نتایج جستجو برای: lpl

تعداد نتایج: 1978  

Journal: :Journal of lipid research 1990
A Pradines-Figuères C Vannier G Ailhaud

The status of lipoprotein lipase (LPL) has been examined in different cell types (adipose, skeletal muscle, and heart muscle cells) and different tissues (adipose, muscle, and cardiac tissues) from mouse, rat, and human. Cell and secreted activities were compared in cycloheximide-, heparin-treated cells present in culture. A gross underestimation of cell LPL activity was found; excess of LPL ov...

Journal: :Comparative biochemistry and physiology. Part D, Genomics & proteomics 2011
Roger S Holmes John L Vandeberg Laura A Cox

Lipoprotein lipase (LIPL or LPL; E.C.3.1.1.34) serves a dual function as a triglyceride lipase of circulating chylomicrons and very-low-density lipoproteins (VLDL) and facilitates receptor-mediated lipoprotein uptake into heart, muscle and adipose tissue. Comparative LPL amino acid sequences and protein structures and LPL gene locations were examined using data from several vertebrate genome pr...

Journal: :Endocrinology 1999
P Ramos A Martín-Hidalgo E Herrera

The effects of insulin on lipoprotein lipase (LPL) messenger RNA (mRNA) were studied in mammary glands from virgin and late-pregnant rats. Virgin and pregnant rats (at 17 days of gestation) were subjected to a continuous infusion (35 ml/day) with 50% glucose for 72 h to produce a prolonged hyperinsulinemic-euglycemic condition. Gestation causes a 4-fold increase in LPL mRNA accompanied by a 3- ...

Journal: :Molecular cancer research : MCR 2015
Uri Rozovski Srdana Grgurevic Carlos Bueso-Ramos David M Harris Ping Li Zhiming Liu Ji Yuan Wu Preetesh Jain William Wierda Jan Burger Susan O'Brien Nitin Jain Alessandra Ferrajoli Michael J Keating Zeev Estrov

UNLABELLED While reviewing chronic lymphocytic leukemia (CLL) bone marrow slides, we identified cytoplasmic lipid vacuoles in CLL cells but not in normal B cells. Because lipoprotein lipase (LPL), which catalyzes hydrolysis of triglycerides into free fatty acids (FFA), is aberrantly expressed in CLL, we investigated whether LPL regulates the oxidative metabolic capacity of CLL cells. We found t...

Journal: :Arteriosclerosis, thrombosis, and vascular biology 2012
Maryam Hosseini Nicole Ehrhardt Daphna Weissglas-Volkov Ching-Mei Lai Hui Z Mao Jo-Ling Liao Elina Nikkola André Bensadoun Marja-Riitta Taskinen Mark H Doolittle Päivi Pajukanta Miklós Péterfy

OBJECTIVE Lipoprotein lipase (LPL) is a principal enzyme in lipoprotein metabolism, tissue lipid utilization, and energy metabolism. LPL is synthesized by parenchymal cells in adipose, heart, and muscle tissues followed by secretion to extracellular sites, where lipolyic function is exerted. The catalytic activity of LPL is attained during posttranslational maturation, which involves glycosylat...

Journal: :Current vascular pharmacology 2003
K Tsutsumi

Lipoprotein lipase (LPL) is a rate-limiting enzyme that hydrolyzes circulating triglyceride-rich lipoprotein such as very low density lipoproteins and chylomicrons. A decrease in LPL activity is associated with an increase in plasma triglycerides (TG) and decrease in high density lipoprotein (HDL) cholesterol. The increase in plasma TG and decrease in HDL cholesterol are risk factors of coronar...

زمینه و هدف: لیپوپروتین لیپاز یکی از مهمترین آنزیم های مرتبط با کنترل لیپیدهای پلاسما می باشد. تری اسیل گلیسرول، شیلومیکرون ها و VLDL به وسیله آنزیم لیپوپروتئین لیپاز (LPL) هیدرولیز می شوند. عوامل بسیاری همچون ApoCI، ApoCII، ApoCIII، ApoA5، ApoE، ANGPTL3، ANGPTL4 و ANGPTL5 در کنترل فعالیت این آنزیم دخالت دارند. بنابراین هدف مطالعه حاضر، مرور مطالعاتی بود که نقش های آنزیم لیپوپروتئین لیپاز را بر...

A.R Bandegi M Firoozray M.R Akbari-Eydgahi

Background & Aims: Lipoprotein lipase (LPL) is one of the key enzymes regulating the metabolism of triglycerides (TG) and HDL cholesterol. The lipoprotein lipase (LPL) gene polymorphisms are possibly involved in the pathophysiology of dyslipidemia. Hind III polymorphism is one of the most common polymorphisms in LPL gene. In some studies, association of Hind III polymorphism with dyslipidemia h...

Journal: :Circulation Research 2008

Journal: :Journal of immunology 2010
Chen Wang Sharon Celeste Morley David Donermeyer Ivan Peng Wyne P Lee Jason Devoss Dimitry M Danilenko Zhonghua Lin Juan Zhang Jie Zhou Paul M Allen Eric J Brown

Engagement of TCRs induces actin rearrangements, which are critical for T cell activation. T cell responses require new actin polymerization, but the significance of higher-order actin structures, such as microfilament bundles, is unknown. To determine the role of the actin-bundling protein leukocyte-plastin (L-plastin; LPL) in this process, T cells from LPL(-/-) mice were studied. LPL(-/-) T c...

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