نتایج جستجو برای: neurovascular decompression

تعداد نتایج: 18486  

Journal: :Neurosurgical focus 2005
Peter J Jannetta Mark R McLaughlin Kenneth F Casey

Vascular compression of the trigeminal nerve in the cerebellopontine angle is now generally accepted as the primary source or "trigger" causing trigeminal neuralgia. A clear clinicopathological association exists in the neurovascular relationship. In general, pain in the third division of the trigeminal nerve is caused by rostral compression, pain in the second division is caused by medial or m...

Journal: :Egyptian Journal of Neurosurgery 2022

Abstract Background Microvascular decompression surgery for trigeminal neuralgia is now considered the benchmark surgical procedure intractable of any variety. The objective this study to share our experience in managing classical with microvascular looking into both immediate and late functional outcomes at 3 6 months post op. We also report a unique case due tributary petrosal vein passing th...

Journal: :AJNR. American journal of neuroradiology 1998
H Mitsuoka A Tsunoda O Okuda K Sato J Makita

BACKGROUND AND PURPOSE We applied a 3D fast spin-echo (3D-FSE) MR imaging technique to the preoperative and postoperative evaluation of patients with hemifacial spasm. METHODS The study group comprised 20 patients. All images were acquired on a 1.5-T MR system with a 3D-FSE sequence. RESULTS In all 20 patients, the courses of the seventh and eighth cranial nerves were depicted separately, a...

Journal: :Archives of otolaryngology--head & neck surgery 2010
Matthew L Carlson Charles W Beatty Brian A Neff Michael J Link Colin L W Driscoll

OBJECTIVE To describe presenting symptoms, evaluation findings, and surgical management of cranial base hyperostosis in patients with Camurati-Engelmann disease (CED). DESIGN Retrospective study and literature review. SETTING The Mayo Clinic, Rochester, Minnesota. PATIENTS A total of 306 patients diagnosed as having CED, including 12 primarily evaluated at our institution between 1968 and...

2015
Kevin R. Choksey Jayprakash V. Modi

INTRODUCTION Dystrophic neurofibromatosis type I, involving upper cervical spines, is rare, which can cause serious complications. Myelopathy develops due to compression of the cord posteriorly. Surgical correction has its inherent risks and difficulties because of poor bone quality, difficult anterior approach because of bizarre deformities, and the necessary manipulation, which might cause mo...

2015
David W. Lowry Scott M. Tuinstra Kevin Liang Joseph A. Sclafani

BACKGROUND Although anterior cervical decompression and fusion (ACDF) can be performed using minimally invasive techniques, the extensive removal of anatomical keystones during decompression requires a segmental fusion to restore biomechanical stability. Treatment with arthrodesis techniques may result in a prolonged recovery time, loss of motion, and the need for further treatment if a pseudar...

Journal: :The Journal of craniofacial surgery 2014
Lei Xia Jun Zhong Jin Zhu Yong-Nan Wang Ning-Ning Dou Ming-Xing Liu Massimiliano Visocchi Shi-Ting Li

Microvascular decompression has been now accepted worldwide as a reasonable treatment for trigeminal neuralgia, yet, as a functional operation in the cerebellopontine angle, this process may be risky and the postoperative outcomes might not be good enough sometimes. To assess the effectiveness and safety of microvascular decompression for treatment of trigeminal neuralgia, we conducted a system...

Journal: :Clinical neurology and neurosurgery 2015
Levent Tanrikulu Torben Scholz Omid Nikoubashman Martin Wiesmann Hans Clusmann

BACKGROUND Neurovascular compression (NVC) in the posterior fossa is characterized by complex, three-dimensional (3D) neurovascular relationships at the root entry zones (REZ) and other parts of cranial nerves, resulting in syndromes such as trigeminal neuralgia (TN), hemifacial spasm, vertigo and glossopharyngeal neuralgia. Microvascular decompression (MVD) requires microsurgical experience an...

2011
Anusha Mishra Eric A. Newman

.................................................................................................... iii List of tables ........................................................................................... viii List of figures............................................................................................ ix Chapter 1: Introduction.................................................

Journal: :British Journal of Sports Medicine 1986

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