نتایج جستجو برای: uniparental disomy

تعداد نتایج: 1450  

Journal: :American journal of medical genetics. Part A 2008
Sibel Kantarci Nicola K Ragge N Simon Thomas David O Robinson Kristin M Noonan Meaghan K Russell Dian Donnai F Lucy Raymond Christopher A Walsh Patricia K Donahoe Barbara R Pober

Donnai-Barrow syndrome [Faciooculoacousticorenal (FOAR) syndrome; DBS/FOAR] is a rare autosomal recessive disorder resulting from mutations in the LRP2 gene located on chromosome 2q31.1. We report a unique DBS/FOAR patient homozygous for a 4-bp LRP2 deletion secondary to paternal uniparental isodisomy for chromosome 2. The propositus inherited the mutation from his heterozygous carrier father, ...

Journal: :The Tohoku journal of experimental medicine 2005
Ikuko Takahashi Tsutomu Takahashi Maki Utsunomiya Goro Takada Akio Koizumi

Uniparental disomy (UPD) is the inheritance of a chromosome pair from one parent and is increasingly recognized as a cause of abnormal phenotypes either due to imprinted genes or, in the case of isodisomy, to homozygosity of recessive alleles. Maternal uniparental disomy for chromosome 14 (matUPD[14]) may cause a characteristic phenotype including precocious puberty. Central precocious puberty ...

Journal: :Blood 2012
Joannah Score Claire Hidalgo-Curtis Amy V Jones Nils Winkelmann Alison Skinner Daniel Ward Katerina Zoi Thomas Ernst Frank Stegelmann Konstanze Döhner Andrew Chase Nicholas C P Cross

The polycomb repressive complex 2 (PRC2) is a highly conserved histone H3 lysine 27 methyltransferase that regulates the expression of developmental genes. Inactivating mutations of the catalytic component of PRC2, EZH2, are seen in myeloid disorders. We reasoned that the other 2 core PRC2 components, SUZ12 and EED, may also be mutational targets in these diseases, as well as associated factors...

Journal: :Human molecular genetics 2003
Bernhard Horsthemke Hülya Nazlican Johannes Hüsing Ludger Klein-Hitpass Uwe Claussen Susanne Michel Christina Lich Gabriele Gillessen-Kaesbach Karin Buiting

Although uniparental disomy often results from the postzygotic rescue of a meiotic non-disjunction event, mosaicism is usually confined to the placenta. We describe a girl with Prader-Willi syndrome (PWS) who is mosaic for normal cells and cells with maternal uniparental disomy 15 [upd(15)mat] in blood and skin. Somatic mosaicism was confirmed by cloning and genotyping of skin fibroblasts. X in...

2016
Artur Brandt Katharina Löhers Manfred Beier Barbara Leube Carmen de Torres Jaume Mora Parineeta Arora Parmjit S. Jat Brigitte Royer-Pokora

We describe a stromal predominant Wilms tumor with focal anaplasia and a complex, tumor specific chromosome 11 aberration: a homozygous deletion of the entire WT1 gene within a heterozygous 11p13 deletion and an additional region of uniparental disomy (UPD) limited to 11p15.5-p15.2 including the IGF2 gene. The tumor carried a heterozygous p.T41A mutation in CTNNB1. Cells established from the tu...

Journal: :Blood 2012
Andres Jerez Yuka Sugimoto Hideki Makishima Amit Verma Anna M Jankowska Bartlomiej Przychodzen Valeria Visconte Ramon V Tiu Christine L O'Keefe Azim M Mohamedali Austin G Kulasekararaj Andrea Pellagatti Kathy McGraw Hideki Muramatsu Alison R Moliterno Mikkael A Sekeres Michael A McDevitt Seiji Kojima Alan List Jacqueline Boultwood Ghulam J Mufti Jaroslaw P Maciejewski

Loss of heterozygosity affecting chromosome 7q is common in acute myeloid leukemia and myelodysplastic syndromes, pointing toward the essential role of this region in disease phenotype and clonal evolution. The higher resolution offered by recently developed genomic platforms may be used to establish more precise clinical correlations and identify specific target genes. We analyzed a series of ...

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