نتایج جستجو برای: npm1 mutation

تعداد نتایج: 292091  

Journal: :Blood 2012
Vera Grossmann Susanne Schnittger Alexander Kohlmann Christiane Eder Andreas Roller Frank Dicker Christoph Schmid Clemens-Martin Wendtner Peter Staib Hubert Serve Karl-Anton Kreuzer Wolfgang Kern Torsten Haferlach Claudia Haferlach

The karyotype is so far the most important prognostic parameter in acute myeloid leukemia (AML). Molecular mutations have been analyzed to subdivide AML with normal karyotype into prognostic subsets. The aim of this study was to develop a prognostic model for the entire AML cohort solely based on molecular markers. One thousand patients with cytogenetic data were investigated for the following ...

Journal: :Haematologica 2010
Thomas Ernst Andrew Chase Katerina Zoi Katherine Waghorn Claire Hidalgo-Curtis Joannah Score Amy Jones Francis Grand Andreas Reiter Andreas Hochhaus Nicholas C P Cross

BACKGROUND Aberrant activation of tyrosine kinases, caused by either mutation or gene fusion, is of major importance for the development of many hematologic malignancies, particularly myeloproliferative neoplasms. We hypothesized that hitherto unrecognized, cytogenetically cryptic tyrosine kinase fusions may be common in non-classical or atypical myeloproliferative neoplasms and related myelody...

2010
Yeo-Kyeoung Kim Hee-Nam Kim Se Ryeon Lee Jae-Sook Ahn Deok-Hwan Yang Je-Jung Lee Il-Kwon Lee Myung-Geun Shin Hyeoung-Joon Kim

BACKGROUND Nucleophosmin (NPM1) gene and fms-like tyrosine kinase 3 gene-internal tandem duplication (FLT3-ITD) mutations are the most frequent mutations in patients with cytogenetically normal (CN)-AML. We analyzed the prognostic impact of these mutations and their interactions in adults with CN-AML. METHODS NPM1 mutation (NPM1mut) and FLT3-ITD mutation (FLT3-ITD+) were analyzed by GeneScan ...

Journal: :Blood 2010
Brunangelo Falini Katja Macijewski Tamara Weiss Ulrike Bacher Susanne Schnittger Wolfgang Kern Alexander Kohlmann Hans-Ulrich Klein Marco Vignetti Alfonso Piciocchi Paola Fazi Maria Paola Martelli Antonella Vitale Stefano Pileri Miriam Miesner Antonella Santucci Claudia Haferlach Franco Mandelli Torsten Haferlach

NPM1-mutated acute myeloid leukemia (AML) is a provisional entity in the 2008 World Health Organization (WHO) classification of myeloid neoplasms. The significance of multilineage dysplasia (MLD) in NPM1-mutated AML is unclear. Thus, in the 2008 WHO classification, NPM1-mutated AML with MLD is classified as AML with myelodysplasia (MD)-related changes (MRCs). We evaluated morphologically 318 NP...

2016
Zhuo Ren Joeri L Aerts Hugo Vandenplas Jiance A Wang Olena Gorbenko Jack P Chen Philippe Giron Carlo Heirman Cleo Goyvaerts Eldad Zacksenhaus Mark D Minden Vuk Stambolic Karine Breckpot Jacques De Grève

Signal transducer and activator of transcription 5 (STAT5) and nucleophosmin (NPM1) are critical regulators of multiple biological and pathological processes. Although a reciprocal regulatory relationship was established between STAT5A and a NPM-ALK fusion protein in T-cell lymphoma, no direct connection between STAT5 and wild-type NPM1 has been documented. Here we demonstrate a mutually regula...

Journal: :American journal of clinical pathology 2010
Jennifer Luo Connie Qi Wei Xu Suzanne Kamel-Reid Joseph Brandwein Hong Chang

Mutations in the nucleophosmin (NPM1) exon 12 resulting in delocalization of NPM1 into the cytoplasm occur in 50% to 60% of acute myeloid leukemia cases with a normal karyotype (AML-NK). As recent studies suggest such patients have a favorable prognosis and there are discordant reports of the immunohistochemical detection of cytoplasmic NPM1 (NPMc+) for predicting NPM1 gene mutations, we correl...

2010
Roberta La Starza Caterina Matteucci Paolo Gorello Lucia Brandimarte Valentina Pierini Barbara Crescenzi Valeria Nofrini Roberto Rosati Enrico Gottardi Giuseppe Saglio Antonella Santucci Laura Berchicci Francesco Arcioni Brunangelo Falini Massimo Fabrizio Martelli Constantina Sambani Anna Aventin Cristina Mecucci

BACKGROUND NPM1 gene at chromosome 5q35 is involved in recurrent translocations in leukemia and lymphoma. It also undergoes mutations in 60% of adult acute myeloid leukemia (AML) cases with normal karyotype. The incidence and significance of NPM1 deletion in human leukemia have not been elucidated. METHODOLOGY AND PRINCIPAL FINDINGS Bone marrow samples from 145 patients with myelodysplastic s...

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