نتایج جستجو برای: pgj2

تعداد نتایج: 308  

2005
Javier Gayarre Konstantinos Stamatakis Marta Renedo Dolores Pérez-Sala Vladimir Skulachev

Cyclopentenone prostaglandins (cyPG) with antiinflammatory and antiproliferative properties have been envisaged as leads for the development of therapeutic agents. Because cyPG effects are mediated in part by the formation of covalent adducts with critical signaling proteins, it is important to assess the specificity of this interaction. By using biotinylated derivatives of 15-deoxy-D-PGJ2 (15d...

2005
Hideyuki Migita John Morser

Induced Gene Expression − α Human Vascular Endothelial Cells : The Effect of 15d-PGJ2 on Tumor Necrosis Factorin γ Activated Receptor− but Not Peroxisome Proliferator α Related Orphan Receptor− (15d-PGJ2) Signals Through Retinoic Acid Receptor 2 -Prostaglandin J 12,14 ∆ 15-DeoxyPrint ISSN: 1079-5642. Online ISSN: 1524-4636 Copyright © 2005 American Heart Association, Inc. All rights reserved. G...

2017
Jianhong Lu Shuyuan Guo Xinli Xue Qun Chen Jing Ge Yujuan Zhuo Huiqin Zhong Buxing Chen Mingming Zhao Wei Han Takashi Suzuki Mingjiang Zhu Lin Xia Claus Schneider Timothy S. Blackwell Ned A Porter Lemin Zheng Sotirios Tsimikas Huiyong Yin

Oxidative stress and inflammation are two major contributing factors to atherosclerosis, a leading cause for cardiovascular diseases (CVDs). Oxidation of phospholipids on the surface of low density lipoprotein (LDL) particles generated http://www.jbc.org/cgi/doi/10.1074/jbc.M116.751909 The latest version is at JBC Papers in Press. Published on February 15, 2017 as Manuscript M116.751909 Copyrig...

2014
Yan Yin Gang Hou Erran Li Qiuyue Wang Jian Kang

BACKGROUND Peroxisome proliferator-activated receptor-gamma (PPARγ) is a ligand-activated transcription factor that exerts multiple biological effects. Growing evidence suggests that PPARγ plays an important role in inflammation; however, the effects of this transcription factor on the inflammation caused by smoking are unclear. METHODS We measured the expression of inflammatory cytokines (le...

Journal: :Circulation research 1998
N Marx U Schönbeck M A Lazar P Libby J Plutzky

Migration of vascular smooth muscle cells (VSMCs) plays an important role in atherogenesis and restenosis after arterial interventions. The expression of matrix metalloproteinases (MMPs), particularly MMP-9, contributes to VSMC migration. This process requires degradation of basal laminae and other components of the arterial extracellular matrix. Peroxisome proliferator-activated receptors (PPA...

Journal: :Cancer research 1986
M D Bregman C Funk M Fukushima

The relative inhibitory potency of prostaglandin A (PGA) and prostaglandin J2 (PGJ2) analogues compared to prostaglandin A1 (PGA1) was determined in a clonogenic assay system. Three human melanoma cell strains (C8146A, C8146C, and C8161), a human melanoma cell line (M1RW5) and a human neuroblastoma cell line (IMR-32) were used. Prostaglandin analogues were screened in the clonogenic assay syste...

Journal: :The Journal of investigative dermatology 1987
K Ikai M Ujihara M Kashihara M Fukushima

Cytotoxic action of various prostaglandins (PGs) was examined on the PAM 212 transformed mouse epidermal cell line, and delta 7-PGA1 was found most active. delta 7-PGA1 exerted a dose-dependent inhibition of PAM 212 cell growth over 0.1 microgram/ml (0.3 microM). At 1.6 microgram/ml (4.6 microM) growth was completely inhibited, and the number of viable cells decreased remarkably during culture....

Journal: :Cancer research 2004
Eun Young Park Il Je Cho Sang Geon Kim

Cancer chemopreventive agents transcriptionally induce glutathione S-transferase (GST), which can protect cells from chemical-induced carcinogenesis. Activation of either NF-E2-related factor-2 (Nrf2) or the CCAAT/enhancer binding protein-beta (C/EBPbeta) contributes to GST induction. Peroxisome proliferator-activated receptor-gamma (PPARgamma) and the retinoic acid X receptor (RXR) play roles ...

Journal: :The Journal of investigative dermatology 2005
Christos C Zouboulis

Prostaglandins (PG) are detected in virtually all organs and cells, whereas their enhanced release follows inflammatory mechanisms. PG synthesis, especially of PGE2, PGD2 and PGF2a, also takes place in epidermal keratinocytes as a result of arachidonic acid (AA) metabolism (Kondoh et al, 1985; Pentland and Needleman, 1986) and is differentially supported by specific enzymatic systems, the const...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید