نتایج جستجو برای: nf e2 related factor 2

تعداد نتایج: 4030098  

2016
Maya Ben-Yehuda Shmuel Ben-Sasson Ron Kohen Martina Meinke

The skin, being the largest organ of the body, functions as a barrier between our body and the environment. It is consistently exposed to various exogenous and endogenous stressors (e.g., air pollutants, ionizing and non-ionizing irradiation, toxins, mitochondrial metabolism, enzyme activity, inflammatory process, etc.) producing reactive oxygen species (ROS) and physical damage (e.g., wounds, ...

Journal: :Molecular biology and evolution 2013
Rachele Cagliani Uberto Pozzoli Diego Forni Andrea Cassinotti Matteo Fumagalli Matteo Giani Maria Fichera Marta Lombardini Sandro Ardizzone Rosanna Asselta Roberto de Franchis Stefania Riva Mara Biasin Giacomo P Comi Nereo Bresolin Mario Clerici Manuela Sironi

Previous studies indicated that a few risk variants for autoimmune diseases are subject to pathogen-driven selection. Nonetheless, the proportion of risk loci that has been targeted by pathogens and the type of infectious agent(s) that exerted the strongest pressure remain to be evaluated. We assessed whether different pathogens exerted a pressure on known Crohn's disease (CD) risk variants and...

Journal: :Free radical research 2012
Abigail F Smith George Loo

Haeme oxygenase-1 (HO-1) is often viewed as a cytoprotective gene. Toxic heavy metals induce HO-1, but it is unclear whether particular metal micronutrients also induce HO-1. Hence, the ability of exogenously-added copper, iron and zinc to influence HO-1 expression in HCT-116 cells was evaluated. Under the chosen experimental conditions, only zinc noticeably increased the expression of HO-1 mRN...

2014
Lei Qiu Zhiqi Song Vijayasaradhi Setaluri

Hypersensitivity of epidermal melanocytes to oxidative stress is known to contribute to vitiligo pathogenesis. Molecular mechanisms that connect melanocyte redox homeostasis to the complex disease phenotype are not fully understood. Jian et al. show that vitiligo melanocytes have impaired nuclear factor erythroid 2-related factor 2 (Nrf2)-antioxidant response element signaling and decreased act...

Journal: :Cell reports 2016
Leonard D Goldstein James Lee Florian Gnad Christiaan Klijn Annalisa Schaub Jens Reeder Anneleen Daemen Corey E Bakalarski Thomas Holcomb David S Shames Ryan J Hartmaier Juliann Chmielecki Somasekar Seshagiri Robert Gentleman David Stokoe

The Nrf2 pathway is frequently activated in human cancers through mutations in Nrf2 or its negative regulator KEAP1. Using a cell-line-derived gene signature for Nrf2 pathway activation, we found that some tumors show high Nrf2 activity in the absence of known mutations in the pathway. An analysis of splice variants in oncogenes revealed that such tumors express abnormal transcript variants fro...

2017
Sandro Satta Ayman M Mahmoud Fiona L Wilkinson M Yvonne Alexander Stephen J White

Free radicals, reactive oxygen/nitrogen species (ROS/RNS), hydrogen sulphide, and hydrogen peroxide play an important role in both intracellular and intercellular signaling; however, their production and quenching need to be closely regulated to prevent cellular damage. An imbalance, due to exogenous sources of free radicals and chronic upregulation of endogenous production, contributes to many...

Journal: :The Journal of pharmacology and experimental therapeutics 2010
Xiaoqing He Qiang Ma

Arsenic activates nuclear factor erythroid 2-related factor 2 (Nrf2) to induce phase II and antioxidative genes. Here we analyzed arsenic-Kelch-like ECH-associated protein 1 (Keap1) cysteine thiol interaction in Nrf2 activation. Arsenic-based Nrf2 activators, fluorescent biarsenical labeling reagent (FlAsH) and phenylarsine oxide (PAO), were used to probe binding of arsenic to Keap1. Strong flu...

2013
Wenpeng Cui Yang Bai Ping Luo Lining Miao Lu Cai

So far, cardiovascular and renal diseases have brought us not only huge economic burden but also serious society problems. Since effective therapeutic strategies are still limited, to find new methods for the prevention or therapy of these diseases is important. Oxidative stress has been found to play a critical role in the initiation and progression of cardiovascular and renal diseases. In add...

2015
Yang Bai Xiaolu Wang Song Zhao Chunye Ma Jiuwei Cui Yang Zheng

Cardiovascular disease (CVD) causes an unparalleled proportion of the global burden of disease and will remain the main cause of mortality for the near future. Oxidative stress plays a major role in the pathophysiology of cardiac disorders. Several studies have highlighted the cardinal role played by the overproduction of reactive oxygen or nitrogen species in the pathogenesis of ischemic myoca...

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