 escap...

Journal: :The Journal of clinical investigation 1997
O J Cohen M Vaccarezza G K Lam B F Baird K Wildt P M Murphy P A Zimmerman T B Nutman C H Fox S Hoover J Adelsberger M Baseler J Arthos R T Davey R L Dewar J Metcalf D J Schwartzentruber J M Orenstein S Buchbinder A J Saah R Detels J Phair C Rinaldo J B Margolick G Pantaleo A S Fauci

HIV-1-infected long-term nonprogressors are a heterogeneous group of individuals with regard to immunologic and virologic markers of HIV-1 disease. CC chemokine receptor 5 (CCR5) has recently been identified as an important coreceptor for HIV-1 entry into CD4+ T cells. A mutant allele of CCR5 confers a high degree of resistance to HIV-1 infection in homozygous individuals and partial protection...

2014
Ayanna J. Flegler Gianguido C. Cianci Thomas J. Hope

CCR5 acts as the principal coreceptor during HIV-1 transmission and early stages of infection. Efficient HIV-1 entry requires a series of processes, many dependent on the conformational state of both viral envelope protein and cellular receptor. Monoclonal antibodies (MAbs) are able to identify different CCR5 conformations, allowing for their use as probes to distinguish CCR5 populations. Not a...

Journal: :Journal of virology 2013
Reem Berro Anila Yasmeen Ravinder Abrol Bartosz Trzaskowski Sarya Abi-Habib Amy Grunbeck Danny Lascano William A Goddard Per Johan Klasse Thomas P Sakmar John P Moore

Small-molecule CCR5 inhibitors such as vicriviroc (VVC) and maraviroc (MVC) are allosteric modulators that impair HIV-1 entry by stabilizing a CCR5 conformation that the virus recognizes inefficiently. Viruses resistant to these compounds are able to bind the inhibitor-CCR5 complex while also interacting with the free coreceptor. CCR5 also interacts intracellularly with G proteins, as part of i...

Journal: :Blood 2006
Claudia Pastori Barbara Weiser Claudia Barassi Caterina Uberti-Foppa Silvia Ghezzi Renato Longhi Giliola Calori Harold Burger Kimdar Kemal Guido Poli Adriano Lazzarin Lucia Lopalco

Exposure to HIV-1 does not necessarily result in infection and progression toward disease, thus suggesting that the control of viral infection may be achieved. Antibodies to CCR5 have been detected in HIV-exposed but uninfected subjects (ESNs); thus, these antibodies could be involved in HIV protection. To assess whether anti-CCR5 antibodies may also contribute to slow HIV disease progression, ...

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